KANO TOSHIKI 狩野俊樹

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Kano Toshiki 狩野俊樹

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Researcher Number	00866733
Other IDs
Affiliation (Current)	2025: 順天堂大学, 医学部, 助教
Affiliation (based on the past Project Information) *help	2021 – 2023: 順天堂大学, 医学部, 助教 2020: 順天堂大学, 医学部, 助手
Review Section/Research Field	Principal Investigator Basic Section 53040:Nephrology-related
Keywords	Principal Investigator 糖鎖異常IgA / 粘膜免疫応答異常 / IgA腎症 / APRIL/BAFF / 腸管粘膜免疫 / Toll Like Receptors / Toll like receptors / 糖鎖異常IgA1 / 無菌環境 / 腸管関連リンパ組織 / 上気道関連リンパ組織

ＩgＡ腎症におけるAPRIL/BAFFを介した腸管粘膜免疫応答異常の解明Principal Investigator
- Principal Investigator
  
  狩野俊樹
- Project Period (FY)
  2022 – 2024
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 53040:Nephrology-related
- Research Institution
  Juntendo University
Dysregulation of mucosal immune response in IgA nephropathyPrincipal Investigator
- Principal Investigator
  
  Kano Toshiki
- Project Period (FY)
  2020 – 2021
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 53040:Nephrology-related
- Research Institution
  Juntendo University

All 2023 2022 2021

All Journal Article Presentation

[Journal Article] The nucleotide-sensing Toll-Like Receptor 9/Toll-Like Receptor 7 system is a potential therapeutic target for IgA nephropathy.2023
- Author(s)
  Lee M, Suzuki H, Ogiwara K, Aoki R, Kato R, Nakayama M, Fukao Y, Nihei Y, Kano T, Makita Y, Muto M, Yamada K, Suzuki Y
- Journal Title
  
  Kidney international
  
  Volume: 104 Issue: 5 Pages: 943-955
- DOI
  10.1016/j.kint.2023.08.013
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-22K16226, KAKENHI-PROJECT-22K08336, KAKENHI-PROJECT-22K08362, KAKENHI-PROJECT-21K08285
[Journal Article] Mucosal immune system dysregulation in the pathogenesis of IgA nephropathy2023
- Author(s)
  Kano T, Suzuki H*, Makita Y, Nihei Y, Fukao Y, Nakayama M, Lee M, Kato R, Aoki R, Yamada K, Muto M, Suzuki Y
- Journal Title
  
  Biomedicines
  
  Volume: 10 Issue: 12 Pages: 3027-3027
- DOI
  10.3390/biomedicines10123027
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-21K08285, KAKENHI-PROJECT-22K08336, KAKENHI-PROJECT-22K16226
[Journal Article] Nasal-associated lymphoid tissue is the major induction site for nephritogenic IgA in murine IgA nephropathy2021
- Author(s)
  Kano T, Suzuki H, Makita Y, Fukao Y, Suzuki Y
- Journal Title
  
  Kidney International
  
  Volume: 100 Issue: 2 Pages: 364-376
- DOI
  10.1016/j.kint.2021.04.026
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-20K17293, KAKENHI-PROJECT-21K08285
[Presentation] Nucleotide-sensing TLR9/TLR7: therapeutic candidate for IgA nephropathy.2023
- Author(s)
  Lee M, Suzuki H, Kato R, Fukao Y, Nakayama M, Nihei Y, Kano T, Makita Y, Suzuki Y
- Organizer
  the 17th international Symposium on IgA Nephropathy
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-22K16226
[Presentation] TLR9/TLR7 are strong candidates for diseas-specific therapeutic targets in IgA nephropathy.2023
- Author(s)
  Lee M, Suzuki H, Kato R, Fukao Y, Nakayama M, Kano T, Makita Y, Suzuki Y
- Organizer
  the World Congress of Nephrology
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-22K16226
[Presentation] Plasmacytoid dendritic cells are involved in the production of galactose-deficient IgA1.2023
- Author(s)
  Fukao Y, Suzuki H, Nihei Y, Makita Y, Kano T, Nakayama M, Lee M, Kato R, Suzuki Y
- Organizer
  the 17th international Symposium on IgA Nephropathy
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-22K16226
[Presentation] 糖鎖異常IgA産生における形質細胞様樹状細胞の役割2022
- Author(s)
  深尾勇輔、鈴木仁、牧田侑子、狩野俊樹、二瓶義人、中山麻衣子、李明峰、加藤莉那、鈴木祐介
- Organizer
  第65回日本腎臓学会学術集会・総会
- Data Source
  KAKENHI-PROJECT-22K16226

1. SUZUKI Hitoshi

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

KANO TOSHIKI 狩野 俊樹

Kano Toshiki 狩野 俊樹

Research Projects

Research Products

Co-Researchers

ＩgＡ腎症におけるAPRIL/BAFFを介した腸管粘膜免疫応答異常の解明Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Dysregulation of mucosal immune response in IgA nephropathyPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] The nucleotide-sensing Toll-Like Receptor 9/Toll-Like Receptor 7 system is a potential therapeutic target for IgA nephropathy.2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Mucosal immune system dysregulation in the pathogenesis of IgA nephropathy2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Nasal-associated lymphoid tissue is the major induction site for nephritogenic IgA in murine IgA nephropathy2021

Author(s)

Journal Title

DOI

Data Source

[Presentation] Nucleotide-sensing TLR9/TLR7: therapeutic candidate for IgA nephropathy.2023

Author(s)

Organizer

Data Source

[Presentation] TLR9/TLR7 are strong candidates for diseas-specific therapeutic targets in IgA nephropathy.2023

Author(s)

Organizer

Data Source

[Presentation] Plasmacytoid dendritic cells are involved in the production of galactose-deficient IgA1.2023

Author(s)

Organizer

Data Source

[Presentation] 糖鎖異常IgA産生における形質細胞様樹状細胞の役割2022

Author(s)

Organizer

Data Source

KANO TOSHIKI 狩野俊樹

Kano Toshiki 狩野俊樹