NISHIKAWA Masashi 西川将司

… Alternative Names

Nishikawa Masashi 西川将司

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Researcher Number	00871758
Other IDs
Affiliation (Current)	2025: 名古屋大学, 理学研究科, 助教
Affiliation (based on the past Project Information) *help	2022 – 2023: 名古屋大学, 理学研究科, 助教 2020 – 2021: 愛知県医療療育総合センター発達障害研究所, 分子病態研究部, リサーチレジデント
Review Section/Research Field	Principal Investigator Basic Section 52050:Embryonic medicine and pediatrics-related / 0902:General internal medicine and related fields
Keywords	Principal Investigator 知的障害 / Gタンパク質 / シグナル伝達 / 細胞骨格 / 発達障害 / 神経発達 / アクチン細胞骨格 / Rho / G蛋白質 / 細胞内シグナル伝達 … More / Cdc42 / 神経発生 / Rho GTPase / Rac / 子宮内胎仔脳遺伝子導入 / 神経細胞移動 / アクチン / 大脳皮質構築 / PAK1 / Rac3 / Rhoファミリー / 子宮内胎児脳電気穿孔法 / 小頭症 / 知的障害(ID) / 発現解析 / マウス組織 / 神経細胞発達 / RhoGEF Less

Cdc42異常に基づく小頭症・脳皮質形成異常症を伴う知的障害の病態メカニズム解析Principal Investigator
- Principal Investigator
  
  西川将司
- Project Period (FY)
  2023 – 2024
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 52050:Embryonic medicine and pediatrics-related
- Research Institution
  Nagoya University
Physiological functions and pathogenic mechanisms of Rac3, an intellectual disability-associated G protein, in cerebral developmentPrincipal Investigator
- Principal Investigator
  
  Nishikawa Masashi
- Project Period (FY)
  2021 – 2022
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 52050:Embryonic medicine and pediatrics-related
- Research Institution
  Nagoya University
  Institute for Developmental Research Aichi Developmental Disability Center
Pathophysiological analyses of PLEKHG2, a responsible gene for neurodevelopmental disorders, during corticogenesisPrincipal Investigator
- Principal Investigator
  
  Nishikawa Masashi
- Project Period (FY)
  2020 – 2021
- Research Category
  
  Grant-in-Aid for Research Activity Start-up
- Review Section
  
  0902:General internal medicine and related fields
- Research Institution
  Institute for Developmental Research Aichi Developmental Disability Center

All 2024 2023 2022 2021 2020

All Journal Article Presentation

[Journal Article] Live Imaging of Migrating Neurons and Glial Progenitors Visualized by in Utero Electroporation2024
- Author(s)
  Nishikawa Masashi、Nagata Koh-ichi、Tabata Hidenori
- Journal Title
  
  Methods in Molecular Biology
  
  Volume: - Pages: 201-209
- DOI
  10.1007/978-1-0716-3810-1_17
- ISBN
  9781071638095, 9781071638101
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23K14946
[Journal Article] A Novel Constitutively Active c.98G>C, p.(R33P) Variant in RAB11A Associated with Intellectual Disability Promotes Neuritogenesis and Affects Oligodendroglial Arborization2023
- Author(s)
  Yumi Tsuneura, Taeko Kawai, Keitaro Yamada, Shintaro Aoki, Mitsuko Nakashima, Shima Eda, Tohru Matsuki, Masashi Nishikawa, Koh-ichi Nagata, Yasushi Enokido, Hirotomo Saitsu, Atsuo Nakayama
- Journal Title
  
  Human Mutation
  
  Volume: 2023 Pages: 8126544-8126544
- DOI
  10.1155/2023/8126544
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-22K15912, KAKENHI-PROJECT-23K14946, KAKENHI-PROJECT-22K07882, KAKENHI-PROJECT-20K08224
[Journal Article] Expression analyses of WAC, a responsible gene for neurodevelopmental disorders, during mouse brain development2023
- Author(s)
  Nishikawa Masashi、Matsuki Tohru、Hamada Nanako、Nakayama Atsuo、Ito Hidenori、Nagata Koh-ichi
- Journal Title
  
  Medical Molecular Morphology
  
  Volume: 56 Issue: 4 Pages: 266-273
- DOI
  10.1007/s00795-023-00364-x
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-23K14946, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08224, KAKENHI-PROJECT-23K24310
[Journal Article] Expression Analyses of Rich2/Arhgap44, a Rho Family GTPase-Activating Protein, during Mouse Brain Development.2023
- Author(s)
  Goto N, Nishikawa M, Ito H, Noda M, Hamada N, Tabata H, Kinoshita M, Nagata K-I
- Journal Title
  
  Dev. Neurosci.
  
  Volume: 45 Issue: 1 Pages: 19-26
- DOI
  10.1159/000529051
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-21K15895, KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-20K08200
[Journal Article] A missense variant at the RAC1-PAK1 binding site of RAC1 inactivates downstream signaling in VACTERL association2023
- Author(s)
  Seyama Rie、Nishikawa Masashi、Ogata Kazuhiro、Nagata Koh-ichi、Matsumoto Naomichi
- Journal Title
  
  Scientific Reports
  
  Volume: 13 Issue: 1 Pages: 9789-9789
- DOI
  10.1038/s41598-023-36381-0
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-23K18278, KAKENHI-PROJECT-23K14946, KAKENHI-PROJECT-21K06051, KAKENHI-PROJECT-23K07229, KAKENHI-PROJECT-23K27520, KAKENHI-PROJECT-23K27568
[Journal Article] Impaired Function of PLEKHG2, a Rho-Guanine Nucleotide-Exchange Factor, Disrupts Corticogenesis in Neurodevelopmental Phenotypes.2022
- Author(s)
  Nishikawa M, Ito H, Tabata H, Ueda H, Nagata KI.
- Journal Title
  
  Cells
  
  Volume: 16 Issue: 4 Pages: 696-696
- DOI
  10.3390/cells11040696
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-19K07043, KAKENHI-PROJECT-20K22888, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-22K06579, KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-23K24310
[Journal Article] Variant-specific changes in RAC3 function disrupt corticogenesis in neurodevelopmental phenotypes2022
- Author(s)
  Scala Marcello et al.
- Journal Title
  
  Brain
  
  Volume: 145 Issue: 9 Pages: 3308-3327
- DOI
  10.1093/brain/awac106
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-21K07309, KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-21K15895, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-23K24310
[Journal Article] Gain-of-function p.F28S variant in<i>RAC3</i>disrupts neuronal differentiation, migration and axonogenesis during cortical development, leading to neurodevelopmental disorder2022
- Author(s)
  Nishikawa Masashi、Scala Marcello、Umair Muhammad、Ito Hidenori、Waqas Ahmed、Striano Pasquale、Zara Federico、Costain Gregory、Capra Valeria、Nagata Koh-ichi
- Journal Title
  
  Journal of Medical Genetics
  
  Volume: 60 Issue: 3 Pages: 223-232
- DOI
  10.1136/jmedgenet-2022-108483
- Peer Reviewed / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-21K15895, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-23K24310
[Journal Article] Expression analyses of Rac3, a Rho family small GTPase, during mouse brain development.2022
- Author(s)
  Nishikawa M, Ito H, Noda M, Hamada N, Tabata H, Nagata K.
- Journal Title
  
  Dev Neurosci
  
  Volume: 44 Issue: 1 Pages: 49-58
- DOI
  10.1159/000521168
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-21K15895, KAKENHI-PROJECT-19H03629
[Journal Article] Pathophysiological Mechanisms in Neurodevelopmental Disorders Caused by Rac GTPases Dysregulation: What’s behind Neuro-RACopathies2021
- Author(s)
  Scala Marcello、Nishikawa Masashi、Nagata Koh-ichi、Striano Pasquale
- Journal Title
  
  Cells
  
  Volume: 10 Issue: 12 Pages: 3395-3395
- DOI
  10.3390/cells10123395
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-21K15895
[Journal Article] Expression Analyses of Mediator Complex Subunit 13-Like: A Responsible Gene for Neurodevelopmental Disorders during Mouse Brain Development2021
- Author(s)
  Hamada Nanako、Iwamoto Ikuko、Nishikawa Masashi、Nagata Koh-ichi
- Journal Title
  
  Developmental Neuroscience
  
  Volume: - Issue: 1 Pages: 1-10
- DOI
  10.1159/000515188
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-20K22888, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K21589
[Journal Article] Expression analyses of PLEKHG2, a Rho family-specific guanine nucleotide exchange factor, during mouse brain development2021
- Author(s)
  Nishikawa Masashi、Ito Hidenori、Noda Mariko、Hamada Nanako、Tabata Hidenori、Nagata Koh-ichi
- Journal Title
  
  Medical Molecular Morphology
  
  Volume: - Issue: 2 Pages: 146-155
- DOI
  10.1007/s00795-020-00275-1
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-20K22888, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-20K21589
[Presentation] Rac-グアニンヌクレオチド交換因子 Prex1 の神経発達における生理機能2023
- Author(s)
  鳥居紗帆, 西川将司, 内山由理, 後藤直樹, 伊東秀記, 上田浩, 松本直通, 永田浩一, 木下専
- Organizer
  第64回日本神経病理学会総会学術研究会/ 第66回日本神経化学会大会合同大会 2023年7月7日
- Data Source
  KAKENHI-PROJECT-23K14946
[Presentation] 神経発達におけるRac1制御分子Rich2の発現プロファイル解析2022
- Author(s)
  後藤直樹, 西川将司, 木下専, 永田浩一
- Organizer
  第54回日本臨床分子形態学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] PLEKHG2遺伝子変異による神経発達障害の発症機構の形態的解析2022
- Author(s)
  永田浩一, 西川将司, 伊東秀記, 田畑秀典
- Organizer
  第54回日本臨床分子形態学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-20K22888
[Presentation] Impaired function of PLEKHG2, a Rho-guanine nucleotide-exchange factor, disrupts corticogenesis in neurodevelopmental phenotypes2022
- Author(s)
  西川将司, 伊東秀記, 田畑秀典, 永田浩一
- Organizer
  NEURO2022
- Data Source
  KAKENHI-PROJECT-20K22888
[Presentation] 知的障害責任分子RAC3による神経発達障害の病態メカニズム2022
- Author(s)
  西川将司, 伊東秀記, 田畑秀典, 永田浩一
- Organizer
  第86回日本生化学会中部支部例会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] RAC3遺伝子変異は神経発達過程の大脳皮質形成を障害する2022
- Author(s)
  西川将司, 伊東秀記, 田畑秀典, 永田浩一
- Organizer
  第95回日本生化学会大会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] Variant-specific changes in RAC3 function disrupt corticogenesis in neurodevelopmental phenotypes2022
- Author(s)
  Koh-ichi Nagata, Masashi Nishikawa, Hidenori Ito
- Organizer
  NEURO2022
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 知的障害責任分子RAC3による神経発達障害の病態メカニズム2022
- Author(s)
  西川将司, 伊東秀記, 田畑秀典, 永田浩一
- Organizer
  第86回日本生化学会中部支部例会・シンポジウム
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 知的障害責任分子群RACファミリーの大脳発達における生理機能と分子病態機構の解明2022
- Author(s)
  西川将司
- Organizer
  第20回東海小児遺伝カンファレンス 2022年2月19日
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] Rac制御分子Rich2は神経発達に伴って発現分布が変化する2022
- Author(s)
  後藤直樹, 西川将司, 伊東秀記, 田畑秀典, 木下専, 永田浩一
- Organizer
  第46回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] アダプタータンパク質NCK2によるSH3ドメインを介したRho活性化因子PLEKHG1の活性制御2021
- Author(s)
  井藤拓哉、後藤未沙紀、中野駿、西川将司、山川央、長瀬隆弘、上田浩
- Organizer
  日本薬学会
- Data Source
  KAKENHI-PROJECT-20K22888
[Presentation] Pathophysiological significance of RAC3 variants in neurodevelopmental disorders2021
- Author(s)
  西川将司, 伊東秀記, 田畑秀典, 永田浩一
- Organizer
  第64回日本神経化学会大会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 発達障害責任遺伝子Rac3の神経組織における発現解析2021
- Author(s)
  西川将司, 伊東秀記, 野田万理子, 浜田奈々子, 田畑秀典, 永田浩
- Organizer
  第53回日本臨床分子形態学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 知的障害責任分子RAC3の大脳発達における生理機能と分子病態機構の解明2021
- Author(s)
  西川将司
- Organizer
  名古屋大学脳とこころの研究センター第6回拡大ワークショップ
- Invited
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 知的障害責任分子RAC3の大脳発達における生理機能と分子病態機構の解明2021
- Author(s)
  西川将司, 伊東秀記, 田畑秀典, 永田浩一
- Organizer
  第43回神経組織培養研究会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 知的障害責任分子RAC3の大脳発達における生理機能と分子病態機構の解明2021
- Author(s)
  永田浩一, 西川将司
- Organizer
  第53回日本臨床分子形態学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-21K15895
[Presentation] 発達障害責任遺伝子PLEKHG2の神経組織における発現解析2020
- Author(s)
  永田浩一、西川将司、伊東秀記、野田万理子、浜田奈々子、田畑秀典
- Organizer
  日本臨床分子形態学会
- Data Source
  KAKENHI-PROJECT-20K22888

1. NODA Mariko

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
2. 上田浩

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
3. 常浦祐未

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
4. 永田浩一

# of Collaborated Projects: 0 results

# of Collaborated Products: 3 results
5. 伊東秀記

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

NISHIKAWA Masashi 西川 将司

Nishikawa Masashi 西川 将司

Research Projects

Research Products

Co-Researchers

Cdc42異常に基づく小頭症・脳皮質形成異常症を伴う知的障害の病態メカニズム解析Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Physiological functions and pathogenic mechanisms of Rac3, an intellectual disability-associated G protein, in cerebral developmentPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Pathophysiological analyses of PLEKHG2, a responsible gene for neurodevelopmental disorders, during corticogenesisPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] Live Imaging of Migrating Neurons and Glial Progenitors Visualized by in Utero Electroporation2024

Author(s)

Journal Title

DOI

ISBN

Data Source

[Journal Article] A Novel Constitutively Active c.98G>C, p.(R33P) Variant in RAB11A Associated with Intellectual Disability Promotes Neuritogenesis and Affects Oligodendroglial Arborization2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Expression analyses of WAC, a responsible gene for neurodevelopmental disorders, during mouse brain development2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Expression Analyses of Rich2/Arhgap44, a Rho Family GTPase-Activating Protein, during Mouse Brain Development.2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] A missense variant at the RAC1-PAK1 binding site of RAC1 inactivates downstream signaling in VACTERL association2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Impaired Function of PLEKHG2, a Rho-Guanine Nucleotide-Exchange Factor, Disrupts Corticogenesis in Neurodevelopmental Phenotypes.2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Variant-specific changes in RAC3 function disrupt corticogenesis in neurodevelopmental phenotypes2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Gain-of-function p.F28S variant in<i>RAC3</i>disrupts neuronal differentiation, migration and axonogenesis during cortical development, leading to neurodevelopmental disorder2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Expression analyses of Rac3, a Rho family small GTPase, during mouse brain development.2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Pathophysiological Mechanisms in Neurodevelopmental Disorders Caused by Rac GTPases Dysregulation: What’s behind Neuro-RACopathies2021

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Expression Analyses of Mediator Complex Subunit 13-Like: A Responsible Gene for Neurodevelopmental Disorders during Mouse Brain Development2021

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Expression analyses of PLEKHG2, a Rho family-specific guanine nucleotide exchange factor, during mouse brain development2021

NISHIKAWA Masashi 西川将司

Nishikawa Masashi 西川将司