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HASEGAWA Hitoki  長谷川 仁紀

ORCIDConnect your ORCID iD *help
Researcher Number 10454381
Other IDs
Affiliation (Current) 2025: 名古屋大学, 医学系研究科, 講師
Affiliation (based on the past Project Information) *help 2011 – 2014: 名古屋大学, 医学(系)研究科(研究院), 研究員
2012: 名古屋大学, 医学系研究科, 研究員
Review Section/Research Field
Principal Investigator
Neurosurgery / Cerebral neurosurgery
Keywords
Principal Investigator
ホメオタンパク質 / ホメオボックスタンパク質 / EMT / 転移 / 浸潤 / PRRX1 / Notch / ホメオボックス / 神経膠芽腫 / 細胞質分裂 … More / USP54 / 脱ユビキチン化酵素 / 細胞周期 / 細胞分裂 / グリオーマ Less
  • Research Projects

    (2 results)
  • Research Products

    (6 results)
  • Co-Researchers

    (2 People)
  •  Analysis of Homeobox genes and specific peptide for molecular therapy in GlioblastomaPrincipal Investigator

    • Principal Investigator
      HASEGAWA Hitoki
    • Project Period (FY)
      2013 – 2014
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Neurosurgery
    • Research Institution
      Nagoya University
  •  Characterization of USP54 and its splicing variant in glioblastoma cellsPrincipal Investigator

    • Principal Investigator
      HASEGAWA Hitoki
    • Project Period (FY)
      2011 – 2012
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Cerebral neurosurgery
    • Research Institution
      Nagoya University

All 2015 2013 2012

All Journal Article Presentation

  • [Journal Article] Paired related homeobox 1 is associated with the invasive properties of glioblastoma cells.2015

    • Author(s)
      Sugiyama M, Hasegawa H, Ito S, Sugiyama K, Maeda M, Aoki K, Wakabayashi T, Hamaguchi M, Natsume A, Senga T.
    • Journal Title

      Oncol Rep.

      Volume: 33 Issue: 3 Pages: 1123-30

    • DOI

      10.3892/or.2014.3681

    • Peer Reviewed
    • Data Source
      KAKENHI-PLANNED-23107010, KAKENHI-PROJECT-24390341, KAKENHI-PROJECT-25861269
  • [Journal Article] ALX1 induces snail expression to promote epithelial-to-mesenchymal transition and invasion of ovarian cancer cells.2013

    • Author(s)
      Yuan H, Kajiyama H, Ito S, Yoshikawa N, Hyodo T, Asano E, Hasegawa H, Maeda M, Shibata K, Hamaguchi M, Kikkawa F, Senga T.
    • Journal Title

      Cancer Res

      Volume: 73(5) Pages: 1581-90

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23791591
  • [Journal Article] Misshapen-like kinase 1 (MINK1) is a novel component of striatin-interacting phosphatase and kinase (STRIPAK) and is required for the completion of cytokinesis.2012

    • Author(s)
      Hyodo T, Ito S, Hasegawa H, Asano E, Maeda M, Urano T, Takahashi M, Hamaguchi M, Senga T.
    • Journal Title

      J Biol Chem

      Volume: 287(30) Pages: 25019-29

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23791591
  • [Journal Article] Forced expression of miR-143 represses ERK5/c-Myc and p68/p72 signaling in concert with miR-145 in gut tumors of Apc(Min) mice2012

    • Author(s)
      Takaoka Y, Shimizu Y, Hasegawa H, Ouchi Y, Qiao S, Nagahara M, Ichihara M, Lee JD, Adachi K, Hamaguchi M, Iwamoto T
    • Journal Title

      PLoS One

      Volume: 7(8)

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23791591
  • [Presentation] Misshapen-like kinase 1 (MINK1) is a novel component of striatin-interacting phosphatase and kinase (STRIPAK) and is required for the completion of cytokinesis2012

    • Author(s)
      兵頭寿典、長谷川仁紀、千賀威
    • Organizer
      分子生物学会
    • Place of Presentation
      神戸
    • Year and Date
      2012-12-11
    • Data Source
      KAKENHI-PROJECT-23791591
  • [Presentation] PLK1 controls cytokinetic furrowing through phosphorylation of an actin/myosin binding protein, supervillin2012

    • Author(s)
      長谷川仁紀、千賀威
    • Organizer
      分子生物学会
    • Place of Presentation
      神戸
    • Year and Date
      2012-12-11
    • Data Source
      KAKENHI-PROJECT-23791591
  • 1.  SENGA Takeshi
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results
  • 2.  夏目 敦至
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results

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