Fuke Satoshi 福家聡

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FUKE Satoshi 福家聡

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Researcher Number	20422660
Other IDs
Affiliation (based on the past Project Information) *help	2019: 滋賀医科大学, 医学部, 助教 2015 – 2017: 滋賀医科大学, 医学部, 助教 2009: The Institute of Physical and Chemical Research, 精神疾患動態研究チーム, 研究員 2008: The Institute of Physical and Chemical Research, 精神疾患動感研究チーム, 研究員 2006 – 2007: 独立行政法人理化学研究所, 精神疾患動態研究チーム, 研究員 2006: 北陸先端科技大, 研究員
Review Section/Research Field	Principal Investigator Psychiatric science / Neurology / Laboratory animal science / Neurochemistry/Neuropharmacology Except Principal Investigator Neurochemistry/Neuropharmacology
Keywords	Principal Investigator mitochondria / パーキンソン病 / ミトコンドリア / mtDNA / CPEO / BrdU / mood disorder / neuron / mitochondrial DNA / POLG … More / bipolar disorder / 神経科学 / パーキンソン / Polg / Parkin / ミトコンドリアDNA / accummulation mtDNA deletion / aging / bipolar disorde / brain / behavioral activity rhythm / ageing / mtDNA deletion / mitochondrial DNA (mtDNA) / mitochondrial polymerase γ … More Except Principal Investigator 早期胚 / ユビキチン化 / ヒストン / モノユビキチン化 / 運命決定 / 初期発生 / ヒストンユビキチン化 / エピゲノム / 未分化性維持 / モノユビキン化 / ヒストンH2B / ES細胞 / 胚盤胞 / ヒストン修飾 / エピジェネティクス / 神経発生 / 神経幹細胞 Less

変異型mtDNA蓄積がパーキンソン病の分子病態に与える影響についての研究Principal Investigator
- Principal Investigator
  
  福家聡
- Project Period (FY)
  2017
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Neurology
- Research Institution
  Shiga University of Medical Science
Molecular mechanisms of the fate determination of neural stem cells
- Principal Investigator
  
  Hitoshi Seiji
- Project Period (FY)
  2016 – 2018
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  Neurochemistry/Neuropharmacology
- Research Institution
  Shiga University of Medical Science
Analysis of the relationship between an accumulation of mutant mtDNA and the mitochondrial quality control in ParkinsonismPrincipal Investigator
- Principal Investigator
  
  Fuke Satoshi
- Project Period (FY)
  2015 – 2016
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Laboratory animal science
- Research Institution
  Shiga University of Medical Science
Research of the relationship between accumulation of mtDNA deletions and bipolar disorder in an animal modelPrincipal Investigator
- Principal Investigator
  
  FUKE Satoshi
- Project Period (FY)
  2008 – 2009
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Psychiatric science
- Research Institution
  The Institute of Physical and Chemical Research
選択的RNAスプライシング調節因子NSSRの分子機能解析と神経分化に関する研究Principal Investigator
- Principal Investigator
  
  福家聡
- Project Period (FY)
  2006
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Neurochemistry/Neuropharmacology
- Research Institution
  Japan Advanced Institute of Science and Technology
双極性障害とミトコンドリアDNA欠失神経細胞に関する研究Principal Investigator
- Principal Investigator
  
  福家聡
- Project Period (FY)
  2006 – 2007
- Research Category
  
  Grant-in-Aid for Young Scientists (Start-up)
- Research Field
  
  Psychiatric science
- Research Institution
  The Institute of Physical and Chemical Research

All 2019 2017 2016 2015 2009 2008 2007 2006 Other

All Journal Article Presentation Book Other

[Book] 日本臨牀 73巻増刊号　“免疫性神経疾患と神経幹細胞”2015
- Author(s)
  福家　聡
- Total Pages
  9
- Publisher
  日本臨牀社
- Data Source
  KAKENHI-PROJECT-15K21081
[Book] 医学のあゆみ “免疫性神経疾患の病態・治療と神経幹細胞”2015
- Author(s)
  福家　聡
- Total Pages
  6
- Publisher
  医歯薬出版株式会社
- Data Source
  KAKENHI-PROJECT-15K21081
[Journal Article] Minocycline Directly Enhances the Self-Renewal of Adult Neural Precursor Cells2017
- Author(s)
  Kuroda Anri、Fuchigami Takahiro、Fuke Satoshi、Koyama Natsu、Ikenaka Kazuhiro、Hitoshi Seiji
- Journal Title
  
  Neurochemical Research
  
  Volume: 43 Issue: 1 Pages: 219-226
- DOI
  10.1007/s11064-017-2422-6
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-15K08669, KAKENHI-PROJECT-16K14578, KAKENHI-PROJECT-16H04671
[Journal Article] Quantitative analysis of the 4977-bp common deletion of mitochondrial DNA in postmortem frontal cortex from patients with bipolar disorder and schizo phre ni a2008
- Author(s)
  Satoshi Fuke
- Journal Title
  
  Neuroscience Letters 439
  
  Pages: 173-177
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-20790865
[Journal Article] Quantitative analysis of the 4977-bp common deletion of mitochondrial DNA in postmortem frontal cortex from patients with bipolar disorder and schizophrenia2008
- Author(s)
  Satoshi Fuke, Mizue Kametani, Tadafumi Kato
- Journal Title
  
  Neuroscience Letters 439
  
  Pages: 172-177
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-20790865
[Journal Article] Hesrl Knockout mice exhibit behavioral alterations through the dopaminergic nervous system.2006
- Author(s)
  Satoshi Fuke
- Journal Title
  
  Journal of Neuroscience research 84巻7号
  
  Pages: 1555-1563
- Data Source
  KAKENHI-PROJECT-18890237
[Presentation] Association between demethylation and differentiation of neural cells by mammalian GCM1 and GCM2.2019
- Author(s)
  Abdullah A, Hayashi Y, Fuke S, Fuchigami T, Morimura N, Koyama N, Hitoshi S
- Organizer
  ISN-ASN 2019
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16H04671
[Presentation] Glial cells missing 1 promote cell differentiation and angiogenesis by growth factor expression.2019
- Author(s)
  Hayashi Y, Fuke S, Go Y, Abdullah A, Fuchigami T, Morimura N, Koyama N, Hitoshi S
- Organizer
  ISN-ASN 2019
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16H04671
[Presentation] The mitochondrial dysfunction due to the accumulation of mtDNA deletions in the brain causes behavioral abnormalities in heterozygous Polg mutant mice.2017
- Author(s)
  Satoshi Fuke, Yasufumi Shigeyoshi, Jin Nakatani, Saori Yoshida, Ken-ichi Mukaisho, Mie Kubota-Sakashita, Natsu Koyama, Hiroyuki Sugihara, Seiji Hitoshi, and Tadafumi Kato
- Organizer
  第60回日本神経化学会
- Data Source
  KAKENHI-PROJECT-17K09754
[Presentation] 脳における欠失mtDNA蓄積とミトコンドリア機能障害がPolgヘテロ変異マウスの行動異常を誘導する2017
- Author(s)
  福家　聡、重吉　康史、中谷　仁、吉田沙織、向所賢一、窪田‐坂下　美恵、杉原　洋行、等　誠司、加藤　忠史
- Organizer
  日本ミトコンドリア学会　第17回年会
- Data Source
  KAKENHI-PROJECT-17K09754
[Presentation] The local accumulation of mtDNA deletions in the brain and behavioral abnormalities in heterozygous Polg mutant mice.2017
- Author(s)
  Satoshi Fuke, Yasufumi Shigeyoshi, Jin Nakatani, Saori Yoshida, Ken-ichi Mukaisho, Natsu Koyama, Mie Kubota-Sakashita, Hiroyuki Sugihara, Seiji Hitoshi, and Tadafumi Kato
- Organizer
  Euromit 2017
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-17K09754
[Presentation] Functional analysis of Glial cell missing 1 in the mammalian brain.2017
- Author(s)
  Hayashi Y, Fuke S, Fuchigami T, Morimura N, Koyama N, Hitoshi S
- Organizer
  第60回日本神経化学会 (仙台)
- Data Source
  KAKENHI-PROJECT-16H04671
[Presentation] The local accumulation of mtDNA deletions in the brain and behavioral abnormalities in heterozygous Polg mutant mice.2016
- Author(s)
  Satoshi Fuke, Yasufumi Shigeyoshi, Natsu Koyama, Mie Kubota-Sakashita, Seiji Hitoshi, Tadafumi Kato
- Organizer
  The 13th Conference of Asian Society for Mitochondrial Research and Medicine & The 16th Conference of Japanese Society of Mitochondrial Research and Medicine
- Place of Presentation
  Tokyo, Japan
- Year and Date
  2016-10-30
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K21081
[Presentation] A heterozygous mutation of Polg gene causes behavioral abnormalities due to the tissue-specific accumulation of multiple mtDNA deletions in mice.2015
- Author(s)
  Satoshi Fuke
- Organizer
  BMB2015（第38回日本分子生物学会年回　第88回日本生化学会大会）
- Place of Presentation
  神戸
- Year and Date
  2015-12-01
- Data Source
  KAKENHI-PROJECT-15K21081
[Presentation] Accumulation of mitochondrial DNA deletions in the brains of Polg^<D257A> knock-in mice2009
- Author(s)
  Satoshi Fuke
- Organizer
  第32回日本分子生物学会年会
- Place of Presentation
  神奈川県・横浜市
- Year and Date
  2009-12-12
- Data Source
  KAKENHI-PROJECT-20790865
[Presentation] Accumulation of mitochondrial DNA deletions in the brains of PolgD257A knock-in mice2009
- Author(s)
  Satoshi Fuke
- Organizer
  第32回日本分子生物学会年会
- Place of Presentation
  神奈川県
- Year and Date
  2009-12-12
- Data Source
  KAKENHI-PROJECT-20790865
[Presentation] Detection of newly generated mitochondrial DNA in neurons using BrdU (5-bromo-2-deoxyuridine)2008
- Author(s)
  Satoshi Fuke
- Organizer
  Euromito 7
- Place of Presentation
  スウェーデン、ストックホルム
- Year and Date
  2008-06-12
- Data Source
  KAKENHI-PROJECT-20790865
[Presentation] 患者死後脳におけるミトコンドリアDNAの4977bp欠失定量解析2008
- Author(s)
  福家聡
- Organizer
  第27回躁うつ病の薬理・生化学的研究懇話会
- Place of Presentation
  神奈川
- Year and Date
  2008-06-20
- Data Source
  KAKENHI-PROJECT-20790865
[Presentation] Detection of newly generated mitochondrial DNA in neurons using BrdU (5-bromo-2-deoxyuridine)2008
- Author(s)
  Satoshi Fuke
- Organizer
  スウェーデン
- Place of Presentation
  ストックホルム
- Year and Date
  2008-06-12
- Data Source
  KAKENHI-PROJECT-20790865
[Presentation] Quantitative analysis of accumulation of the 4977-bp common deletion of mitochondrial DNA in the postmortem brain from patients with bipolar disorder and schizophrenia2008
- Author(s)
  Satoshi Fuke
- Organizer
  2nd World Federation of Societies of Biological Psychiatry Asia-Pacific Congress
- Place of Presentation
  富山
- Year and Date
  2008-09-12
- Data Source
  KAKENHI-PROJECT-20790865
[Presentation] BrdU(5-bromo-2-deoxyuridine)を用いた神経細胞における新生ミトコンドリアDNAの検出2007
- Author(s)
  福家聡
- Organizer
  第29回日本生物学的精神医学会・第37回日本神経精神薬理学会合同年会
- Place of Presentation
  札幌
- Year and Date
  2007-07-13
- Data Source
  KAKENHI-PROJECT-18890237
[]

1. Hitoshi Seiji (70300895)

# of Collaborated Projects: 1 results

# of Collaborated Products: 3 results
2. 中林一彦 (10415557)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
3. 小山なつ

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

Fuke Satoshi 福家 聡

FUKE Satoshi 福家 聡

Research Projects

Research Products

Co-Researchers

変異型mtDNA蓄積がパーキンソン病の分子病態に与える影響についての研究Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Molecular mechanisms of the fate determination of neural stem cells

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Analysis of the relationship between an accumulation of mutant mtDNA and the mitochondrial quality control in ParkinsonismPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Research of the relationship between accumulation of mtDNA deletions and bipolar disorder in an animal modelPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

選択的RNAスプライシング調節因子NSSRの分子機能解析と神経分化に関する研究Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

双極性障害とミトコンドリアDNA欠失神経細胞に関する研究Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Book] 日本臨牀 73巻増刊号 “免疫性神経疾患と神経幹細胞”2015

Author(s)

Total Pages

Publisher

Data Source

[Book] 医学のあゆみ “免疫性神経疾患の病態・治療と神経幹細胞”2015

Author(s)

Total Pages

Publisher

Data Source

[Journal Article] Minocycline Directly Enhances the Self-Renewal of Adult Neural Precursor Cells2017

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Quantitative analysis of the 4977-bp common deletion of mitochondrial DNA in postmortem frontal cortex from patients with bipolar disorder and schizo phre ni a2008

Author(s)

Journal Title

Data Source

[Journal Article] Quantitative analysis of the 4977-bp common deletion of mitochondrial DNA in postmortem frontal cortex from patients with bipolar disorder and schizophrenia2008

Author(s)

Journal Title

Data Source

[Journal Article] Hesrl Knockout mice exhibit behavioral alterations through the dopaminergic nervous system.2006

Author(s)

Journal Title

Data Source

[Presentation] Association between demethylation and differentiation of neural cells by mammalian GCM1 and GCM2.2019

Author(s)

Organizer

Data Source

[Presentation] Glial cells missing 1 promote cell differentiation and angiogenesis by growth factor expression.2019

Author(s)

Organizer

Data Source

[Presentation] The mitochondrial dysfunction due to the accumulation of mtDNA deletions in the brain causes behavioral abnormalities in heterozygous Polg mutant mice.2017

Author(s)

Organizer

Data Source

Fuke Satoshi 福家聡

FUKE Satoshi 福家聡

[Book] 日本臨牀 73巻増刊号　“免疫性神経疾患と神経幹細胞”2015