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Tsumiyama Ken  積山 賢

ORCIDConnect your ORCID iD *help
Researcher Number 20514607
Other IDs
Affiliation (Current) 2025: 株式会社膠原病研究所, 研究部, 主任研究員
Affiliation (based on the past Project Information) *help 2018 – 2020: 株式会社膠原病研究所, 研究部, 主任研究員
2014 – 2017: 九州大学, 大学病院, 助教
Review Section/Research Field
Principal Investigator
Immunology / Basic Section 48040:Medical biochemistry-related
Keywords
Principal Investigator
小胞体ストレス / 全身性エリテマトーデス(SLE) / 全身性エリテマトーデス / 抗原のクロスプレゼンテーション / CD4 T細胞 / タンパク質分解 / 免疫関連疾患 / 抗原のクロプレゼンテーション / SLE
  • Research Projects

    (3 results)
  • Research Products

    (6 results)
  •  Studies on the induction of pathogenic T cell by endoplasmic reticulum stress and the pathogenesis of systemic lupus erythematosusPrincipal Investigator

    • Principal Investigator
      Tsumiyama Ken
    • Project Period (FY)
      2018 – 2020
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 48040:Medical biochemistry-related
    • Research Institution
      株式会社膠原病研究所
  •  Studies on the pathogenesis of systemic lupus erythematosus mediated by endoplasmic reticulum stress and antigen cross-presentationPrincipal Investigator

    • Principal Investigator
      Tsumiyama Ken
    • Project Period (FY)
      2016 – 2018
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Immunology
    • Research Institution
      株式会社膠原病研究所
      Kyushu University
  •  Studies on the mechanism of the induction of systemic lupus erythematosus mediated via antigen cross-presentationPrincipal Investigator

    • Principal Investigator
      Tsumiyama Ken
    • Project Period (FY)
      2014 – 2015
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Immunology
    • Research Institution
      Kyushu University

All 2019 2018 2016

All Presentation

  • [Presentation] SLEの発症を誘導する自己抗体産生誘導性CD4 T細胞(aiCD4 T細胞)の解析;濾胞性ヘルパーT細胞とIL-21産生CXCR5-ICOShiPD-1hi CD4 T細胞2019

    • Author(s)
      積山賢、塩沢俊一
    • Organizer
      第63回日本リウマチ学会総会・学術集会
    • Data Source
      KAKENHI-PROJECT-18K06933
  • [Presentation] SLE発症の自己臨界点説(SLEが自己臨界点を超える繰り返す抗原刺激の結果発症する)の証明:DOCK8陽性CD4 T細胞=aiCD4 T細胞によるSLEの発症2019

    • Author(s)
      塩沢俊一、積山賢、櫻井恵一、香川英俊、山根隆志、織部元廣、石井宏治、堀内孝彦、吉原良祐、村田美紀、塩沢和子
    • Organizer
      第63回日本リウマチ学会総会・学術集会
    • Data Source
      KAKENHI-PROJECT-18K06933
  • [Presentation] Systemic lupus erythematosus (SLE) is caused by expanded DOCK8-positive autoantibody-inducing CD4 T (aiCD4 T) cell2019

    • Author(s)
      Shiozawa S, Tsumiyama K, Miyazaki Y, Sakurai K, Horiuchi T, Oribe M, Yamane T, Kagawa H, Shiozawa K
    • Organizer
      アメリカリウマチ学会2019
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-18K06933
  • [Presentation] 小胞体ストレスは抗原のクロスプレセンテーションを促進してSLEの組織傷害を引き起こす2018

    • Author(s)
      積山賢、塩沢俊一
    • Organizer
      第61回日本リウマチ学会総会・学術集会
    • Data Source
      KAKENHI-PROJECT-16K21219
  • [Presentation] Endoplasmic reticulum stress induces lupus kidney disease by facilitating antigen cross-presentation via the increase of endosomal Sec61.2016

    • Author(s)
      Tsumiyama K, Shiozawa S.
    • Organizer
      アメリカリウマチ学会2016
    • Place of Presentation
      ワシントンDC(アメリカ合衆国)
    • Year and Date
      2016-11-13
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K21219
  • [Presentation] Endoplasmic reticulum stress facilitates antigen cross-presentation by increasing endosomal Sec61 to generate lupus kidney disease.2016

    • Author(s)
      Tsumiyama K, Shiozawa S.
    • Organizer
      第45回日本免疫学会総会・学術集会
    • Place of Presentation
      沖縄コンベンションセンター(沖縄県・宜野湾市)
    • Year and Date
      2016-12-05
    • Data Source
      KAKENHI-PROJECT-16K21219

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