Onmori Rie 御守里絵

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OMMORI RIE 御守里絵

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Researcher Number	20533722
Other IDs
Affiliation (Current)	2025: 奈良県立医科大学, 医学部, 特任助教
Affiliation (based on the past Project Information) *help	2018 – 2022: 奈良県立医科大学, 医学部, 特任助教
Review Section/Research Field	Principal Investigator Basic Section 53050:Dermatology-related
Keywords	Principal Investigator human β-defensin / 抗菌ペプチド / 自然免疫応答 / ざ瘡様皮疹 / 薬疹 / 分子標的治療薬 / EGFR阻害薬

Elucidation of the pathogenic mechanism of acneiform skin eruptions caused by molecular targeted drugsPrincipal Investigator
- Principal Investigator
  
  Ommori Rie
- Project Period (FY)
  2020 – 2022
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 53050:Dermatology-related
- Research Institution
  Nara Medical University
Study of skin innate immune response in cutaneous adverse effects caused by molecular targeted agentsPrincipal Investigator
- Principal Investigator
  
  OMMORI RIE
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 53050:Dermatology-related
- Research Institution
  Nara Medical University

All 2023 2022 2021 2020 2019 2018 2017

All Journal Article Presentation

[Journal Article] Serum Soluble OX40 as a Diagnostic and Prognostic Biomarker for Drug-Induced Hypersensitivity Syndrome/Drug Reaction with Eosinophilia and Systemic Symptoms2022
- Author(s)
  Mitsui Y, Shinkuma S, Nakamura-Nishimura Y, Ommori R, Ogawa K, Miyagawa F, Mori Y, Tohyama M, Asada H
- Journal Title
  
  J Allergy Clin Immunol Pract
  
  Volume: 10 Issue: 2 Pages: 585-565
- DOI
  10.1016/j.jaip.2021.10.042
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-19K08797, KAKENHI-PROJECT-19K17811, KAKENHI-PROJECT-20K17357, KAKENHI-PROJECT-21K08306
[Journal Article] Reduced induction of human β-defensins is involved in the pathological mechanism of cutaneous adverse effects caused by epidermal growth factor receptor monoclonal antibodies.2020
- Author(s)
  Ommori R, Nakamura Y, Miyagawa F, Shobatake C, Ogawa K, Koyama F, Sho M, Ota I, Kitahara T, Hontsu S, Muro S, Asada H
- Journal Title
  
  Clin Exp Dermatol
  
  Volume: 45 Issue: 8 Pages: 1055-1058
- DOI
  10.1111/ced.14311
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-19K08797, KAKENHI-PROJECT-20K08573, KAKENHI-PROJECT-20K17357, KAKENHI-PROJECT-18K16037
[Journal Article] Predominant contribution of CD4 T cells to human herpesvirus 6 (HHV-6) load in the peripheral blood of patients with drug-induced hypersensitivity syndrome and persistent HHV-6 Infection.2018
- Author(s)
  Miyagawa F, Nakamura Y, Ommori R, Miyashita K, Iioka H, Miyashita N, Nishikawa M, Himuro Y, Ogawa K, Asada H
- Journal Title
  
  Acta Derm Venereol
  
  Volume: 98 Issue: 1 Pages: 146-148
- DOI
  10.2340/00015555-2791
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17K10250, KAKENHI-PROJECT-15K09779, KAKENHI-PROJECT-18K16037
[Journal Article] Serum thymus and activation-regulated chemokine is associated with the severity of drug reaction with eosinophilia and systemic symptoms/drug-induced hypersensitivity syndrome.2018
- Author(s)
  Nakamura-Nishimura Y, Miyagawa F, Miyashita K, Ommori R, Azukizawa H, Asada H
- Journal Title
  
  Br J Dermatol
  
  Volume: 178 Issue: 6 Pages: 1430-1432
- DOI
  10.1111/bjd.16132
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17K10250, KAKENHI-PROJECT-18K16037
[Journal Article] Up-regulation of human herpesvirus 6B-derived microRNAs in the serum of patients with drug-induced hypersensitivity syndrome/drug reaction with eosinophilia and systemic symptoms.2018
- Author(s)
  3.Miyashita K, Miyagawa F, Nakamura Y, Onmori R, Azukizawa H, Asada H
- Journal Title
  
  Acta Derm Venereol
  
  Volume: 98 Issue: 6 Pages: 612-613
- DOI
  10.2340/00015555-2925
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17K10250, KAKENHI-PROJECT-18K16037
[Journal Article] EGFR inhibitory monoclonal antibodies and EGFR tyrosine kinase inhibitors have distinct effects on the keratinocyte innate immune response.2017
- Author(s)
  Ommori R, Park K, Miyagawa F, Azukizawa H, Kanno M, Asada H.
- Journal Title
  
  Br J Dermatol
  
  Volume: - Issue: 3 Pages: 796-797
- DOI
  10.1111/bjd.15445
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-26461699, KAKENHI-PROJECT-15K09779, KAKENHI-PROJECT-18K16037
[Presentation] Acneiform eruptions caused by EGFR inhibitors may result from reduced expression of human β-defensin-3 induced by S. epidermidis.2023
- Author(s)
  Rie Ommori, Satoru Shinkuma, Hideo Asada.
- Organizer
  International Societies for Investigative Dermatology Meeting 2023
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] EGFR阻害薬によるブドウ球菌由来のβ-defensin産生阻害とざ瘡様皮疹発症への影響2022
- Author(s)
  御守里絵、西村友紀、宮川史、正畠千夏、小川浩平、新熊悟、浅田秀夫
- Organizer
  第52回日本皮膚免疫アレルギー学会総会学術大会
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] S. epidermidis enhance human β-defensin-3 production via EGFR by inducing TGF-α.2022
- Author(s)
  Rie Ommori, Yuki Nishimura, Fumi Miyagawa, Chinatsu Shobatake, Kohei Ogawa, Satoru Shinkuma, Hideo Asada.
- Organizer
  47th Annual Meeting of JSID
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] EGFR阻害薬によるブドウ球菌由来のβ-defensin産生阻害とざ瘡様皮疹発症への影響2021
- Author(s)
  御守里絵、西村友紀、正畠千夏、小川浩平、宮川史、新熊悟、浅田秀夫
- Organizer
  第51回日本皮膚免疫アレルギー学会総会学術大会
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] Cutaneous adverse events caused by EGFR inhibitors may result from reduced expression of human ß-defensins induced by staphylococci.2021
- Author(s)
  Rie Ommori, Yuki Nishimura, Fumi Miyagawa, Chinatsu Shobatake, Kohei Ogawa, Satoru Shinkuma, Hideo Asada.
- Organizer
  46th Annual Meeting of JSID
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] Reduced induction of human β-defensins is involved in the pathological mechanism of cutaneous adverse effects caused by EGFR monoclonal antibodies.2020
- Author(s)
  Rie Ommori, Yuki Nakamura, Fumi Miyagawa, Chinatsu Shobatake, Kohei Ogawa, Fumikazu Koyama, Masayuki Sho, Ichiro Ota, Tadashi Kitahara, Shigeto Hontsu, Sigeo Muro, Hideo Asada.
- Organizer
  45th Annual Meeting of JSID
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] Reduced induction of human β-defensins is involved in the pathological mechanism of cutaneous adverse effects caused by EGFR monoclonal antibodies2020
- Author(s)
  Rie Ommori, Yuki Nakamura, Fumi Miyagawa, Chinatsu Shobatake, Kohei Ogawa, Fumikazu Koyama, Masayuki Sho, Ichiro Ota, Tadashi Kitahara, Shigeto Hontsu, Sigeo Muro, Hideo Asada.
- Organizer
  45th Annual Meeting of JSID（国際学会）
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16037
[Presentation] 表皮自然免疫応答に着目したEGFR阻害薬による薬疹の病態解明2020
- Author(s)
  御守里絵、西村友紀、正畠千夏、小川浩平、宮川史、浅田秀夫
- Organizer
  第50回日本皮膚免疫アレルギー学会総会学術大会
- Data Source
  KAKENHI-PROJECT-18K16037
[Presentation] 表皮自然免疫応答に着目したEGFR阻害薬による薬疹の病態解明2020
- Author(s)
  御守里絵、西村友紀、正畠千夏、小川浩平、宮川史、浅田秀夫
- Organizer
  第50回日本皮膚免疫アレルギー学会総会学術大会
- Data Source
  KAKENHI-PROJECT-20K17357
[Presentation] Human β-defensins are involved with pathological mechanism of cutaneous adverse effects caused by EGFR inhibitors.2019
- Author(s)
  Rie Ommori, Fumi Miyagawa, Hiroaki Azukizawa, Hideo Asada
- Organizer
  49th ESDR Annual Meeting
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16037
[Presentation] Human β-defensins are involved with pathological mechanism of cutaneous adverse effects caused by EGFR inhibitors.2019
- Author(s)
  Rie Ommori, Fumi Miyagawa, Hiroaki Azukizawa, Hideo Asada
- Organizer
  44th Annual Meeting of the JSID
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16037
[Presentation] EGFR阻害薬による皮膚障害の発症機序2018
- Author(s)
  御守里絵
- Organizer
  第48回日本皮膚免疫アレルギー学会総会学術大会
- Invited
- Data Source
  KAKENHI-PROJECT-18K16037

1. ASADA Hideo

# of Collaborated Projects: 0 results

# of Collaborated Products: 4 results
2. 宮川史

# of Collaborated Projects: 0 results

# of Collaborated Products: 6 results
3. 森康子

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

Onmori Rie 御守 里絵

OMMORI RIE 御守 里絵

Research Projects

Research Products

Co-Researchers

Elucidation of the pathogenic mechanism of acneiform skin eruptions caused by molecular targeted drugsPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Study of skin innate immune response in cutaneous adverse effects caused by molecular targeted agentsPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] Serum Soluble OX40 as a Diagnostic and Prognostic Biomarker for Drug-Induced Hypersensitivity Syndrome/Drug Reaction with Eosinophilia and Systemic Symptoms2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Reduced induction of human β-defensins is involved in the pathological mechanism of cutaneous adverse effects caused by epidermal growth factor receptor monoclonal antibodies.2020

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Predominant contribution of CD4 T cells to human herpesvirus 6 (HHV-6) load in the peripheral blood of patients with drug-induced hypersensitivity syndrome and persistent HHV-6 Infection.2018

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Serum thymus and activation-regulated chemokine is associated with the severity of drug reaction with eosinophilia and systemic symptoms/drug-induced hypersensitivity syndrome.2018

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Up-regulation of human herpesvirus 6B-derived microRNAs in the serum of patients with drug-induced hypersensitivity syndrome/drug reaction with eosinophilia and systemic symptoms.2018

Author(s)

Journal Title

DOI

Data Source

[Journal Article] EGFR inhibitory monoclonal antibodies and EGFR tyrosine kinase inhibitors have distinct effects on the keratinocyte innate immune response.2017

Author(s)

Journal Title

DOI

Data Source

[Presentation] Acneiform eruptions caused by EGFR inhibitors may result from reduced expression of human β-defensin-3 induced by S. epidermidis.2023

Author(s)

Organizer

Data Source

[Presentation] EGFR阻害薬によるブドウ球菌由来のβ-defensin産生阻害とざ瘡様皮疹発症への影響2022

Author(s)

Organizer

Data Source

[Presentation] S. epidermidis enhance human β-defensin-3 production via EGFR by inducing TGF-α.2022

Author(s)

Organizer

Data Source

[Presentation] EGFR阻害薬によるブドウ球菌由来のβ-defensin産生阻害とざ瘡様皮疹発症への影響2021

Author(s)

Organizer

Data Source

[Presentation] Cutaneous adverse events caused by EGFR inhibitors may result from reduced expression of human &#223;-defensins induced by staphylococci.2021

Author(s)

Organizer

Data Source

[Presentation] Reduced induction of human β-defensins is involved in the pathological mechanism of cutaneous adverse effects caused by EGFR monoclonal antibodies.2020

Author(s)

Organizer

Data Source

[Presentation] Reduced induction of human β-defensins is involved in the pathological mechanism of cutaneous adverse effects caused by EGFR monoclonal antibodies2020

Author(s)

Organizer

Data Source

[Presentation] 表皮自然免疫応答に着目したEGFR阻害薬による薬疹の病態解明2020

Author(s)

Organizer

Data Source

[Presentation] 表皮自然免疫応答に着目したEGFR阻害薬による薬疹の病態解明2020

Onmori Rie 御守里絵

OMMORI RIE 御守里絵

[Presentation] Cutaneous adverse events caused by EGFR inhibitors may result from reduced expression of human ß-defensins induced by staphylococci.2021