Ito Takashi 伊藤孝

… Alternative Names

伊藤孝イトウタカシ

ITO Takashi 伊藤孝

Less

Researcher Number	20597124
Other IDs	https://orcid.org/0000-0002-2521-0237
Affiliation (Current)	2024: 国立研究開発法人理化学研究所, 環境資源科学研究センター, 上級研究員
Affiliation (based on the past Project Information) *help	2022: 国立研究開発法人理化学研究所, 環境資源科学研究センター, 上級研究員 2020: 国立研究開発法人理化学研究所, 環境資源科学研究センター, 研究員 2013: 千葉大学, 医学部附属病院, 特任助教 2012: 千葉大学, 医学(系)研究科(研究院), 研究員
Review Section/Research Field	Principal Investigator Basic Section 59040:Nutrition science and health science-related / General medical chemistry
Keywords	Principal Investigator 寿命 / 腸内細菌 / mTOR / エネルギー代謝 / 乳酸菌 / ミトコンドリア / 解糖系 / 呼吸 / 乳酸 / ミトコンドリア病 … More / 個体老化 / PAK2 / 老化 / 動脈硬化 / CDC42 / 血管内皮細胞 / 細胞老化 / 慢性炎症 Less

Mechanisms of mitochondrial disease suppression and lifespan extension by lactic acid bacteriaPrincipal Investigator
- Principal Investigator
  
  伊藤孝
- Project Period (FY)
  2020 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 59040:Nutrition science and health science-related
- Research Institution
  Institute of Physical and Chemical Research
Role of CDC42 in pro-inflammatory phenotype of senescent vascular endothelial cellsPrincipal Investigator
- Principal Investigator
  
  ITO Takashi
- Project Period (FY)
  2012 – 2013
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  General medical chemistry
- Research Institution
  Chiba University

All 2022 2021 2020 2013 2012 Other

All Journal Article Presentation Patent

[Journal Article] PKC downregulation upon rapamycin treatment attenuates mitochondrial disease2020
- Author(s)
  Martin-Perez Miguel、Grillo Anthony S.、Ito Takashi K.、Valente Anthony S.、Han Jeehae、Entwisle Samuel W.、Huang Heather Z.、Kim Dayae、Yajima Masanao、Kaeberlein Matt、Vill?n Judit
- Journal Title
  
  Nature Metabolism
  
  Volume: 2 Issue: 12 Pages: 1472-1481
- DOI
  10.1038/s42255-020-00319-x
- Peer Reviewed / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K11553
[Patent] ミトコンドリア機能改善剤2020
- Inventor(s)
  伊藤孝　他4名
- Industrial Property Rights Holder
  伊藤孝　他4名
- Industrial Property Rights Type
  特許
- Filing Date
  2020
- Overseas
- Data Source
  KAKENHI-PROJECT-20K11553
[Patent] ミトコンドリア機能改善剤2020
- Inventor(s)
  伊藤孝、他
- Industrial Property Rights Holder
  伊藤孝、他
- Industrial Property Rights Type
  特許
- Filing Date
  2020
- Overseas
- Data Source
  KAKENHI-PROJECT-20K11553
[Presentation] Lactate inhibits glycolysis and attenuates mitochondrial disease2022
- Author(s)
  Takashi Ito, Haruna Nishimura, Sari Nurmila, Hiroki Kobayashi, Minoru Yoshida
- Organizer
  Cell Symposia: Multifaceted Mitochondri
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K11553
[Presentation] mTOR経路阻害でリー脳症モデルNdufs4 KOマウス寿命を延長する分子機構2021
- Author(s)
  伊藤　孝、小林　大貴、吉田　稔
- Organizer
  J-mit 特別オンラインシンポジウム
- Data Source
  KAKENHI-PROJECT-20K11553
[Presentation] How does inhibition of mTOR promote longevity in mammals?2020
- Author(s)
  Takashi Ito
- Organizer
  8th Symposium of the Smart-Aging Research Center
- Invited
- Data Source
  KAKENHI-PROJECT-20K11553
[Presentation] mTOR経路と腸内細菌がマウス寿命に及ぼす影響2020
- Author(s)
  伊藤　孝
- Organizer
  第43回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-20K11553
[Presentation] CDC42経路が老化血管内皮細胞の炎症誘導を制御する2013
- Author(s)
  伊藤孝, 南野徹
- Organizer
  第13回日本抗加齢医学総会
- Invited
- Data Source
  KAKENHI-PROJECT-24790272
[Presentation] CDC42 and NFκB signaling contribute to pro-inflammatory phenotype of senescent endothelial cells2012
- Author(s)
  Ito T, Minamino T
- Organizer
  41st Annual Meeting of The American Aging Association
- Place of Presentation
  (口頭発表)
- Data Source
  KAKENHI-PROJECT-24790272
[Presentation] CDC42 and NFκB signaling contribute to pro-inflammatory phenotype of senescent endothelial cells.2012
- Author(s)
  伊藤　孝
- Organizer
  Keystone Symposium on Aging and Diseases of Aging (S2)
- Place of Presentation
  東京
- Data Source
  KAKENHI-PROJECT-24790272
[Presentation] CDC42 and NFκB signaling contribute to pro-inflammatory phenotype of senescent endothelial cells2012
- Author(s)
  Ito T, Minamino T
- Organizer
  Keystone Symposium Aging and Diseases of Aging(S2)
- Place of Presentation
  (ポスター発表かつ口頭発表, Travel award受賞)
- Data Source
  KAKENHI-PROJECT-24790272
[Presentation] 老化血管内皮細胞による炎症誘導にCDC42経路とNFκB経路が寄与する2012
- Author(s)
  伊藤孝, 南野徹
- Organizer
  第35回日本分子生物学会年会
- Place of Presentation
  (ポスター発表かつ口頭発表)
- Data Source
  KAKENHI-PROJECT-24790272
[Presentation] CDC42経路が老化血管内皮細胞の炎症誘導を制御する
- Author(s)
  伊藤　孝
- Organizer
  第13回日本抗加齢医学総会
- Place of Presentation
  横浜パシフィコ
- Invited
- Data Source
  KAKENHI-PROJECT-24790272

1. MINAMINO Tohru (90328063)

# of Collaborated Projects: 1 results

# of Collaborated Products: 4 results

Ito Takashi 伊藤 孝

伊藤 孝 イトウ タカシ

ITO Takashi 伊藤 孝

Research Projects

Research Products

Co-Researchers

Mechanisms of mitochondrial disease suppression and lifespan extension by lactic acid bacteriaPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Role of CDC42 in pro-inflammatory phenotype of senescent vascular endothelial cellsPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] PKC downregulation upon rapamycin treatment attenuates mitochondrial disease2020

Author(s)

Journal Title

DOI

Data Source

[Patent] ミトコンドリア機能改善剤2020

Inventor(s)

Industrial Property Rights Holder

Industrial Property Rights Type

Filing Date

Data Source

[Patent] ミトコンドリア機能改善剤2020

Inventor(s)

Industrial Property Rights Holder

Industrial Property Rights Type

Filing Date

Data Source

[Presentation] Lactate inhibits glycolysis and attenuates mitochondrial disease2022

Author(s)

Organizer

Data Source

[Presentation] mTOR経路阻害でリー脳症モデルNdufs4 KOマウス寿命を延長する分子機構2021

Author(s)

Organizer

Data Source

[Presentation] How does inhibition of mTOR promote longevity in mammals?2020

Author(s)

Organizer

Data Source

[Presentation] mTOR経路と腸内細菌がマウス寿命に及ぼす影響2020

Author(s)

Organizer

Data Source

[Presentation] CDC42経路が老化血管内皮細胞の炎症誘導を制御する2013

Author(s)

Organizer

Data Source

[Presentation] CDC42 and NFκB signaling contribute to pro-inflammatory phenotype of senescent endothelial cells2012

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] CDC42 and NFκB signaling contribute to pro-inflammatory phenotype of senescent endothelial cells.2012

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] CDC42 and NFκB signaling contribute to pro-inflammatory phenotype of senescent endothelial cells2012

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 老化血管内皮細胞による炎症誘導にCDC42経路とNFκB経路が寄与する2012

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] CDC42経路が老化血管内皮細胞の炎症誘導を制御する

Author(s)

Organizer

Place of Presentation

Data Source

Ito Takashi 伊藤孝

伊藤孝イトウタカシ

ITO Takashi 伊藤孝