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sasawatari shigemi  笹渡 繁巳

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笹渡 繁巳  ササワタリ シゲミ

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Researcher Number 30450597
Affiliation (Current) 2025: 株式会社メディネット(先端医科学研究所), 先端医科学研究所, 研究員
Affiliation (based on the past Project Information) *help 2010: 独立行政法人国立国際医療研究センター, 消化器疾患研究部・消化器免疫研究室, 研究員
2009: 国立国際医療センター(研究所), 消化器疾患研究部・消化管疾患研究室, 研究員
2008: 国立国際医療センター(研究所), 消化器疾患研究部消化管疾患研究室, 研究員
2007: 国立国際医療センター, (研究所)・消火器疾患研究部消化管疾患研究室, 研究員
Review Section/Research Field
Except Principal Investigator
Experimental pathology / Immunology
Keywords
Except Principal Investigator
抑制性レセプター / 樹状細胞 / TLR / colitis / histidine / lysosome / cytokine / NOD1 / Solute carrier family 15A4 / TLR9 / MHCクラスI / 炎症
  • Research Projects

    (2 results)
  • Research Products

    (14 results)
  • Co-Researchers

    (3 People)
  •  炎症性細胞に特有の新しいTLR9制御機構の解明

    • Principal Investigator
      反町 典子
    • Project Period (FY)
      2009 – 2010
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      Experimental pathology
    • Research Institution
      Research Institute, International Medical Center of Japan
  •  A novel regulatory mechanism of inflammatory responses by inhibitory receptors.

    • Principal Investigator
      SORIMACHI Norio
    • Project Period (FY)
      2007 – 2008
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Immunology
    • Research Institution
      Research Institute, International Medical Center of Japan

All 2011 2010 2009 2008 2007 Other

All Journal Article Presentation

  • [Journal Article] The solute carrier family 15A4 regulates TLR9 and NOD1 functions in the innate immune system and promotes colitis in mice.2011

    • Author(s)
      Sasawatari, S., et al.
    • Journal Title

      Gastroenterology

      Volume: 140 Pages: 1513-1525

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-21390123
  • [Journal Article] Ly49Q plays a crucial role in neutrophil polarization and migration by regulating raft trafficking2010

    • Author(s)
      Sasawatari, S, Yoshizaki, M, Taya C., et al.
    • Journal Title

      Immunity 32

      Pages: 200-213

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-21390123
  • [Journal Article] Spatiotemporal regulation of intracellular trafficking of TLR9 by an inhibitory receptor, Ly49Q.

    • Author(s)
      Yoshizaki M., Tazawa A., Kasumi E., Sasawatari S., Itoh K., Dohi T., Sasazuki T., Inaba K., Makrigiannis A.P. and Toyama-Sorimachi, N.
    • Journal Title

      Blood in press

      Pages: 0-0

    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] 自然免疫制御におけるSLC15A4の重要性2010

    • Author(s)
      笹渡繁巳、反町典子
    • Organizer
      第19回東京免疫フォーラム
    • Place of Presentation
      東京
    • Year and Date
      2010-02-24
    • Data Source
      KAKENHI-PROJECT-21390123
  • [Presentation] A crucial role of Ly49Q in neutrophil polarization and migration by regulating raft trafficking.2010

    • Author(s)
      Sasawatari, S., et al.
    • Organizer
      12^<th> Meeting of the Society for Natural Immunity
    • Place of Presentation
      Croatia
    • Data Source
      KAKENHI-PROJECT-21390123
  • [Presentation] Ly49Q plays a crucial role in neutrophil polarization and migration by regulating raft trafficking.2010

    • Author(s)
      Sasawatari, S., et al.
    • Organizer
      14th International Congress of Immunology
    • Place of Presentation
      神戸
    • Data Source
      KAKENHI-PROJECT-21390123
  • [Presentation] A crucial role of Ly49Q-mediated dynamic regulation of lipid rafts in neutrophils2009

    • Author(s)
      笹渡繁巳
    • Organizer
      第39回日本免疫学会総会・学術集会
    • Place of Presentation
      大阪
    • Data Source
      KAKENHI-PROJECT-21390123
  • [Presentation] Ly49Q is crucial for regulation of plasmacytoid dendritic cell survival2008

    • Author(s)
      Sasawatari S. Toyama-Sorimachi, N., 他
    • Organizer
      第38回日本免疫学会総会・学術集会
    • Place of Presentation
      京都
    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] Regulation of TLR4 signaling by an inhibitory receptor, Ly49Q.2008

    • Author(s)
      Kasumi E., Tazawa A., Yoshizaki M., Sasawatari S., Dohi T., Toyama-Sorimachi, N.
    • Organizer
      第38回日本免疫学会総会・学術集会
    • Place of Presentation
      京都
    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] A crucial role of Ly49Q in TLR9-triggering cytokine production.2008

    • Author(s)
      Yoshizaki M., Tazawa A., Kasumi E., Sasawatari S., Dohi T., Toyama-Sorimachi, N.
    • Organizer
      第38回日本免疫学会総会・学術集会
    • Place of Presentation
      京都
    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] Ly14Q is crucial for regulation of plasmacytoid dendritic cell survival.2008

    • Author(s)
      Sasawatari S., Tazawa A., Yoshizaki M., Dohi T., Inaba K., Toyama-Sorimachi, N.
    • Organizer
      第38回日本免疫学会総会・学術集会
    • Place of Presentation
      京都
    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] A novel regulatory mechanism of recruitment and type I interferon secretion of plasmacytoid dendritic cells.2007

    • Author(s)
      Sasawatari, S., Dohi, T., Inaba, K., Toyama-Sorimachi, N.
    • Organizer
      第37回日本免疫学会総会・学術集会
    • Place of Presentation
      東京
    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] An inhibitory MHC class I receptor, Ly49Q, is crucial for neutrophils polarization and infiltration by regulating cellular distribution of SHP-1.2007

    • Author(s)
      Toyama-Sorimachi, N., Sasawatari, S., Inaba, K., Dohi, T.
    • Organizer
      第37回日本免疫学会総会・学術集会
    • Place of Presentation
      東京
    • Data Source
      KAKENHI-PROJECT-19590507
  • [Presentation] 抑制性MHCレセプターLy49Qによる炎症応答制御と免疫恒常性の維持2007

    • Author(s)
      笹渡繁巳、反町典子
    • Organizer
      第16回東京免疫フォーラム
    • Year and Date
      2007-03-15
    • Data Source
      KAKENHI-PROJECT-19590507
  • 1.  SORIMACHI Norio (30217468)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 0 results
  • 2.  吉崎 真理子 (20517809)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 4 results
  • 3.  遠山 典子
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 9 results

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