ABE WAKANA 阿部若菜

Researcher Number	30457618
Affiliation (based on the past Project Information) *help	2014 – 2016: 大分大学, 医学部, 病院特任助教 2015: 大分大学, 医学部, 助教 2015: 大分大学, 医学部, 特任助教 2013: 大分大学, 医学部, 助教
Review Section/Research Field	Principal Investigator Obstetrics and gynecology Except Principal Investigator Obstetrics and gynecology
Keywords	Principal Investigator micro RNA / microRNA / エピジェネティクス / 子宮内膜症 Except Principal Investigator mTOR / Akt / PI3 kinase / Ras homology A / ゲルコントラクションアッセイ / コラーゲンゲル3次元培養法 … More / statin / mevalonate / Ras homology / 薬物療法 / 瘢痕化 / 子宮内膜症 Less

microRNA is involved in the pathogenesis of endometriosis.Principal Investigator
- Principal Investigator
  
  ABE WAKANA
- Project Period (FY)
  2014 – 2016
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Obstetrics and gynecology
- Research Institution
  Oita University
Pathogenesis and medical treatment of endometriosis-associated fibrosis
- Principal Investigator
  
  Nasu Kaei
- Project Period (FY)
  2013 – 2015
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Obstetrics and gynecology
- Research Institution
  Oita University

All 2014 2013 Other

All Journal Article Presentation

[Journal Article] Death receptor 6 is epigenetically silenced by histone deacetylation in endometriosis and promotes the pathogenesis of endometriosis.2013
- Author(s)
  Kai K, Nasu K, Kawano Y, Aoyagi Y, Tsukamoto Y, Hijiya N, Abe W, Okamoto M, Moriyama M, Narahara H.
- Journal Title
  
  Am J Reprod Immunol
  
  Volume: 70 Issue: 6 Pages: 485-496
- DOI
  10.1111/aji.12155
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23592407, KAKENHI-PROJECT-24591952, KAKENHI-PROJECT-25460419, KAKENHI-PROJECT-25462564
[Journal Article] CCAAT/enhancer-binding protein  is epigenetically silenced by histone deacetylation in endometriosis and promotes the pathogenesis of endometriosis.2013
- Author(s)
  Kawano Y, Nasu K, Hijiya N, Tsukamoto Y, Amada K, Abe W, Kai K, Moriyama M, Narahara H
- Journal Title
  
  J Clin Endocrinol Metab
  
  Volume: 98 Issue: 9 Pages: E1474-E1482
- DOI
  10.1210/jc.2013-1608
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23592407, KAKENHI-PROJECT-25460419, KAKENHI-PROJECT-25462564, KAKENHI-PROJECT-25861500
[Journal Article] miR-196b targets c-myc and Bcl-2 expression, inhibits proliferation and induces apoptosis in endometriotic stromal cells2013
- Author(s)
  Abe W, Nasu K, Nakada C, Kawano Y, Moriyama M, Narahara H
- Journal Title
  
  Hum Reprod
  
  Volume: 28 Issue: 3 Pages: 750-761
- DOI
  10.1093/humrep/des446
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23592407, KAKENHI-PROJECT-24590485, KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜症におけるアポトーシス受容体DR6の発現低下と病態形成への関与2014
- Author(s)
  甲斐健太郎，奈須家栄，川野由紀枝，阿部若菜，岡本真実子，楢原久司
- Organizer
  第35回日本エンドメトリオーシス学会
- Place of Presentation
  鹿児島市・城山観光ホテル
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜症病態形成におけるがん抑制遺伝子C/EBPαの関与についての検討2013
- Author(s)
  川野由紀枝，奈須家栄，阿部若菜，甲斐健太郎，岡本真実子，河野康志，楢原久司
- Organizer
  第65回日本産科婦人科学会学術講演会
- Place of Presentation
  札幌市・ロイトン札幌，ホテルさっぽろ芸文館，札幌プリンスホテル，札幌市教育文化会館
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] Application of the histone deacetylase inhibitors for the treatment of endometriosis: histone modifications as pathogenesis and novel therapeutic target2013
- Author(s)
  Yukie Kawano, Kaei Nasu, Wakana Abe, Yasushi Kawano, Hisashi Narahara
- Organizer
  The 9th Conference of the Pacific Rim Society for Fertility and Sterility
- Place of Presentation
  Kobe, Japan・Kobe International Conference Center
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜症におけるエピジェネティクス異常2013
- Author(s)
  川野由紀枝，奈須家栄，阿部若菜，甲斐健太郎，岡本真実子，河野康志，楢原久司
- Organizer
  第45回日本臨床分子形態学会学術講演会
- Place of Presentation
  福岡市・アクロス福岡
- Invited
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜症間質細胞におけるmiR-196bの発現低下は子宮内膜症の病態形成に関与する2013
- Author(s)
  奈須家栄，阿部若菜，川野由紀枝，甲斐健太郎，岡本真実子，河野康志，楢原久司
- Organizer
  第65回日本産科婦人科学会学術講演会
- Place of Presentation
  札幌市・ロイトン札幌，ホテルさっぽろ芸文館，札幌プリンスホテル，札幌市教育文化会館
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] miR-196b targets c-myc and Bcl-2 expression, inhibits proliferation and incuces apoptosis in endometriotic stromal cells2013
- Author(s)
  Wakana Abe, Kaei Nasu, Chisato Nakada, Yukie Kawano, Masatsugu Moriyama, Hisashi Narahara
- Organizer
  The 9th Conference of the Pacific Rim Society for Fertility and Sterility
- Place of Presentation
  Kobe, Japan・Kobe International Conference Center
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜症におけるDNAメチル化の異常
- Author(s)
  川野由紀枝，奈須家栄，阿部若菜，甲斐健太郎，河野康志，楢原久司
- Organizer
  第70回九州・沖縄生殖医学会
- Place of Presentation
  福岡市・エルガーラホール
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜間質細胞におけるmiR-196bの発現低下は増殖促進とapoptosis抑制を介して子宮内膜症の病態形成に関与する
- Author(s)
  阿部若菜，奈須家栄，川野由紀枝，河野康志，楢原久司
- Organizer
  第70回九州・沖縄生殖医学会
- Place of Presentation
  福岡市・エルガーラホール
- Data Source
  KAKENHI-PROJECT-25462564
[Presentation] 子宮内膜症間質細胞ではmiR-503の発現が低下し、その増殖促進とapoptosis抵抗性に関与する
- Author(s)
  阿部若菜
- Organizer
  第66回日本産科婦人科学会学術講演会
- Place of Presentation
  東京国際フォーラム（東京都千代田区）
- Year and Date
  2014-04-18 – 2014-04-20
- Data Source
  KAKENHI-PROJECT-26861336

1. Nasu Kaei (30274757)

# of Collaborated Projects: 1 results

# of Collaborated Products: 11 results
2. KAWANO YASUSHI (40274758)

# of Collaborated Projects: 1 results

# of Collaborated Products: 7 results
3. NARAHARA HISASHI (60211447)

# of Collaborated Projects: 1 results

# of Collaborated Products: 11 results
4. KAWANO YUKIE (60516128)

# of Collaborated Projects: 1 results

# of Collaborated Products: 8 results
5. YUGE AKITOSHI (90452911)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
6. 甲斐健太郎 (90457622)

# of Collaborated Projects: 1 results

# of Collaborated Products: 5 results

ABE WAKANA 阿部 若菜

Research Projects

Research Products

Co-Researchers

microRNA is involved in the pathogenesis of endometriosis.Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Pathogenesis and medical treatment of endometriosis-associated fibrosis

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] Death receptor 6 is epigenetically silenced by histone deacetylation in endometriosis and promotes the pathogenesis of endometriosis.2013

Author(s)

Journal Title

DOI

Data Source

[Journal Article] CCAAT/enhancer-binding protein &#61537; is epigenetically silenced by histone deacetylation in endometriosis and promotes the pathogenesis of endometriosis.2013

Author(s)

Journal Title

DOI

Data Source

[Journal Article] miR-196b targets c-myc and Bcl-2 expression, inhibits proliferation and induces apoptosis in endometriotic stromal cells2013

Author(s)

Journal Title

DOI

Data Source

[Presentation] 子宮内膜症におけるアポトーシス受容体DR6の発現低下と病態形成への関与2014

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 子宮内膜症病態形成におけるがん抑制遺伝子C/EBPαの関与についての検討2013

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] Application of the histone deacetylase inhibitors for the treatment of endometriosis: histone modifications as pathogenesis and novel therapeutic target2013

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 子宮内膜症におけるエピジェネティクス異常2013

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 子宮内膜症間質細胞におけるmiR-196bの発現低下は子宮内膜症の病態形成に関与する2013

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] miR-196b targets c-myc and Bcl-2 expression, inhibits proliferation and incuces apoptosis in endometriotic stromal cells2013

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 子宮内膜症におけるDNAメチル化の異常

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 子宮内膜間質細胞におけるmiR-196bの発現低下は増殖促進とapoptosis抑制を介して子宮内膜症の病態形成に関与する

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 子宮内膜症間質細胞ではmiR-503の発現が低下し、その増殖促進とapoptosis抵抗性に関与する

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

ABE WAKANA 阿部若菜

[Journal Article] CCAAT/enhancer-binding protein  is epigenetically silenced by histone deacetylation in endometriosis and promotes the pathogenesis of endometriosis.2013