NAKAMURA TAKANORI 中村貴紀

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NAKAMURA Takanori 中村貴紀

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Researcher Number	30707576
Other IDs	https://orcid.org/0000-0002-2915-2257
Affiliation (Current)	2025: 愛媛大学, プロテオサイエンスセンター, 助教
Affiliation (based on the past Project Information) *help	2024: 愛媛大学, プロテオサイエンスセンター, 特定講師 2023: 愛媛大学, プロテオサイエンスセンター, 講師 2013 – 2022: 東京大学, 医科学研究所, 助教
Review Section/Research Field	Principal Investigator General medical chemistry / Basic Section 52050:Embryonic medicine and pediatrics-related / Basic Section 44010:Cell biology-related / Basic Section 48040:Medical biochemistry-related Except Principal Investigator Pathological medical chemistry
Keywords	Principal Investigator 中心体 / PLK4 / 発育不全 / 小頭症 / 染色体不安定性 / 中心体複製 / 発癌 / シグナル伝達 / 染色体安定性 / SAPK … More / 疾患プロテオーム / プロテオーム解析 / 近位ビオチン標識 / 遺伝子変異 / 中心体輸送 / 胚発生 / がん / 初期発生 / 個体発生 / 中心体蛋白質 / 中心体過剰複製 / 抗癌剤 / 数理解析 / 母中心小体 / PCM / 中心小体 / 不妊 / 男性不妊 / 繊毛病 / 細胞周期 / リン酸化 / ストレス応答MAPK / MAPキナーゼ / MAPK / ストレス応答 / p53 / JNK / p38 … More Except Principal Investigator EMT / 生体分子 / リン酸化 / 発現制御 / 癌 / 遺伝子 / シグナル伝達 / 転写制御 / CtBP / MAPキナーゼ / ERK / がん / エピゲノム制御 Less

初期発生制御分子群の相互作用マップの創出~発育不全病態の包括的理解~Principal Investigator
- Principal Investigator
  
  中村貴紀
- Project Period (FY)
  2024 – 2026
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 52050:Embryonic medicine and pediatrics-related
- Research Institution
  Ehime University
The etiological mechanism of developmental disorder derived from deregulation of centrosomal proteinsPrincipal Investigator
- Principal Investigator
  
  Nakamura Takanori
- Project Period (FY)
  2021 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 44010:Cell biology-related
- Research Institution
  Ehime University
  The University of Tokyo
The mechanism underlying centrosome localization and its application to screening for anticancer agentPrincipal Investigator
- Principal Investigator
  
  Nakamura Takanori
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 48040:Medical biochemistry-related
- Research Institution
  The University of Tokyo
SAPKs and PLK4 maintain the numerical integrity of centrosomesPrincipal Investigator
- Principal Investigator
  
  Nakamura Takanori
- Project Period (FY)
  2015 – 2016
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  General medical chemistry
- Research Institution
  The University of Tokyo
Regulation of gene expression by novel ERK substrates and its failure in cancer
- Principal Investigator
  
  Takekawa Mutsuhiro
- Project Period (FY)
  2015 – 2017
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  Pathological medical chemistry
- Research Institution
  The University of Tokyo
Molecular mechanisms that maintain centrosome integrity by SAPK and mechanisms that regulate PLK4 localization to centrosomes.Principal Investigator
- Principal Investigator
  
  NAKAMURA Takanori
- Project Period (FY)
  2013 – 2014
- Research Category
  
  Grant-in-Aid for Research Activity Start-up
- Research Field
  
  General medical chemistry
- Research Institution
  The University of Tokyo

All 2023 2022 2020 2019 2018 2017 2016 2015 2014 2013 Other

All Journal Article Presentation

[Journal Article] Stress granule formation inhibits stress-induced apoptosis by selectively sequestering executioner caspases2023
- Author(s)
  Daichi Fujikawa, Takanori Nakamura, Daisuke Yoshioka, Zizheng Li, Hisashi Moriizumi, Mari Taguchi, Noriko Tokai-Nishizumi, Hiroko Kozuka-Hata, Masaaki Oyama, and Mutsuhiro Takekawa
- Journal Title
  
  Current Biology
  
  Volume: 33 Issue: 10 Pages: 1-15
- DOI
  10.1016/j.cub.2023.04.012
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-21K06147, KAKENHI-PLANNED-21H05280, KAKENHI-PROJECT-22H04922, KAKENHI-PROJECT-21H02692
[Journal Article] Stress-responsive MTK1 SAPKKK serves as a redox sensor that mediates delayed and sustained activation of SAPKs by oxidative stress2020
- Author(s)
  Matsushita Moe、Nakamura Takanori、Moriizumi Hisashi、Miki Hiroaki、Takekawa Mutsuhiro
- Journal Title
  
  Science Advances
  
  Volume: 6 Issue: 26 Pages: 1-1
- DOI
  10.1126/sciadv.aay9778
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-INTERNATIONAL-16K21739, KAKENHI-ORGANIZER-16H06573, KAKENHI-PLANNED-16H06574, KAKENHI-PROJECT-18H02609, KAKENHI-PROJECT-18K15042, KAKENHI-PROJECT-16H06276, KAKENHI-PROJECT-20H03515
[Journal Article] Stress-responsive MTK1 SAPKKK serves as a redox sensor that mediates delayed and sustained activation of SAPKs by oxidative stress.2020
- Author(s)
  Moe Matsushita, Takanori Nakamura, Hisashi Moriizumi, Hiroaki Miki and Mutsuhiro Takekawa
- Journal Title
  
  Science Advances
  
  Volume: 6
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K15042
[Journal Article] MCRIP1 promotes the expression of lung surfactant proteins by disrupting CtBP-mediated epigenetic silencing.2019
- Author(s)
  Jane S. Weng, Takanori Nakamura, Hisashi Moriizumi, Hiroshi Takano, Ryoji Yao and Mutsuhiro Takekawa
- Journal Title
  
  Communications Biology
  
  Volume: 2 Pages: 227-227
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K15042
[Journal Article] MCRIP1, an ERK substrate, mediates ERK-induced epigenetic gene silencing during epithelial-mesenchymal transition by regulating the co-repressor CtBP2015
- Author(s)
  Kenji Ichikawa, Yuji Kubota, Takanori Nakamura, Jane S. Weng, Taichiro Tomida, Haruo Saito and Mutsuhiro Takekawa
- Journal Title
  
  Molecular Cell
  
  Volume: 58 Issue: 1 Pages: 35-46
- DOI
  10.1016/j.molcel.2015.01.023
- Peer Reviewed / Acknowledgement Compliant / Open Access
- Data Source
  KAKENHI-ORGANIZER-22117001, KAKENHI-PLANNED-22117003, KAKENHI-PROJECT-24247034, KAKENHI-PROJECT-24390079, KAKENHI-PROJECT-25440043, KAKENHI-PROJECT-25650003, KAKENHI-PROJECT-15K19003, KAKENHI-PUBLICLY-15H01205, KAKENHI-PROJECT-15H04703, KAKENHI-PROJECT-15J07710
[Journal Article] MCRIP1, an ERK substrate, mediates ERK-induced gene silencing during epithelial-mesenchymal transition by regulating the co-repressor CtBP.2015
- Author(s)
  Ichikawa K, Kubota Y, Nakamura T, Weng JS, Tomida T, Saito H and Takekawa M.
- Journal Title
  
  Molecular Cell
  
  Volume: 58 Pages: 35-46
- Peer Reviewed / Acknowledgement Compliant / Open Access
- Data Source
  KAKENHI-PROJECT-25893039
[Journal Article] SAPK pathways and p53 cooperatively regulate PLK4 activity and centrosome integrity under stress.2013
- Author(s)
  Nakamura, T.
- Journal Title
  
  Nature Commun.
  
  Volume: 4 Issue: 1 Pages: 1775-1775
- DOI
  10.1038/ncomms2752
- Peer Reviewed
- Data Source
  KAKENHI-ORGANIZER-22117001, KAKENHI-PLANNED-22117003, KAKENHI-PROJECT-24247034, KAKENHI-PROJECT-24390079, KAKENHI-PROJECT-25893039
[Presentation] 中心小体輸送の分子基盤と発育不全疾患におけるその破綻2023
- Author(s)
  中村貴紀, 西住紀子, 河西通, 中澤崇, 森竜樹, 武田洋幸, 鈴木貴, 武川睦寛
- Organizer
  CREST 多細胞領域第 4 回領域会議
- Invited
- Data Source
  KAKENHI-PROJECT-21K06147
[Presentation] 発育不全疾患の発症メカニズムの解明と神経変性疾患治療を目指した取り組み2023
- Author(s)
  中村貴紀
- Organizer
  愛媛大学PROS 第1回合同教員会議ショートセミナー
- Invited
- Data Source
  KAKENHI-PROJECT-21K06147
[Presentation] 中心体複製開始における分子制御機構とがんにおけるその破綻2022
- Author(s)
  中村貴紀 , 渡海紀子 , 中澤崇 , 森竜樹 , 鈴木貴 , 武川睦寛
- Organizer
  第81回日本癌学会学術総会
- Invited
- Data Source
  KAKENHI-PROJECT-21K06147
[Presentation] 非膜型オルガネラを介した生体内恒常性の制御：その破綻に伴う癌および発育不全疾患の発症機構2022
- Author(s)
  中村貴紀
- Organizer
  愛媛大学プロテオサイエンスセンターPROSセミナー
- Invited
- Data Source
  KAKENHI-PROJECT-21K06147
[Presentation] mTORC1経路2022
- Author(s)
  中村貴紀, 名田茂之, 鈴木貴, 岡田雅人
- Organizer
  2022年度数理腫瘍学年末研究会
- Invited
- Data Source
  KAKENHI-PROJECT-21K06147
[Presentation] ストレス応答MAPKは転写因子リン酸化依存的にmiR-Xの発現を抑制する2022
- Author(s)
  渡海紀子 , 中村貴紀 , 武川睦寛
- Organizer
  第81回日本癌学会学術総会
- Data Source
  KAKENHI-PROJECT-21K06147
[Presentation] Mathematical modeling of the recruitment of centriole biogenesis regulators to mothe centrioles in the S-phase2020
- Author(s)
  Takanori Nakamura
- Organizer
  JSPS Core-to-Core Program “Establishing international Research Network of Mathematical Oncology”
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] ストレス顆粒の新規構成因子およびその生理機能の解析2020
- Author(s)
  吉岡大介、中村貴紀、武川睦寛
- Organizer
  第43回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] ストレス顆粒形成を制御する分子機構の解析2019
- Author(s)
  橋本夏葉、中村貴紀、武川睦寛
- Organizer
  第42回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] Mathematical analysis of the spatio-temporal regulation of the SAPK pathway2019
- Author(s)
  森泉寿士、中村貴紀、曺永旻、鈴木貴、武川睦寛
- Organizer
  令和元年医科学研究所研究成果発表会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] Mathematical analysis of the spatio-temporal regulation of SAPK pathway2019
- Author(s)
  Hisashi Moriizumi, Takanori Nakamura, Cho Youngmin, Takashi Suzuki and Mutsuhiro Takekawa
- Organizer
  Core-to-core meeting
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を活用した中心体複製開始を制御する分子機構の解明2019
- Author(s)
  中村貴紀、渡海紀子、中澤嵩、森竜樹、鈴木貴、武川睦寛
- Organizer
  第78回日本癌学会総会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] ストレス応答MAPキナーゼによるmiRNAの発現調節とアポトーシス誘導機構の解明2019
- Author(s)
  渡海紀子、中村貴紀、武川睦寛
- Organizer
  第78回日本癌学会総会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 中心体複製初期に起こるPLK4の中心体輸送機構2019
- Author(s)
  中村貴紀、西住(渡海) 紀子、中澤嵩、森竜樹、鈴木貴、武川睦寛
- Organizer
  第19回日本蛋白質科学年会・第71回日本細胞生物学会合同年次大会
- Invited
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] The molecular mechanism that recruits centriole biogenesis regulators to mother centrioles in centriole duplication phase2019
- Author(s)
  Takanori Nakamura, Nishizumi-Tokai Noriko, Takashi Nakazawa, Tatsuki Mori, Takashi Suzuki and Mutsuhiro Takekawa
- Organizer
  新学術領域「数理シグナル」第１回国際シンポジウム
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] Mathematical modeling of the recruitment of centriole biogenesis regulators to mother centrioles in centriole duplication phase2019
- Author(s)
  Takanori Nakamura, Nishizumi-Tokai Noriko, Takashi Nakazawa, Tatsuki Mori, Takashi Suzuki and Mutsuhiro Takekawa
- Organizer
  Core-to-core meeting
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を活用したSAPKシグナル時空間制御機構の解明2018
- Author(s)
  森泉寿士、中村貴紀、曺永旻、鈴木貴、武川睦寛
- Organizer
  第41回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] ストレス応答MAPキナーゼp38, JNKによるアポトーシス関連miRNAの発現抑制2018
- Author(s)
  渡海紀子、中村貴紀、武川睦寛
- Organizer
  第77回日本癌学会学術総会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を用いた中心体複製の開始制御機構の解明2018
- Author(s)
  中村貴紀、西住紀子、中澤嵩、森竜樹、鈴木貴、武川睦寛
- Organizer
  新学術領域「数理シグナル」第２回若手ワークショップ
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を駆使した中心体複製開始を制御する分子機構の解明2018
- Author(s)
  中村貴紀、西住紀子、中澤嵩、森竜樹、鈴木貴、武川睦寛
- Organizer
  第41回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を活用したSAPKシグナル時空間制御機構の解明2018
- Author(s)
  森泉寿士、中村貴紀、武川睦寛
- Organizer
  第18回東京大学生命科学シンポジウム
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を活用したSAPKシグナル時空間制御機構の解明2018
- Author(s)
  森泉寿士、中村貴紀、曺永旻、鈴木貴、武川睦寛
- Organizer
  新学術領域「数理シグナル」第２回若手ワークショップ
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] Formation of stress granules suppresses apoptosis by sequestering proapoptotic proteins2018
- Author(s)
  Daichi Fujikawa, Takanori Nakamura, Mutsuhiro Takekawa
- Organizer
  平成30年度東京大学医科学研究所研究成果発表会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] ストレス応答MAPキナーゼによるアポトーシス抑制性miRNAの発現制御2018
- Author(s)
  渡海紀子、中村貴紀、武川睦寛
- Organizer
  第41回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 癌抑制遺伝子MKK4のフィードバック・リン酸化の機能解析2018
- Author(s)
  森泉寿士, 中村貴紀, 武川睦寛
- Organizer
  第1回MMDS、医科学研究所、新領域創成科学研究科合同ワークショップ
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] ストレス顆粒を構成する新規因子の同定とその機能解析2018
- Author(s)
  田口真梨、中村貴紀、武川睦寛
- Organizer
  第41回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 数理解析を用いた中心体複製の開始制御機構の解明2018
- Author(s)
  中村貴紀、西住紀子、中澤嵩、森竜樹、鈴木貴、武川睦寛
- Organizer
  日本応用数理学会
- Invited
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 癌抑制遺伝子MKK4のフィードバック・リン酸化の機能解析2018
- Author(s)
  森泉寿士, 中村貴紀, 武川睦寛
- Organizer
  新学術領域数理シグナル第二回公開シンポジウム
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] ストレス顆粒形成によるアポトーシス抑制機構の解明2018
- Author(s)
  藤川大地、中村貴紀、武川睦寛
- Organizer
  第41回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-18K15042
[Presentation] 中心体複製開始を司るPLK4中心体局在機構の解明2017
- Author(s)
  中村貴紀、西住（渡海）紀子、中澤嵩、鈴木貴、武川睦寛
- Organizer
  2017年度生命科学系学会合同年次大会（ConBio2017）
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Comprehensive analysis of stress granule components2017
- Author(s)
  Daichi Fujikawa, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  CIMR(Cambridge Institute for Medical Research)-University of Tokyo Symposium 2017
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] The molecular mechanisms that maintain the numerical integrity of centrosomes under stress2017
- Author(s)
  Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  EMBO Conference “Centrosomes and Spindle Pole Bodies”
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of a novel ERK substrate, MCRIP12017
- Author(s)
  Jane Weng, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  12th International Symposium of the institute Network
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Identification of a novel redox-sensor that mediates delayed activation of stress-responsive MAPKs by oxidative stress2017
- Author(s)
  Moe Matsushita, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  IMSUT G2リトリート
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of a novel ERK substrate, MCRIP12017
- Author(s)
  Jane Weng, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  24th East Asia Joint Symposium on Biomedical Research
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] 新規ERK基質分子MCRIP1の生理機能解析2017
- Author(s)
  Jane Weng、中村貴紀、武川睦寛
- Organizer
  2017年度生命科学系学会合同年次大会（ConBio2017）
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Comprehensive analysis of stress granule components2017
- Author(s)
  Daichi Fujikawa, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  Dundee-Tokyo Symposium 2017
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of feedback-phosphorylation of MKK4 by MAPKs2017
- Author(s)
  Hisashi Moriizumi, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  第76回日本癌学会学術総会
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of feedback-phosphorylation of MKK4 by MAPKs2017
- Author(s)
  Hisashi Moriizumi, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  AACR Annual Meeting 2017
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] ストレス応答キナーゼMTK1による新たなストレス感知・応答機構2017
- Author(s)
  松下萌恵、中村貴紀、武川睦寛
- Organizer
  2017年度生命科学系学会合同年次大会（ConBio2017）
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] ストレス顆粒の形成機構および生理機能の解明2017
- Author(s)
  藤川大地、中村貴紀、武川睦寛
- Organizer
  新学術領域研究　数理シグナル第１回若手ワークショップ
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] ストレス応答MAPK経路によって制御されるmiRNAの機能解析2017
- Author(s)
  渡海紀子、中村貴紀、武川睦寛
- Organizer
  2017年度生命科学系学会合同年次大会（ConBio2017）
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] 癌抑制遺伝子MKK4のフィードバック・リン酸化の機能解析2017
- Author(s)
  森泉寿士、中村貴紀、武川睦寛
- Organizer
  新学術領域研究　数理シグナル第１回若手ワークショップ
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Identification of a novel redox-sensor that mediates activation of stress-responsive MAPK pathways2016
- Author(s)
  Moe Matsushita, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  医科学研究所創立記念研究発表会
- Place of Presentation
  東京大学（東京）
- Year and Date
  2016-06-04
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] The molecular mechanisms that maintain the numerical integrity of centrosomes2016
- Author(s)
  Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  American Society of Cell Biology (ASCB) annual meeting
- Place of Presentation
  San Francisco (USA)
- Year and Date
  2016-12-03
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of feedback-phosphorylation of MKK4 by MAPKs2016
- Author(s)
  Moriizumi Hisashi, Takanori Nakamura and Mutsuhito Takekawa
- Organizer
  IARU International Symposium on Aging, Longevity and Health
- Place of Presentation
  東京大学（東京）
- Year and Date
  2016-09-26
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of feedback-phosphorylation of MKK4 by MAPKs2016
- Author(s)
  Hisashi Moriizumi, Takanori Nakamura and Mutsuhito Takekawa
- Organizer
  IARU International Symposium on Aging, Longevity and Health
- Place of Presentation
  東京
- Year and Date
  2016-11-04
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] ストレス応答MAPK経路依存的に発現調節されるmiRNAの同定2016
- Author(s)
  西住（渡海）紀子、中村貴紀、武川睦寛
- Organizer
  第39回日本分子生物学会年会
- Place of Presentation
  パシフィコ横浜（横浜）
- Year and Date
  2016-11-30
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of feedback-phosphorylation of MKK4 by MAPKs2016
- Author(s)
  Moriizumi Hisashi, Takanori Nakamura and Mutsuhiro Takekawa
- Organizer
  第75回日本癌学会学術総会
- Place of Presentation
  パシフィコ横浜（横浜）
- Year and Date
  2016-10-06
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Functional analysis of feedback-phosphorylation of MKK4 by MAPKs2016
- Author(s)
  Hisashi Moriizumi, Takanori Nakamura and Mutsuhito Takekawa
- Organizer
  第７５回日本癌学会学術総会
- Place of Presentation
  横浜
- Year and Date
  2016-10-06
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] The molecular mechanisms that maintain the numerical integrity of centrosomes.2016
- Author(s)
  Takanori Nakamura and Mutsuhito Takekawa
- Organizer
  American Society of Cell Biology Annual meeting 2016
- Place of Presentation
  San Franscisco, USA
- Year and Date
  2016-12-03
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] ストレス応答MAPK経路依存的に発現調節されるmiRNAの同定2016
- Author(s)
  西住（渡海）紀子、中村貴紀、武川睦寛
- Organizer
  日本分子生物学会
- Place of Presentation
  横浜
- Year and Date
  2016-11-30
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] 癌抑制遺伝子MKK4とp53による中心体数および染色体安定性の保持機構と癌におけるその破綻2015
- Author(s)
  中村貴紀、武川睦寛
- Organizer
  第52回日本臨床分子医学会
- Place of Presentation
  みやこめっせ（京都）
- Year and Date
  2015-04-10
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Tumour suppressors MKK4 and p53 cooperatively maintain centrosome integrity and chromosomal stability2015
- Author(s)
  Takanori Nakamura, Noriko Nishizumi-Tokai, Eriko Mikoshi, Moe Matsushita, Mutsuhiro Takekawa
- Organizer
  日本癌学会
- Place of Presentation
  名古屋
- Year and Date
  2015-10-08
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] The Functional Analysis of a novel ERK Substrate, MCRIP12015
- Author(s)
  Jane S. Weng, Takanori Nakamura, Mutsuhiro Takekawa
- Organizer
  日本分子生物学会
- Place of Presentation
  神戸
- Year and Date
  2015-12-01
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] 癌抑制遺伝子MKK4のフィードバック・リン酸化の意義2015
- Author(s)
  森泉寿士、中村貴紀、武川睦寛
- Organizer
  日本細胞生物学会
- Place of Presentation
  東京
- Year and Date
  2015-06-30
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] Identification of a novel redox-sensor that mediates activation of stress-responsive MAPK pathways2015
- Author(s)
  Matsushita M, Nakamura T, and Takekawa M
- Organizer
  OIST-IMSUT Student Exchange Event 2015
- Place of Presentation
  東京大学（東京）
- Year and Date
  2015-08-27
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] 新規ERK基質分子MCRIP1の生理機能解析2015
- Author(s)
  Jane S. Weng、中村貴紀、武川睦寛
- Organizer
  BMB2015（日本分子生物学会・生化学会合同総会）
- Place of Presentation
  神戸ポートピアホテル（神戸）
- Year and Date
  2015-12-01
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] 癌抑制遺伝子MKK4のフィードバック・リン酸化の意義2015
- Author(s)
  森泉寿士、中村貴紀、武川睦寛
- Organizer
  第67回日本細胞生物学会大会
- Place of Presentation
  タワーホール船堀（東京）
- Year and Date
  2015-06-30
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] 癌抑制遺伝子MKK4とp53による中心体数および染色体安定性の保持機構と癌におけるその破綻2015
- Author(s)
  中村貴紀、武川睦寛
- Organizer
  日本臨床分子医学会
- Place of Presentation
  京都
- Year and Date
  2015-04-10
- Invited
- Data Source
  KAKENHI-PROJECT-15K19003
[Presentation] Tumor suppressors MKK4 and p53 cooperatively maintain centrosome integrity and chromosomal stability2015
- Author(s)
  中村貴紀、西住-渡海紀子、見越江里子、松下萌恵、武川睦寛
- Organizer
  第74回日本癌学会学術総会
- Place of Presentation
  名古屋国際会議場（名古屋）
- Year and Date
  2015-10-08
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Molecular mechanisms that maintain centrosome integrity under stress2015
- Author(s)
  Nakamura T, Nishizumi-Tokai N, Mikoshi E, and Takekawa M
- Organizer
  42rd IMSUT Founding Commemorative Symposium
- Place of Presentation
  東京大学（東京）
- Year and Date
  2015-06-01
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] Identification of a novel redox-sensor that mediates activation of stress-responsive MAPK pathways.2015
- Author(s)
  Matsushita M, Nakamura T, and Takekawa M
- Organizer
  22nd East Asia Joint symposium on Biomedical Research
- Place of Presentation
  OIST (沖縄）
- Year and Date
  2015-11-11
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15H04703
[Presentation] ストレス環境下における PLK4、p53、SAPK 経路による中心体保持機構2014
- Author(s)
  中村貴紀、武川睦寛
- Organizer
  日本癌学会
- Place of Presentation
  横浜
- Data Source
  KAKENHI-PROJECT-25893039
[Presentation] Centrosome integrity under stress is maintained by a network of PLK4, p53 and SAPK pathways2014
- Author(s)
  中村貴紀、武川睦寛
- Organizer
  細胞生物学会
- Place of Presentation
  名古屋
- Invited
- Data Source
  KAKENHI-PROJECT-25893039
[Presentation] Centrosome integrity under stress is maintained by a network of PLK4, p53 and SAPK pathways2014
- Author(s)
  中村貴紀、武川睦寛
- Organizer
  日本分子生物学会
- Place of Presentation
  神戸
- Data Source
  KAKENHI-PROJECT-25893039
[Presentation] 癌抑制遺伝子MKK4とp53による中心体数および染色体安定性の保持機構と癌におけるその破綻
- Author(s)
  中村貴紀、武川睦寛
- Organizer
  日本臨床分子医学会
- Place of Presentation
  京都
- Year and Date
  2015-04-10 – 2015-04-11
- Invited
- Data Source
  KAKENHI-PROJECT-25893039

1. Takekawa Mutsuhiro (30322332)

# of Collaborated Projects: 1 results

# of Collaborated Products: 30 results
2. KUBOTA YUJI (70614973)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
3. NISHIZUMI NORIKO (30396882)

# of Collaborated Projects: 1 results

# of Collaborated Products: 4 results
4. ICHIKAWA Kazuhisa

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

NAKAMURA TAKANORI 中村 貴紀

NAKAMURA Takanori 中村 貴紀

Research Projects

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Co-Researchers

初期発生制御分子群の相互作用マップの創出~発育不全病態の包括的理解~Principal Investigator

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The etiological mechanism of developmental disorder derived from deregulation of centrosomal proteinsPrincipal Investigator

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The mechanism underlying centrosome localization and its application to screening for anticancer agentPrincipal Investigator

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SAPKs and PLK4 maintain the numerical integrity of centrosomesPrincipal Investigator

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Regulation of gene expression by novel ERK substrates and its failure in cancer

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Molecular mechanisms that maintain centrosome integrity by SAPK and mechanisms that regulate PLK4 localization to centrosomes.Principal Investigator

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[Journal Article] Stress granule formation inhibits stress-induced apoptosis by selectively sequestering executioner caspases2023

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[Journal Article] Stress-responsive MTK1 SAPKKK serves as a redox sensor that mediates delayed and sustained activation of SAPKs by oxidative stress2020

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[Journal Article] Stress-responsive MTK1 SAPKKK serves as a redox sensor that mediates delayed and sustained activation of SAPKs by oxidative stress.2020

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[Journal Article] MCRIP1 promotes the expression of lung surfactant proteins by disrupting CtBP-mediated epigenetic silencing.2019

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[Journal Article] MCRIP1, an ERK substrate, mediates ERK-induced epigenetic gene silencing during epithelial-mesenchymal transition by regulating the co-repressor CtBP2015

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[Journal Article] MCRIP1, an ERK substrate, mediates ERK-induced gene silencing during epithelial-mesenchymal transition by regulating the co-repressor CtBP.2015

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[Journal Article] SAPK pathways and p53 cooperatively regulate PLK4 activity and centrosome integrity under stress.2013

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[Presentation] 中心小体輸送の分子基盤と発育不全疾患におけるその破綻2023

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[Presentation] 発育不全疾患の発症メカニズムの解明と神経変性疾患治療を目指した取り組み2023

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NAKAMURA TAKANORI 中村貴紀

NAKAMURA Takanori 中村貴紀

[Presentation] 非膜型オルガネラを介した生体内恒常性の制御：その破綻に伴う癌および発育不全疾患の発症機構2022