Nibe Yoichi 仁部洋一

Researcher Number	30793351
Affiliation (Current)	2025: 東京科学大学, 総合研究院, プロジェクト助教
Affiliation (based on the past Project Information) *help	2024: 東京医科歯科大学, 高等研究院, プロジェクト助教 2022: 東京医科歯科大学, 難治疾患研究所, 助教 2021: 東京医科歯科大学, 難治疾患研究所, プロジェクト助教 2019 – 2020: 東京医科歯科大学, 難治疾患研究所, 助教
Review Section/Research Field	Principal Investigator Basic Section 53010:Gastroenterology-related
Keywords	Principal Investigator 炎症性腸疾患 / GOMED / 新規オートファジー / 腸炎 / オートファジー / ユビキチン / 単一遺伝子 / サイトカイン / ユビキチンリガーゼ / ユビキチン修飾

ゴルジ体ストレス応答制御機構の炎症性腸疾患における病態生理学的役割Principal Investigator
- Principal Investigator
  
  仁部洋一
- Project Period (FY)
  2024 – 2026
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 53010:Gastroenterology-related
- Research Institution
  Tokyo Medical and Dental University
The role of alternative autophagy-related ubiquitin ligase in the pathogenesis of colitisPrincipal Investigator
- Principal Investigator
  
  Nibe Yoichi
- Project Period (FY)
  2021 – 2022
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 53010:Gastroenterology-related
- Research Institution
  Tokyo Medical and Dental University
The role of alternative-autophagy related proteins in pathophysiology of colitis .Principal Investigator
- Principal Investigator
  
  Nibe Yoichi
- Project Period (FY)
  2019 – 2020
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 53010:Gastroenterology-related
- Research Institution
  Tokyo Medical and Dental University

All 2022 2020

All Journal Article

[Journal Article] Zranb1-mutant mice display abnormal colonic mucus production and exacerbation of DSS-induced colitis.2022
- Author(s)
  Tamura A, Ito G, Matsuda H, Nibe-Shirakihara Y, Hiraoka Y, Kitagawa S, Hiraguri Y, Nagata S, Aonuma E, Otsubo K, Nemoto Y, Nagaishi T, Watanabe M, Okamoto R, Oshima S.
- Journal Title
  
  Biochem Biophys Res Commun.
  
  Volume: 628 Pages: 147-154
- DOI
  10.1016/j.bbrc.2022.08.046
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-22K07978, KAKENHI-PROJECT-21KK0146, KAKENHI-PROJECT-22H00465, KAKENHI-PROJECT-22H00472, KAKENHI-PROJECT-20H03658, KAKENHI-PROJECT-21H02897, KAKENHI-PROJECT-20K08304, KAKENHI-PROJECT-21K15968
[Journal Article] Nickel particles are present in Crohn's disease tissue and exacerbate intestinal inflammation in IBD susceptible mice.2022
- Author(s)
  Hiroki Matsuda, Yoichi Nibe-Shirakihara, Akiko Tamura, Emi Aonuma, Satoko Arakawa, Kana Otsubo, Yasuhiro Nemoto, Takashi Nagaishi, Kiichiro Tsuchiya, Shigeomi Shimizu, Averil Ma, Mamoru Watanabe, Motohiro Uo, Ryuichi Okamoto, Shigeru Oshima.
- Journal Title
  
  Biochemical and Biophysical Research Communications.
  
  Volume: 592 Pages: 74-80
- DOI
  10.1016/j.bbrc.2021.12.111
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-19K08364, KAKENHI-PROJECT-19K08388, KAKENHI-PROJECT-19K08438, KAKENHI-PROJECT-20K21597, KAKENHI-PROJECT-20H00467, KAKENHI-PLANNED-17H06414, KAKENHI-PUBLICLY-20H05314, KAKENHI-PROJECT-19H01050, KAKENHI-PROJECT-19H03634, KAKENHI-PROJECT-20H03658, KAKENHI-PROJECT-21H02897, KAKENHI-PROJECT-21H02898, KAKENHI-PROJECT-20K08304, KAKENHI-PUBLICLY-21H05743, KAKENHI-PROJECT-22H00472, KAKENHI-PROJECT-20K07353, KAKENHI-PROJECT-21K15968, KAKENHI-WRAPUP-22H04899, KAKENHI-PUBLICLY-22H04639
[Journal Article] Receptor-Interacting Protein Kinase 3 (RIPK3) inhibits autophagic flux during necroptosis in intestinal epithelial cells.2020
- Author(s)
  Otsubo K, Maeyashiki C, Nibe Y, Tamura A, Aonuma E, Matsuda H, Kobayashi M, Onizawa M, Nemoto Y, Nagaishi T, Okamoto R, Tsuchiya K, Nakamura T, Torii S, Itakura E, Watanabe M, Oshima S.
- Journal Title
  
  FEBS letters
  
  Volume: 594 Issue: 10 Pages: 1586-1595
- DOI
  10.1002/1873-3468.13748
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-19K08364, KAKENHI-PROJECT-19K22413, KAKENHI-PROJECT-19H01050, KAKENHI-PROJECT-16H06167, KAKENHI-PROJECT-17K09372, KAKENHI-PROJECT-19K08388, KAKENHI-PROJECT-19K08438, KAKENHI-PROJECT-19K08461, KAKENHI-PROJECT-20K08304, KAKENHI-PROJECT-20H03658, KAKENHI-PROJECT-18K07934, KAKENHI-PROJECT-19K17426, KAKENHI-PROJECT-17H04024, KAKENHI-PROJECT-17K19675
[Journal Article] Nickel ions attenuate autophagy flux and induce transglutaminase 2 (TG2) mediated post-translational modification of SQSTM1/p62.2020
- Author(s)
  Emi Aonuma, Akiko Tamura, Hiroki Matsuda, Takehito Asakawa, Yuriko Sakamaki, Kana Otsubo, Yoichi Nibe, Michio Onizawa, Yasuhiro Nemoto, Takashi Nagaishi, Kiichiro Tsuchiya, Tetsuya Nakamura, Motohiro Uo, Mamoru Watanabe, Ryuichi Okamoto, Shigeru Oshima.
- Journal Title
  
  Biochem Biophys Res Commun.
  
  Volume: 542 Pages: 17-23
- DOI
  10.1016/j.bbrc.2021.01.023
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-19K08364, KAKENHI-PROJECT-19K08388, KAKENHI-PROJECT-19K08438, KAKENHI-PROJECT-19K08461, KAKENHI-PROJECT-20K08304, KAKENHI-PROJECT-19H01050, KAKENHI-PROJECT-18H02791, KAKENHI-PROJECT-20H03658, KAKENHI-PROJECT-18K07934, KAKENHI-PROJECT-19K17426, KAKENHI-PROJECT-17K19675, KAKENHI-PROJECT-21K15968

1. TORII Satoru

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
2. 永石宇治

# of Collaborated Projects: 0 results

# of Collaborated Products: 3 results
3. 渡辺守

# of Collaborated Projects: 0 results

# of Collaborated Products: 2 results
4. 鬼澤道夫

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

Nibe Yoichi 仁部 洋一

Research Projects

Research Products

Co-Researchers

ゴルジ体ストレス応答制御機構の炎症性腸疾患における病態生理学的役割Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The role of alternative autophagy-related ubiquitin ligase in the pathogenesis of colitisPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The role of alternative-autophagy related proteins in pathophysiology of colitis .Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] Zranb1-mutant mice display abnormal colonic mucus production and exacerbation of DSS-induced colitis.2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Nickel particles are present in Crohn's disease tissue and exacerbate intestinal inflammation in IBD susceptible mice.2022

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Receptor-Interacting Protein Kinase 3 (RIPK3) inhibits autophagic flux during necroptosis in intestinal epithelial cells.2020

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Nickel ions attenuate autophagy flux and induce transglutaminase 2 (TG2) mediated post-translational modification of SQSTM1/p62.2020

Author(s)

Journal Title

DOI

Data Source

Nibe Yoichi 仁部洋一