KAWAGISHI Hiroyuki 川岸裕幸

Researcher Number	30819082
Other IDs
Affiliation (Current)	2022: 信州大学, 学術研究院医学系, 助教
Affiliation (based on the past Project Information) *help	2021: 信州大学, 学術研究院医学系, 助教 2019 – 2020: 信州大学, 先鋭領域融合研究群バイオメディカル研究所, 助教 2018: 信州大学, 先鋭領域融合研究群バイオメディカル研究所, 助教(特定雇用)
Review Section/Research Field	Principal Investigator Basic Section 48020:Physiology-related / 0802:Biomedical structure and function and related fields
Keywords	Principal Investigator 心筋細胞 / 最終分化 / マウス / 新生児 / 心臓 / 成熟化 / シグナル伝達 / スクリーニング / 薬剤スクリーニング / ホルモン … More / 分裂増殖 / gp130 / 分裂 / アデノ随伴ウイルス / 液性因子 / 糖代謝 / カテコラミン / アドレナリン Less

Novel physiological function of heart in neonatal hypoglycemiaPrincipal Investigator
- Principal Investigator
  
  川岸裕幸
- Project Period (FY)
  2021 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 48020:Physiology-related
- Research Institution
  Shinshu University
Factors inducing final cardiomyocyte maturation in newborn micePrincipal Investigator
- Principal Investigator
  
  KAWAGISHI Hiroyuki
- Project Period (FY)
  2019 – 2020
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 48020:Physiology-related
- Research Institution
  Shinshu University
マウス個体を用いた心筋細胞の成熟化因子の探索Principal Investigator
- Principal Investigator
  
  川岸裕幸
- Project Period (FY)
  2018 – 2019
- Research Category
  
  Grant-in-Aid for Research Activity Start-up
- Review Section
  
  0802:Biomedical structure and function and related fields
- Research Institution
  Shinshu University

All 2022 2021 2020 2019

All Journal Article Presentation

[Journal Article] βアレスチンバイアスAT1受容体アゴニストは新しい乳幼児心不全治療薬の有力候補である。2021
- Author(s)
  川岸裕幸, 山田充彦
- Journal Title
  
  医学のあゆみ
  
  Volume: 276 Pages: 1137-1138
- Data Source
  KAKENHI-PROJECT-19K16488
[Journal Article] β-Arrestin-Biased AT 1 Agonist TRV027 Causes a Neonatal-Specific Sustained Positive Inotropic Effect Without Increasing Heart Rate2020
- Author(s)
  Kashihara T, Kawagishi H, Nakada T, Numaga-Tomita T, Kadota S, Wolf EE, Du CK, Shiba Y, Morimoto S, Yamada M.
- Journal Title
  
  JACC: Basic to Translational Science
  
  Volume: 5 Pages: 1057-1057
- DOI
  10.1016/j.jacbts.2020.08.011
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-19K07116, KAKENHI-PROJECT-19K07299, KAKENHI-PROJECT-18K06870, KAKENHI-PROJECT-19K16488
[Presentation] Rapid increase in aerobic glycolysis in neonatal mouse ventricular cardiomyocytes in response to acute β-adrenergic stimulation2022
- Author(s)
  Hiroyuki Kawagishi，Tsutomu Nakada，Takuro Tomita-Numaga，Mitsuhiko Yamada
- Organizer
  第95回日本薬理学会年会
- Data Source
  KAKENHI-PROJECT-21K06780
[Presentation] 新たなAT1アンジオテンシン受容体修飾法による小児心不全治療法の開発2021
- Author(s)
  川岸裕幸、冨田(沼賀)拓郎、中田勉、柏原俊英、山田充彦
- Organizer
  第94回日本薬理学会年会シンポジウム
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] アンジオテンシンII 1型受容体ーβアレスチン経路を活性化する小児心不全治療薬の開発2020
- Author(s)
  川岸裕幸、柏原俊英、冨田(沼賀)拓郎、中田勉、山田充彦
- Organizer
  第30回日本循環薬理学会
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] Physiological role of gp130 receptor in newborn mouse cardiomyocyte development2020
- Author(s)
  Hiroyuki KAWAGISHI, Tsutomu NAKADA, Takuro NUMAGA-TOMITA, Mitsuhiko YAMADA
- Organizer
  第93回日本薬理学会年会（誌上開催）
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] βアレスチンバイアスアンジオテンシン１型受容体アゴニストは、新生児特有の持続性陽性変力作用を示す2020
- Author(s)
  川岸裕幸、柏原俊英、冨田(沼賀)拓郎、中田勉、山田充彦
- Organizer
  第142回日本薬理学会関東部会
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] 新たな方法でAT1受容体を利用した小児心不全治療薬の開発2020
- Author(s)
  川岸裕幸、冨田(沼賀)拓郎、中田勉、山田充彦
- Organizer
  第143回日本薬理学会関東部会シンポジウム
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] Postnatal loss of gp130 caused impaired cardiac contractility accompanied by inhibition of physiological proliferation of cardiomyocytes2020
- Author(s)
  Hiroyuki Kawagishi, Tsutomu Nakada, Takuro Numaga-Tomita, Mitsuhiko Yamada
- Organizer
  生理研心血管国際集会2020
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] マウス心筋細胞の生下後の機能的最終分化の分子メカニズムの解析2019
- Author(s)
  川岸裕幸、中田勉、冨田（沼賀）拓郎、山田充彦
- Organizer
  第140回日本薬理学会関東部会
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] 新生児マウスにおける、gp130受容体を介した心筋細胞の発達機構2019
- Author(s)
  川岸裕幸、Maite LARRA AGA、冨田（沼賀）拓郎、中田勉、山田充彦
- Organizer
  筋生理の集い
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] Physiological role of gp130 receptor in newborn mouse cardiomyocyte development2019
- Author(s)
  Hiroyuki Kawagishi, Tsutomu Nakada, Takuro Numaga-Tomita, Mitsuhiko Yamada
- Organizer
  The 50th NIPS International Symposium 'MIRACLES' in Cardiovascular Physiology
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-19K16488
[Presentation] Factors inducing final cardiomyocyte maturation in newborn mice2019
- Author(s)
  川岸裕幸、中田勉、山田充彦
- Organizer
  第92回　日本薬理学会年会
- Data Source
  KAKENHI-PROJECT-18H06124

1. 冨田拓郎

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
2. 中田勉

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
3. 山田充彦

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

KAWAGISHI Hiroyuki 川岸 裕幸

Research Projects

Research Products

Co-Researchers

Novel physiological function of heart in neonatal hypoglycemiaPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Factors inducing final cardiomyocyte maturation in newborn micePrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

マウス個体を用いた心筋細胞の成熟化因子の探索Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] βアレスチンバイアスAT1受容体アゴニストは新しい乳幼児心不全治療薬の有力候補である。2021

Author(s)

Journal Title

Data Source

[Journal Article] β-Arrestin-Biased AT 1 Agonist TRV027 Causes a Neonatal-Specific Sustained Positive Inotropic Effect Without Increasing Heart Rate2020

Author(s)

Journal Title

DOI

Data Source

[Presentation] Rapid increase in aerobic glycolysis in neonatal mouse ventricular cardiomyocytes in response to acute β-adrenergic stimulation2022

Author(s)

Organizer

Data Source

[Presentation] 新たなAT1アンジオテンシン受容体修飾法による小児心不全治療法の開発2021

Author(s)

Organizer

Data Source

[Presentation] アンジオテンシンII 1型受容体ーβアレスチン経路を活性化する小児心不全治療薬の開発2020

Author(s)

Organizer

Data Source

[Presentation] Physiological role of gp130 receptor in newborn mouse cardiomyocyte development2020

Author(s)

Organizer

Data Source

[Presentation] βアレスチンバイアスアンジオテンシン１型受容体アゴニストは、新生児特有の持続性陽性変力作用を示す2020

Author(s)

Organizer

Data Source

[Presentation] 新たな方法でAT1受容体を利用した小児心不全治療薬の開発2020

Author(s)

Organizer

Data Source

[Presentation] Postnatal loss of gp130 caused impaired cardiac contractility accompanied by inhibition of physiological proliferation of cardiomyocytes2020

Author(s)

Organizer

Data Source

[Presentation] マウス心筋細胞の生下後の機能的最終分化の分子メカニズムの解析2019

Author(s)

Organizer

Data Source

[Presentation] 新生児マウスにおける、gp130受容体を介した心筋細胞の発達機構2019

Author(s)

Organizer

Data Source

[Presentation] Physiological role of gp130 receptor in newborn mouse cardiomyocyte development2019

Author(s)

Organizer

Data Source

[Presentation] Factors inducing final cardiomyocyte maturation in newborn mice2019

Author(s)

Organizer

Data Source

KAWAGISHI Hiroyuki 川岸裕幸