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OKADA Hirokazu  岡田 浩一

ORCIDConnect your ORCID iD *help
… Alternative Names

岡田 浩一  オカダ ヒロカズ

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Researcher Number 60233342
Other IDs
Affiliation (Current) 2025: 埼玉医科大学, 医学部, 教授
Affiliation (based on the past Project Information) *help 2013 – 2024: 埼玉医科大学, 医学部, 教授
2010 – 2012: 埼玉医科大学, 医学部, 准教授
2004: Saitama Medical School, Department of Nephrology, Associate Professor, 医学部, 助教授
2001 – 2003: 埼玉医科大学, 医学部, 講師
1993: 慶應義塾大学, 医学部, 助手
Review Section/Research Field
Principal Investigator
Kidney internal medicine / Basic Section 53040:Nephrology-related / Kidney internal medicine
Except Principal Investigator
Kidney internal medicine / Basic Section 53040:Nephrology-related / Kidney internal medicine / Urology / 膠原病・アレルギー・感染症内科学
Keywords
Principal Investigator
腎線維化 / 線維芽細胞 / 尿細管上皮細胞 / 線維化 / 細胞周期 / 慢性腎臓病 / 急性腎障害 / アポトーシス / CCN2 / 遺伝子改変動物 … More / 増殖因子 / 形質転換 / 慢性腎臓病(CKD) / 急性腎障害(AKI) / カスパーゼ / gene therapy / fibroblast / fibrosis / Rapidly progressive glomerulo-nephritis / gancyclovir / thymidine kinase / トランスジェニックマウス / 間質線維化 / 抗基底膜抗体腎炎 / FSP1 / 腎間質線維化 / 急速進行性糸球体腎炎 / 遺伝子治療 / 急性進行性糸球体腎炎 / ノックインマウス / CKD / 糸球体硬化 / NFkBシグナル / ポドサイト / NFkB / NFkB 経路 / 老化 / 腎臓 / サイトカイン / 拒絶反応 / 移植 … More
Except Principal Investigator
慢性腎臓病 / CCN2 / 腎線維化 / focal adhesion kinase / β-catenin / FAK / 線維化 / 腎臓内科 / 内科 / インテグリン / gene transfection / promoter analysis / Osteopontin / iKB / プロモーター解析 / 遺伝子導入 / オステオポンチン / promoter / transfection / 尿細管上皮 / オステオポンチン(OPN) / 分子標的薬 / 転移性腎癌 / 進行性腎癌 / チロシンキナーゼ阻害薬 / 分子標的治療薬 / 蛋白尿 / 腎癌 / 半月体 / メサンギウム細胞 / 糸球体腎炎 / 治療薬 / TLR4 / ポドサイト / FSP1 / Jagged1 / Th17アジュバントcurdlan / Th2アジュバント / アトピー性皮膚炎 / 腎炎 / NODマウス / Jaggedl / Notch ligands / curdlan / Th17アジュバント / アジュバント / アレルギー学 Less
  • Research Projects

    (13 results)
  • Research Products

    (34 results)
  • Co-Researchers

    (16 People)
  •  CCN2による線維化促進性情報伝達系を標的とした慢性腎臓病の進展抑制療法の開発Principal Investigator

    • Principal Investigator
      岡田 浩一
    • Project Period (FY)
      2024 – 2026
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 53040:Nephrology-related
    • Research Institution
      Saitama Medical University
  •  Development of a method to suppress the severity of chronic kidney disease through elucidation of mechanism of transition from acute kidney injuryPrincipal Investigator

    • Principal Investigator
      Okada Hirokazu
    • Project Period (FY)
      2020 – 2022
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 53040:Nephrology-related
    • Research Institution
      Saitama Medical University
  •  Development of novel therapeutics for chronic and acute kidney injury through the regulation of CCN2 function

    • Principal Investigator
      Inoue Tsutomu
    • Project Period (FY)
      2019 – 2023
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 53040:Nephrology-related
    • Research Institution
      Saitama Medical University
  •  Pathogenesis and treatment of tubular epithelium-mediated AKI to CKD transitionPrincipal Investigator

    • Principal Investigator
      Okada Hirokazu
    • Project Period (FY)
      2016 – 2018
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Saitama Medical University
  •  Development of a new treatment approach for chronic kidney disease by controlling CCN2 function

    • Principal Investigator
      Inoue Tsutomu
    • Project Period (FY)
      2016 – 2019
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Saitama Medical University
  •  Clinical analysis of urinary biomarkers predictive of renal injury due to tyrosine kinase inhibitors for advanced renal cell carcinoma

    • Principal Investigator
      oyama masafumi
    • Project Period (FY)
      2014 – 2016
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Urology
    • Research Institution
      Saitama Medical University
  •  Generation of anti-fibrotic therapy to combat progression of CKD via CCN2 blockadePrincipal Investigator

    • Principal Investigator
      Okada Hirokazu
    • Project Period (FY)
      2013 – 2015
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Saitama Medical University
  •  Novel strategy for the treatment of active nephritis using secreted FSP1

    • Principal Investigator
      IWANO Masayuki
    • Project Period (FY)
      2012 – 2014
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      Kidney internal medicine
    • Research Institution
      University of Fukui
  •  Effects of renal tissue-specific NFkB blockade on aging kidneysPrincipal Investigator

    • Principal Investigator
      OKADA Hirokazu
    • Project Period (FY)
      2010 – 2012
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Saitama Medical University
  •  Qualitative analysis of adjuvant activities and its application to immunity-related diseases

    • Principal Investigator
      MATSUSHITA Sho
    • Project Period (FY)
      2008 – 2010
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      膠原病・アレルギー・感染症内科学
    • Research Institution
      Saitama Medical University
  •  Generation of an anti-fibrotic therapeutic gene using a tubular epithelium-specific expression gene casette and iKB cDNA

    • Principal Investigator
      KANNO Yoshihiko
    • Project Period (FY)
      2002 – 2004
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Saitama Medical School, School of Medicine
  •  Effect of fibroblast-specific suicidal-gene against rapidly progressive glomerulonephritisPrincipal Investigator

    • Principal Investigator
      OKADA Hirokazu
    • Project Period (FY)
      2001 – 2002
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Saitama Medical School
  •  移植腎の拒絶反応におけるサイトカインおよび細胞接着分子の遺伝子発現Principal Investigator

    • Principal Investigator
      岡田 浩一
    • Project Period (FY)
      1993
    • Research Category
      Grant-in-Aid for Encouragement of Young Scientists (A)
    • Research Field
      Kidney internal medicine
    • Research Institution
      Keio University

All 2023 2022 2021 2020 2019 2018 2017 2016 2010 2009 2003 Other

All Journal Article Presentation

  • [Journal Article] Module 4-Deficient CCN2/Connective Tissue Growth Factor Attenuates the Progression of Renal Fibrosis via Suppression of Focal Adhesion Kinase Phosphorylation in Tubular Epithelial Cells2023

    • Author(s)
      Amano Hiroaki、Inoue Tsutomu、Kusano Takeru、Fukaya Daichi、Kosakai Wakako、Okada Hirokazu
    • Journal Title

      Molecular and Cellular Biology

      Volume: 43 Issue: 10 Pages: 515-530

    • DOI

      10.1080/10985549.2023.2253130

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Journal Article] Analysis of the Function of CCN2 in Tubular Epithelium Cells with a Focus on Renal Fibrogenesis2022

    • Author(s)
      Amano Hiroaki、Inoue Tsutomu、Kusano Takeru、Okada Hirokazu
    • Journal Title

      Methods Mol Biol

      Volume: 13 Pages: 411-426

    • DOI

      10.1007/978-1-0716-2744-0_28

    • ISBN
      9781071627433, 9781071627440
    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-19K08731, KAKENHI-PROJECT-20K08614
  • [Journal Article] Tocilizumab-induced immunocomplex glomerulonephritis: a report of two cases2020

    • Author(s)
      Fukaya Daichi、Inoue Tsutomu、Kogure Yuta、Kajiyama Hiroshi、Ishizawa Keisuke、Seto Takeru、Hasegawa Hajime、Mimura Toshihide、Okada Hirokazu
    • Journal Title

      CEN Case Reports

      Volume: 9 Issue: 4 Pages: 318-325

    • DOI

      10.1007/s13730-020-00478-6

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-19K08731, KAKENHI-PROJECT-20K08805
  • [Journal Article] Glomerular solidification is associated with nephritis-related clinical parameters in IgA nephropathy2019

    • Author(s)
      Inoue Tsutomu、Luo Yankun、Seto Takeru、Suzuki Hiromichi、Okada Hirokazu
    • Journal Title

      Renal Failure

      Volume: 41 Issue: 1 Pages: 893-898

    • DOI

      10.1080/0886022x.2019.1665545

    • Peer Reviewed / Open Access / Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-19K08731, KAKENHI-PROJECT-16K09627
  • [Journal Article] Cellular communication network factor 2 (CCN2) promotes the progression of acute kidney injury to chronic kidney disease2019

    • Author(s)
      Inoue Tsutomu、Kusano Takeru、Amano Hiroaki、Nakamoto Hidetomo、Okada Hirokazu
    • Journal Title

      Biochemical and Biophysical Research Communications

      Volume: 517 Issue: 1 Pages: 96-102

    • DOI

      10.1016/j.bbrc.2019.07.024

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-19K08731, KAKENHI-PROJECT-16K09627
  • [Journal Article] 【腎硬化症への対策】 腎硬化症の薬物治療 糖尿病合併例、有蛋白尿例を含む2017

    • Author(s)
      岡田浩一
    • Journal Title

      日本医事新報

      Volume: 4854 Pages: 34-40

    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Journal Article] 【動脈硬化up to date】 腎線維化を考慮したCKD治療戦略 腎線維化はどこまで解明されたか2017

    • Author(s)
      岡田浩一
    • Journal Title

      循環plus

      Volume: 17 Pages: 2-6

    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Journal Article] 腎線維化を考慮したCKD治療戦略 腎線維化はどこまで解明されたか2016

    • Author(s)
      岡田浩一
    • Journal Title

      循環plus.

      Volume: 17 Pages: 4-6

    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Journal Article] CKDの進展抑制と治療 RA系阻害薬を中心とする薬物療法の現状と今後への期待2016

    • Author(s)
      岡田浩一
    • Journal Title

      内科

      Volume: 118 Pages: 83-87

    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Journal Article] 腎機能障害 慢性腎臓病(CKD)2016

    • Author(s)
      岡田浩一
    • Journal Title

      臨床泌尿器科

      Volume: 70 Pages: 150-153

    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Journal Article] Fibroblasts stimulated via HLA-II produce PGE 2 and regulate cytokine production from Th cells.2010

    • Author(s)
      Nahoko Kato-Kogoe, Hideki Ohyama, Fusanori Nishimura, Michio Meguro, Sayuri Yoshizawa, Yuka Okada, Keiji Nakasho, Koji Yamanegi, Naoko Yamada, Masaki Hata, Takehiro Higashi, Nobuyuki Terada, Sho Matsushita
    • Journal Title

      Lab Invest 90

      Pages: 1747-1756

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20591192
  • [Journal Article] Human leukocyte histocompatibility antigen class II-induced cytokines from human gingival fibroblasts promote proliferation of human umbilical vein endothelial cells : potential association with enhanced angiogenesis in chronic periodontal inflammation2009

    • Author(s)
      Okada Y.
    • Journal Title

      J Periodont Res. 44

      Pages: 103-109

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20591192
  • [Journal Article] D1-Like Receptor Antagonist Inhibits IL-17 Expression and Attenuates Crescent Formation in Nephrotoxic Serum Nephritis.2009

    • Author(s)
      Okada,H., Inoue T., Hashimoto K., Suzuki H., Matsushita S.
    • Journal Title

      Am J Nephrol 30

      Pages: 274-279

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20591192
  • [Journal Article] D1-Like Receptor Antagonist Inhibits IL-17 Expression and Attenuates Crescent Formation in Nephrotoxic Serum Nephritis2009

    • Author(s)
      Okada, H.
    • Journal Title

      Am J Nephrol. 30

      Pages: 274-279

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20591192
  • [Journal Article] Dopamine D1-like receptor antagonist, SCH23390, exhibits a preventive effect on diabetes mellitus that occurs naturally in NOD mice2009

    • Author(s)
      Hashimoto, K., Inoue, T., Higashi T., Takei S., Katayama S., Takagi, R., Okada, H., Matsushita S.
    • Journal Title

      Biochem. Biophys. Res. Commun 383

      Pages: 460-463

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20591192
  • [Journal Article] Human leukocyte histocompatibility antigen class II-induced cytokines from human gingival fibroblasts promote proliferation of human umbilical vein endothelial cells : potential association with enhanced angiogenesis in chronic periodontal inflammation.2009

    • Author(s)
      Okada Y, Meguro M, Ohyama H, Yoshizawa S, Takeuchi-Hatanaka K, Kato N, Matsushita S, Takashiba S, Nishimura F.
    • Journal Title

      J Periodont Res 44

      Pages: 103-109

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20591192
  • [Journal Article] Direct contact between human peripheral blood mononuclear cells and renal fibroblasts facilitates the expression of monocyte chemoattractant protein-12003

    • Author(s)
      Hao, L., H.Okada, Y.Kanno, T.Inoue, T.Kobayashi, Y.Watanabe, et al.
    • Journal Title

      Am J Nephrol 23・4

      Pages: 208-213

    • Data Source
      KAKENHI-PROJECT-14571033
  • [Presentation] 腎線維化の進行にはCCN2-integrinを介したFAKのリン酸化が関与する2021

    • Author(s)
      深谷大地、井上勉、天野博明、岡田浩一
    • Organizer
      日本CCNファミリー研究会
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Presentation] CCN2/CTGF Causes Renal Fibrosis Progression Through the Integrin/FAK Signal Pathway2021

    • Author(s)
      Daichi Fukaya, Tsutomu Inoue, Hiroaki Amano, Yusuke Watanabe, Hirokazu Okada
    • Organizer
      Kidney Week 2021, Annual Meeting of American Society of Nephrology
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Presentation] CCN2/CTGFはintegrin/FAKを介して腎線維化を進行する2021

    • Author(s)
      深谷 大地, 天野 博明, 井上 勉, 草野 武, 岡田 浩一
    • Organizer
      第64回日本腎臓学会学術総会
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Presentation] CCN2/CTGFはintegrin/FAKを介して腎線維化を進行する2021

    • Author(s)
      深谷大地、井上勉、天野博明、岡田浩一
    • Organizer
      日本分子腎臓フォーラム
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Presentation] CCN2 Module IV-Derived Decoy Peptides Attenuate Renal Fibrogenesis Through Inhibition of FAK Pathway in the Tubular Epithelium2019

    • Author(s)
      Hiroaki Amano, Tsutomu Inoue, Hirokazu Okada
    • Organizer
      Annual Meeting of American Society of Nephrology
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Presentation] CCN2 Module IV-Derived Decoy Peptides Attenuate Renal Fibrogenesis Through Inhibition of FAK Pathway in the Tubular Epithelium2019

    • Author(s)
      Hiroaki Amano, Tsutomu Inoue, Hirokazu Okada
    • Organizer
      Annual Meeting of American Society of Nephrology
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Presentation] CCN2 module 4はFAKのリン酸化を介して腎線維化を促進する2019

    • Author(s)
      天野博明、井上勉、草野武、岡田浩一
    • Organizer
      日本腎臓学会学術総会
    • Data Source
      KAKENHI-PROJECT-19K08731
  • [Presentation] CCN2 module 4はFAKのリン酸化を介して腎線維化を促進する2019

    • Author(s)
      天野博明、井上勉、草野武、岡田浩一
    • Organizer
      日本腎臓学会学術総会
    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Presentation] CCN2 module-IV promotes renal fibrosis through activation of the FAK pathway in the tubular epithelium2018

    • Author(s)
      天野博明、井上勉、草野武、岡田浩一
    • Organizer
      The annual meeting ofthe American Society of Nephrology
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K09626
  • [Presentation] CCN2 Module-IV Promotes Renal Fibrosis Through Activation of the FAK Pathway in the Tubular Epithelium2018

    • Author(s)
      Hiroaki Amano, Tustomu Inoue, Hirokazu Okada
    • Organizer
      Annual meeting of American Society of Nephrology
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Presentation] 初代皮膚線維芽細胞におけるCCN2 module-IVの作用機序について2018

    • Author(s)
      天野博明、井上勉、草野武、岡田浩一
    • Organizer
      第61回日本腎臓学会学術総会
    • Data Source
      KAKENHI-PROJECT-16K09626
  • [Presentation] 初代皮膚線維芽細胞におけるCCN2 module-IVの作用機序について2018

    • Author(s)
      天野 博明, 井上 勉, 草野 武, 岡田 浩一
    • Organizer
      日本腎臓学会学術集会
    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Presentation] Module IV-defected mutant CCN2 knock-in transgenic mice grow and develop normally, but fibrotic properties are attenuated in a number of kidney diseases.2017

    • Author(s)
      天野博明、井上勉、草野武、岡田浩一
    • Organizer
      The annual meeting ofthe American Society of Nephrology
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K09626
  • [Presentation] Module IV-defected mutant CCN2 knock-in transgenic mice grow and develop normally, but fibrotic properties are attenuated in a number of kidney diseases.2017

    • Author(s)
      Tsutomu Inoue, Ono Atsushi, Hirokazu Okada.
    • Organizer
      Annual Meeting of American Society of Nephrology 2018 (Kidney Week 2018)
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Presentation] 尿細管上皮細胞のCaspase活性とCCN2発現はAKIからCKDへの移行を修飾する2016

    • Author(s)
      草野武、井上勉、中元秀友、岡田浩一
    • Organizer
      第59回日本腎臓学会学術総会
    • Place of Presentation
      横浜
    • Year and Date
      2016-06-17
    • Data Source
      KAKENHI-PROJECT-16K09626
  • [Presentation] 尿細管上皮細胞のCaspase活性とCCN2発現はAKIからCKDへの移行を修飾する2016

    • Author(s)
      草野 武, 井上 勉, 中元 秀友, 岡田 浩一
    • Organizer
      第59回日本腎臓学会学術総会
    • Place of Presentation
      横浜
    • Year and Date
      2016-06-17
    • Data Source
      KAKENHI-PROJECT-16K09627
  • [Presentation] A Module IV-Defective CCN2 Mutant and Module IV-Derived Decoy Peptides Attenuate Renal Fibrogenesis

    • Author(s)
      Tsutomu Inoue, MD, PhD, Takeru Kusano, MD, Hiroaki Amano, MD, Kei Sugiyama, MD, Hiromichi Suzuki, MD, PhD, Hirokazu Okada, MD, PhD
    • Organizer
      Kidney Week 2014, The annual meeting of the American Society of Nephrology
    • Place of Presentation
      Philadelphia Convention Center, Philadelphia, PA, USA
    • Year and Date
      2014-11-13 – 2014-11-16
    • Data Source
      KAKENHI-PROJECT-25461228
  • 1.  INOUE Tsutomu (30406475)
    # of Collaborated Projects: 8 results
    # of Collaborated Products: 22 results
  • 2.  KANNO Yoshihiko (30276232)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 1 results
  • 3.  MATSUSHITA Sho (50167649)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 4 results
  • 4.  HIGASHI Takehiro (00468381)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 2 results
  • 5.  AWATA Takuya (40184303)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 6.  KOUSAKA Hitoshi (00251554)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 7.  SIMOJYO Naoki (40221303)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 8.  IWANO Masayuki (20275324)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 9.  KIMURA Hideki (20283187)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 10.  NAKATANI Kimihiko (80398445)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 11.  oyama masafumi (70276351)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 12.  SUZUKI Hiromichi (80129494)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 13.  KIKUTA Tomohiro (70383239)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 14.  城武 卓 (10528805)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 15.  天野 博明 (10862675)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 16.  MIYAWAKI Atsushi
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results

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