Yogosawa Satomi 與五沢里美

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與五沢里美ヨゴサワサトミ

YOGOSAWA Satomi 與五沢里美

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Researcher Number	60392437
Other IDs	https://orcid.org/0000-0002-3161-6735
Affiliation (Current)	2024: 東京慈恵会医科大学, 医学部, 講師
Affiliation (based on the past Project Information) *help	2022: 東京慈恵会医科大学, 医学部, 講師 2017 – 2020: 東京慈恵会医科大学, 医学部, 助教 2013 – 2014: 群馬大学, 生体調節研究所, 研究員 2008: Gunma University, 生体調節研究所, グローバルCOE研究員 2007: Tokyo University of Science, 生命科学研究所, ポストドクトラル研究員
Review Section/Research Field	Principal Investigator Basic Section 49010:Pathological biochemistry-related / Pathological medical chemistry / Metabolomics / Functional biochemistry
Keywords	Principal Investigator リン酸化酵素 / 大腸癌 / 脂質 / 脂質代謝 / 癌 / TGFb / TGFβ / 脂肪細胞 / 脂肪蓄積 / 糖尿病 … More / 肥満 / B細胞 / タンパク質 / 蛋白質 / 細胞生物学 / シグナル伝達 / 輸送 / 受容体 Less

Molecular mechanism of novel anti-tumor signal that suppress the colorectal cancer progressionPrincipal Investigator
- Principal Investigator
  
  與五沢里美
- Project Period (FY)
  2022 – 2024
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 49010:Pathological biochemistry-related
- Research Institution
  Jikei University School of Medicine
Molecular mechanism of tumor progression by the regulation of glucose and lipid metabolismPrincipal Investigator
- Principal Investigator
  
  Yogosawa Satomi
- Project Period (FY)
  2017 – 2020
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Pathological medical chemistry
- Research Institution
  Jikei University School of Medicine
Elucidating the molecular mechanism of novel ALK7 ligand in fat accumulation in obesityPrincipal Investigator
- Principal Investigator
  
  YOGOSAWA Satomi
- Project Period (FY)
  2013 – 2014
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Metabolomics
- Research Institution
  Gunma University
Functional analysis of CMTM3/BNAS2 in B cell receptor endocytosisPrincipal Investigator
- Principal Investigator
  
  YOGOSAWA Satomi
- Project Period (FY)
  2007 – 2008
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Functional biochemistry
- Research Institution
  Gunma University
  Tokyo University of Science

All 2023 2021 2020 2019 2018 2013 2007 Other

All Journal Article Presentation Book

[Book] ALK7による脂肪分解抑制作用2013
- Author(s)
  與五沢里美、泉哲郎
- Publisher
  科学評論社
- Data Source
  KAKENHI-PROJECT-25860739
[Journal Article] Carbonic anhydrase 13 suppresses bone metastasis in breast cancer2021
- Author(s)
  Yogosawa Satomi、Nakayama Jun、Nishi Mayuko、Ryo Akihide、Yoshida Kiyotsugu
- Journal Title
  
  Cancer Treatment and Research Communications
  
  Volume: 27 Pages: 100332-100332
- DOI
  10.1016/j.ctarc.2021.100332
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-20H03519, KAKENHI-PROJECT-17K08672, KAKENHI-PROJECT-18K09000, KAKENHI-PROJECT-18K19484
[Journal Article] Tumor suppressive role for kinases phosphorylating p53 in DNA damage-induced apoptosis2018
- Author(s)
  Yogosawa Satomi、Yoshida Kiyotsugu
- Journal Title
  
  Cancer Science
  
  Volume: 109 Issue: 11 Pages: 3376-3382
- DOI
  10.1111/cas.13792
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17K08672, KAKENHI-PROJECT-17H03584
[Journal Article] Roles of activin receptor-like kinase 7 signaling and its target, peroxisome proliferator-activated receptor γ, in lean and obese adipocytes.2013
- Author(s)
  Yogosawa S, and Izumi T
- Journal Title
  
  Adipocyte
  
  Volume: 2 Issue: 4 Pages: 246-250
- DOI
  10.4161/adip.24974
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-24390068, KAKENHI-PROJECT-24659442, KAKENHI-PUBLICLY-25126702, KAKENHI-PROJECT-25860739
[Presentation] 病理形態解析が明らかにするDYRK2欠損マウスの多様な組織形成異常2023
- Author(s)
  與五沢里美
- Organizer
  先端モデル動物支援プラットフォーム2022年度成果発表会
- Invited
- Data Source
  KAKENHI-PROJECT-22K06918
[Presentation] DYRK2欠損マウスは先天性奇形症候群の疾患モデルとなる2020
- Author(s)
  與五沢里美、吉田清嗣
- Organizer
  2019年度先端モデル動物支援プラットフォーム成果発表会
- Data Source
  KAKENHI-PROJECT-17K08672
[Presentation] Carbonic anhydrase 13 suppresses bone metastasis of breast cancer cells.2020
- Author(s)
  Yogosawa S, Nakayama J, Nishi M, Ryo A, Yoshida K.
- Organizer
  第７９回日本癌学会学術総会
- Data Source
  KAKENHI-PROJECT-17K08672
[Presentation] DYRK2欠損マウスは肺低形成により呼吸不全となり、出生直後致死となる2019
- Author(s)
  與五沢里美、吉田清嗣
- Organizer
  平成30年度先端モデル動物支援プラットフォーム成果発表会
- Data Source
  KAKENHI-PROJECT-17K08672
[Presentation] B細胞抗原受容体のエンドサイトーシスに関与するCMTM3/BNAS2の機能解析2007
- Author(s)
  與五沢里美
- Organizer
  日本分子生物学会
- Place of Presentation
  神奈川県横浜市
- Year and Date
  2007-12-13
- Data Source
  KAKENHI-PROJECT-19770115
[Presentation] B細胞抗原受容体のエンドサイトーシスに関与するCMTM3/BNAS2の機能解析2007
- Author(s)
  與五沢里美
- Organizer
  第30回日本分子生物学会
- Place of Presentation
  横浜
- Year and Date
  2007-12-13
- Data Source
  KAKENHI-PROJECT-19770115
[Presentation] 新規脂肪蓄積遺伝子ALK7の機能解析
- Author(s)
  與五沢里美、泉哲郎
- Organizer
  第３４回日本肥満学会学術大会
- Place of Presentation
  東京国際フォーラム（東京都千代田区）
- Data Source
  KAKENHI-PROJECT-25860739
[Presentation] 糖尿病・肥満モデルマウスにおける新たな脂肪蓄積遺伝子ALK7の解析
- Author(s)
  與五沢里美、水谷伸、小川佳宏、泉哲郎
- Organizer
  第５６回日本糖尿病学会年次学術集会
- Place of Presentation
  熊日生涯学習プラザ（熊本県熊本市）
- Data Source
  KAKENHI-PROJECT-25860739

1. 吉田清嗣 (70345312)

# of Collaborated Projects: 1 results

# of Collaborated Products: 5 results
2. 泉哲郎

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

Yogosawa Satomi 與五沢 里美

與五沢 里美 ヨゴサワ サトミ

YOGOSAWA Satomi 與五沢 里美

Research Projects

Research Products

Co-Researchers

Molecular mechanism of novel anti-tumor signal that suppress the colorectal cancer progressionPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Molecular mechanism of tumor progression by the regulation of glucose and lipid metabolismPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Elucidating the molecular mechanism of novel ALK7 ligand in fat accumulation in obesityPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Functional analysis of CMTM3/BNAS2 in B cell receptor endocytosisPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Book] ALK7による脂肪分解抑制作用2013

Author(s)

Publisher

Data Source

[Journal Article] Carbonic anhydrase 13 suppresses bone metastasis in breast cancer2021

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Tumor suppressive role for kinases phosphorylating p53 in DNA damage-induced apoptosis2018

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Roles of activin receptor-like kinase 7 signaling and its target, peroxisome proliferator-activated receptor γ, in lean and obese adipocytes.2013

Author(s)

Journal Title

DOI

Data Source

[Presentation] 病理形態解析が明らかにするDYRK2欠損マウスの多様な組織形成異常2023

Author(s)

Organizer

Data Source

[Presentation] DYRK2欠損マウスは先天性奇形症候群の疾患モデルとなる2020

Author(s)

Organizer

Data Source

[Presentation] Carbonic anhydrase 13 suppresses bone metastasis of breast cancer cells.2020

Author(s)

Organizer

Data Source

[Presentation] DYRK2欠損マウスは肺低形成により呼吸不全となり、出生直後致死となる2019

Author(s)

Organizer

Data Source

[Presentation] B細胞抗原受容体のエンドサイトーシスに関与するCMTM3/BNAS2の機能解析2007

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] B細胞抗原受容体のエンドサイトーシスに関与するCMTM3/BNAS2の機能解析2007

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] 新規脂肪蓄積遺伝子ALK7の機能解析

Author(s)

Organizer

Yogosawa Satomi 與五沢里美

與五沢里美ヨゴサワサトミ

YOGOSAWA Satomi 與五沢里美