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Kon Shunsuke  昆 俊亮

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… Alternative Names

昆 俊亮  コン シュンスケ

SHUNSUKE Kon  昆 俊亮

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Researcher Number 70506641
Other IDs
Affiliation (Current) 2025: 東京理科大学, 研究推進機構生命医科学研究所, 准教授
Affiliation (based on the past Project Information) *help 2023 – 2024: 東京理科大学, 研究推進機構生命医科学研究所, 准教授
2018 – 2022: 東京理科大学, 研究推進機構生命医科学研究所, 講師
2014 – 2015: 北海道大学, 遺伝子病制御研究所, 助教
2012: 東北大学, 加齢医学研究科, 助教
2011 – 2012: 東北大学, 加齢医学研究所, 助教
Review Section/Research Field
Principal Investigator
Basic Section 42030:Animal life science-related / Complex systems / Biological Sciences / Medium-sized Section 50:Oncology and related fields / Medium-sized Section 48:Biomedical structure and function and related fields / Functional biochemistry
Except Principal Investigator
Medium-sized Section 50:Oncology and related fields
Keywords
Principal Investigator
細胞競合 / 多段階発がん / EndMT / リンパ行性転移 / 細胞競合マウスモデル / 正常間質細胞 / シンギュラリティ / がん臨界 / 近接細胞蛍光標識 / がん変異細胞 … More / sGRAPHIC / 近接細胞蛍光標識技術 / 細胞競合マーカー / 超初期がん細胞マーカー / 乳糜管 / 腫瘍リンパ管新生 / がん転移 / 発がん / 内皮間葉転換 / シングルセルRNAシークエンス解析 / リンパ管侵襲 / シンギュラリティがん細胞 / MMP / びまん性浸潤 / 腸管上皮細胞 / びまん性発がん / 腸管幹細胞 / MMP21 / NF-κB / BioID / オートファゴソームの非分解機能 / 慢性膵炎 / オートファジー / 生体内イメージング / がん間質化 / ex vivoイメージング / 正常線維芽細胞 / がんシンギュラリティ / 家族性大腸がん / マウスモデル / 国際情報交流 アメリカ合衆国 / 白血病 / 骨髄異形成症候群 / SMAP1 / 細胞内小胞輸送 … More
Except Principal Investigator
浸潤性 / ショウジョウバエ / 平面内細胞極性 / 腫瘍浸潤 / 組織構造 / 細胞増殖 / 上皮組織 / 腫瘍形成 Less
  • Research Projects

    (10 results)
  • Research Products

    (31 results)
  • Co-Researchers

    (3 People)
  •  Elucidation of EndMT induction of lymphatic endothelial cellsPrincipal Investigator

    • Principal Investigator
      昆 俊亮
    • Project Period (FY)
      2024 – 2026
    • Research Category
      Grant-in-Aid for Challenging Research (Exploratory)
    • Review Section
      Medium-sized Section 50:Oncology and related fields
    • Research Institution
      Tokyo University of Science
  •  Dissection for elimination of transformed cells by normal epithelial cellsPrincipal Investigator

    • Principal Investigator
      昆 俊亮
    • Project Period (FY)
      2023 – 2025
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Review Section
      Basic Section 42030:Animal life science-related
    • Research Institution
      Tokyo University of Science
  •  Singularity at emergence of cancer cells in normal stromaPrincipal Investigator

    • Principal Investigator
      昆 俊亮
    • Project Period (FY)
      2021 – 2022
    • Research Category
      Grant-in-Aid for Scientific Research on Innovative Areas (Research in a proposed research area)
    • Review Section
      Complex systems
    • Research Institution
      Tokyo University of Science
  •  Analysis of tumor-induced lymphangiogenesisPrincipal Investigator

    • Principal Investigator
      Kon Shunsuke
    • Project Period (FY)
      2021 – 2023
    • Research Category
      Grant-in-Aid for Challenging Research (Exploratory)
    • Review Section
      Medium-sized Section 48:Biomedical structure and function and related fields
    • Research Institution
      Tokyo University of Science
  •  Delineation of the tumor-suppressive autophagy mode through cell competitionPrincipal Investigator

    • Principal Investigator
      昆 俊亮
    • Project Period (FY)
      2020 – 2021
    • Research Category
      Grant-in-Aid for Scientific Research on Innovative Areas (Research in a proposed research area)
    • Review Section
      Biological Sciences
    • Research Institution
      Tokyo University of Science
  •  Molecular dissection of malfunction of cell competition-mediated elimination of transformed cells in intestinePrincipal Investigator

    • Principal Investigator
      Kon Shunsuke
    • Project Period (FY)
      2020 – 2022
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Review Section
      Basic Section 42030:Animal life science-related
    • Research Institution
      Tokyo University of Science
  •  singularity in normal stroma at emergence of cancer cellsPrincipal Investigator

    • Principal Investigator
      昆 俊亮
    • Project Period (FY)
      2019 – 2020
    • Research Category
      Grant-in-Aid for Scientific Research on Innovative Areas (Research in a proposed research area)
    • Review Section
      Complex systems
    • Research Institution
      Tokyo University of Science
  •  Structural analysis of tumor hotspots, an epithelial tissue-intrinsic novel oncogenic niche

    • Principal Investigator
      Tamori Yoichiro
    • Project Period (FY)
      2018 – 2021
    • Research Category
      Fund for the Promotion of Joint International Research (Fostering Joint International Research (B))
    • Review Section
      Medium-sized Section 50:Oncology and related fields
    • Research Institution
      Kyoto University
      Hokkaido University
  •  多段階発がん過程における細胞競合の関与Principal Investigator

    • Principal Investigator
      昆 俊亮
    • Project Period (FY)
      2014 – 2015
    • Research Category
      Grant-in-Aid for Scientific Research on Innovative Areas (Research in a proposed research area)
    • Review Section
      Biological Sciences
    • Research Institution
      Hokkaido University
  •  Dysregulation of clathrin-dependent traffic causes hematopoietic neoplasmPrincipal Investigator

    • Principal Investigator
      SHUNSUKE Kon
    • Project Period (FY)
      2011 – 2012
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Functional biochemistry
    • Research Institution
      Tohoku University

All 2023 2022 2021 2020 2019 2016 2015 2013 2012 2011 Other

All Journal Article Presentation Book

  • [Book] 哺乳類における細胞競合の生理的・病理的役割2015

    • Author(s)
      昆 俊亮、藤田 恭之
    • Total Pages
      5
    • Publisher
      臨床免疫
    • Data Source
      KAKENHI-PUBLICLY-26112701
  • [Book] クラスリン小胞形成因子SMAP1の欠損は細胞内小胞輸送の異常を来し、骨髄異形成症候群を誘引する2013

    • Author(s)
      昆俊亮
    • Publisher
      細胞工学
    • Data Source
      KAKENHI-PROJECT-23770137
  • [Book] 細胞工学2013

    • Author(s)
      昆 俊亮
    • Total Pages
      2
    • Publisher
      学研メディカル秀潤社
    • Data Source
      KAKENHI-PROJECT-23770137
  • [Journal Article] Wnt activation-induced disturbance of cell competition causes diffuse invasion of transformed cells through upregulation of NF-κB-mediated MMP212023

    • Author(s)
      Nakai, K., Lin, H., Yamano, S., Tanaka, S., Kitamoto, S., Sakuma, K., Kurauchi, J., Akter, E., Konno, M., Koseki, J., Takahashi, H., Yokoyama, H., Shiraki, Y., Enomoto, A., Abe, S., Hayakawa, Y., Ushiku, T., Mutoh, M., Fujita, Y. and Kon, S.
    • Journal Title

      Nature Communications

      Volume: 14 Issue: 1 Pages: 7048-7048

    • DOI

      10.1038/s41467-023-42774-6

    • Peer Reviewed / Open Access / Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-21K19357, KAKENHI-PLANNED-21H05285, KAKENHI-PROJECT-21H05039, KAKENHI-PROJECT-23K24941, KAKENHI-PROJECT-23K27089
  • [Journal Article] Cell competition-induced apical elimination of transformed cells, EDAC, orchestrates the cellular homeostasis2021

    • Author(s)
      Kon Shunsuke、Fujita Yasuyuki
    • Journal Title

      Developmental Biology

      Volume: 476 Pages: 112-116

    • DOI

      10.1016/j.ydbio.2021.03.015

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-20K21411, KAKENHI-PROJECT-18KK0234, KAKENHI-PUBLICLY-20H05347, KAKENHI-PUBLICLY-21H00441, KAKENHI-PLANNED-21H05285, KAKENHI-PROJECT-21H05039, KAKENHI-PROJECT-20H03166, KAKENHI-PROJECT-21K19357, KAKENHI-PROJECT-22H04922
  • [Journal Article] GRP30-expressing gastric chief cells do not differentiate but are eliminated via PDK-dependent cell competition during development of metaplasia.2020

    • Author(s)
      1.Hata M, Kinoshita H, Hayakawa Y, Konishi M, Tsuboi M, Oya Y, Hayata Y, Nakagawa H, Tateishi K, Fujiwara H, Hirata Y, Worthley DL, Muranishi Y, Furukawa T, Kon S, Tomita H, Wang TC, Koike K.
    • Journal Title

      Gastroenterology

      Volume: 158 Issue: 6 Pages: 1650-1666

    • DOI

      10.1053/j.gastro.2020.01.046

    • Peer Reviewed / Int'l Joint Research
    • Data Source
      KAKENHI-PUBLICLY-19H05432, KAKENHI-PROJECT-18H02593, KAKENHI-PROJECT-17H05081, KAKENHI-PROJECT-17K09347, KAKENHI-PROJECT-20H03656, KAKENHI-PROJECT-16H06276
  • [Journal Article] A role of the sphingosine-1-phosphate (S1P)- S1P receptor 2 pathway in Epithelial Defense Against Cancer (EDAC).2016

    • Author(s)
      Yamamoto, S., Yako, Y., Fujioka, Y., Kajita, M., Kameyama, T., Kon, S., Ishikawa, S., Ohba, Y., Ohno, Y., Kihara, A., and Fujita, Y.
    • Journal Title

      Molecular Biology of the Cell

      Volume: 27 Pages: 491-499

    • NAID

      120005740332

    • Peer Reviewed / Int'l Joint Research
    • Data Source
      KAKENHI-PUBLICLY-26112701
  • [Journal Article] The cell competition-based high-throughput screening identifies small compounds that promote the elimination of RasV12-transformed cells from epithelia.2015

    • Author(s)
      Yamauchi, H., Matsumaru, T., Morita, T., Ishikawa, S., Maenaka, K., Takigawa, I., Semba, K., Kon, S. and Fujita, Y.
    • Journal Title

      Scientific Reports

      Volume: 5 Pages: 15336-15336

    • NAID

      120005674073

    • Peer Reviewed / Open Access / Int'l Joint Research
    • Data Source
      KAKENHI-PUBLICLY-26112701
  • [Journal Article] EPLIN is a crucial regulator for extrusion of RasV12-transformed cells.2015

    • Author(s)
      Ohoka, A., Kajita, M., Ikenouchi, J., Yako, Y., Kitamoto, S., Kon, S., Ikegawa, M., Shimada, T., Ishikawa, S., Fujita, Y.
    • Journal Title

      Journal of Cell Science

      Volume: 128 Pages: 781-789

    • Peer Reviewed / Int'l Joint Research
    • Data Source
      KAKENHI-PUBLICLY-26112701
  • [Journal Article] Smap1 deficiency perturbs receptor trafficking and predisposes mice to myelodysplasia.2013

    • Author(s)
      Kon et al.
    • Journal Title

      J. Clin. Invest.

      Volume: 123 Issue: 3 Pages: 1123-1137

    • DOI

      10.1172/jci63711

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23501256, KAKENHI-PROJECT-23770137, KAKENHI-PROJECT-23770148, KAKENHI-PROJECT-24220011, KAKENHI-PROJECT-24591404, KAKENHI-PROJECT-24659458
  • [Presentation] 細胞競合によるがん変異細胞検出法の開発2023

    • Author(s)
      昆 俊亮
    • Organizer
      日本がん予防学術学会
    • Invited
    • Data Source
      KAKENHI-PROJECT-21K19357
  • [Presentation] 細胞競合によるがん変異細胞検出法の開発2023

    • Author(s)
      昆 俊亮
    • Organizer
      日本がん予防学術学会
    • Invited
    • Data Source
      KAKENHI-PROJECT-23K27089
  • [Presentation] デノボがん細胞のリンパ管侵襲機構2022

    • Author(s)
      昆 俊亮
    • Organizer
      日本リンパ学会
    • Data Source
      KAKENHI-PROJECT-21K19357
  • [Presentation] デノボがん細胞のリンパ行性転移機構2022

    • Author(s)
      昆 俊亮
    • Organizer
      第31回 日本がん転移学会
    • Invited
    • Data Source
      KAKENHI-PUBLICLY-21H00441
  • [Presentation] デノボがん細胞のリンパ管侵襲機構2022

    • Author(s)
      昆 俊亮
    • Organizer
      第46回 日本リンパ学会
    • Data Source
      KAKENHI-PROJECT-20H03166
  • [Presentation] デノボがん細胞のリンパ行性転移機構2022

    • Author(s)
      昆 俊亮
    • Organizer
      第31回 日本がん転移学会
    • Data Source
      KAKENHI-PROJECT-20H03166
  • [Presentation] デノボがん細胞のリンパ行性転移機構2022

    • Author(s)
      昆 俊亮
    • Organizer
      日本がん転移学会
    • Data Source
      KAKENHI-PROJECT-21K19357
  • [Presentation] がん細胞が誕生したときの生体内反応2021

    • Author(s)
      昆 俊亮
    • Organizer
      第53回 フォーラム富山「創薬」(招待講演)
    • Data Source
      KAKENHI-PUBLICLY-20H05347
  • [Presentation] がん細胞が誕生したときの生体内反応2021

    • Author(s)
      昆 俊亮
    • Organizer
      第53回 フォーラム富山「創薬」
    • Invited
    • Data Source
      KAKENHI-PROJECT-21K19357
  • [Presentation] がん細胞が誕生したときの生体内反応2021

    • Author(s)
      昆 俊亮
    • Organizer
      第53回 フォーラム富山「創薬」
    • Invited
    • Data Source
      KAKENHI-PROJECT-18KK0234
  • [Presentation] がん細胞が誕生したときの生体内反応2021

    • Author(s)
      昆 俊亮
    • Organizer
      第53回 フォーラム富山「創薬」
    • Invited
    • Data Source
      KAKENHI-PROJECT-20H03166
  • [Presentation] がん細胞が誕生したときの生体内反応2021

    • Author(s)
      昆 俊亮
    • Organizer
      第53回 フォーラム富山「創薬」(招待講演)
    • Invited
    • Data Source
      KAKENHI-PUBLICLY-21H00441
  • [Presentation] Singularity at emergence of cancer cells in normal stroma2020

    • Author(s)
      昆 俊亮
    • Organizer
      第58回 日本生物物理学会
    • Invited
    • Data Source
      KAKENHI-PUBLICLY-19H05432
  • [Presentation] がん細胞が出現した正常間質組織でのシンギュラリティ現象の解明2020

    • Author(s)
      昆 俊亮
    • Organizer
      新学術領域研究「シンギュラリティ生物学」,「細胞ダイバース」合同会
    • Invited
    • Data Source
      KAKENHI-PUBLICLY-19H05432
  • [Presentation] 細胞競合研究からがん発生研究へ2019

    • Author(s)
      昆 俊亮
    • Organizer
      大阪大学蛋白研シンポジウム「がん研究の新機軸」
    • Invited
    • Data Source
      KAKENHI-PUBLICLY-19H05432
  • [Presentation] 多段階発がんにおける細胞競合の役割2016

    • Author(s)
      昆 俊亮
    • Organizer
      厚生労働省委託事業「ヒト幹細胞情報化推進事業」Stemcell Knowledge & Information Portal 【SKIP Seminar】Cutting Edge of Cancer Stem Cell
    • Place of Presentation
      慶應義塾大学医学部(東京都・信濃町)
    • Year and Date
      2016-03-15
    • Invited
    • Data Source
      KAKENHI-PUBLICLY-26112701
  • [Presentation] マウスモデルを用いた多段階発がんにおける細胞競合の意義の解明2016

    • Author(s)
      昆 俊亮、松本 朋大、江上 陸、渡邊 裕貴、石橋 公二朗、藤田 恭之
    • Organizer
      「個体レベルでのがん研究支援活動」ワークショップ
    • Place of Presentation
      琵琶湖ホテル (滋賀県・大津市)
    • Year and Date
      2016-02-03
    • Data Source
      KAKENHI-PUBLICLY-26112701
  • [Presentation] クラスリン小胞形成因子SMAP1の欠損は細胞内小胞輸送の異常をきたし、骨髄異型性症候群を誘引する2012

    • Author(s)
      昆 俊亮
    • Organizer
      第35回日本分子生物学会年会
    • Place of Presentation
      福岡
    • Data Source
      KAKENHI-PROJECT-23770137
  • [Presentation] クラスリン小胞形成因子SMAP1の欠損は細胞内小胞輸送の異常をきたし、骨髄異型性症候群を誘引する2012

    • Author(s)
      昆俊亮、峯岸直子、田邊賢司、渡邊利雄、船木智、坂元大輔、樋口雄大、清成寛、浅野克敏、福本学、真田昌、小川誠司、中村卓郎、佐竹正延
    • Organizer
      第35回分子生物学会年会ワークショップ「メンブレントラフィックと疾患」
    • Place of Presentation
      福岡
    • Data Source
      KAKENHI-PROJECT-23770137
  • [Presentation] Dysregulation of clathrin-dependent traffic predisposes SMAP1-targeted mice to develop myelodysplastic syndrome and acute myeloid leukemia2011

    • Author(s)
      昆俊亮、船木智、峯岸直子、清成寛、田邊賢司、渡邊利雄、中村卓郎、佐竹正延
    • Organizer
      文部科学省科学研究費補助金新学術領域研究『がん研究分野の特性等を踏まえた支援活動』がん若手研究者ワークショップ
    • Place of Presentation
      蓼科
    • Data Source
      KAKENHI-PROJECT-23770137
  • [Presentation] Dysregulation of clathrin-dependent traffic predisposes SMAP1-targeted mice to develop myelodysplastic syndrome and acute myeloid leukemia

    • Author(s)
      昆 俊亮
    • Organizer
      文部科学省科学研究費補助金 新学術領域研究『がん研究分野の特性等を踏まえた支援活動』 がん若手研究者ワークショップ
    • Place of Presentation
      蓼科
    • Data Source
      KAKENHI-PROJECT-23770137
  • 1.  Tamori Yoichiro (10717325)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 2.  石原 誠一郎 (10719933)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 3.  藤田 恭之
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results

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