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Hamada Nanako  浜田 奈々子

ORCIDConnect your ORCID iD *help
Researcher Number 70721835
Other IDs
Affiliation (Current) 2025: 愛知県医療療育総合センター発達障害研究所, 分子病態研究部, 主任研究員
Affiliation (based on the past Project Information) *help 2023: 愛知県医療療育総合センター発達障害研究所, 分子病態研究部, 主任研究員
2019 – 2022: 愛知県医療療育総合センター発達障害研究所, 分子病態研究部, 研究員
2018: 愛知県心身障害者コロニー発達障害研究所, 分子病態研究部, リサーチレジデント
2014 – 2017: 愛知県心身障害者コロニー発達障害研究所, 神経制御学部, 特別研究員
Review Section/Research Field
Principal Investigator
Basic Section 52050:Embryonic medicine and pediatrics-related / Pediatrics
Keywords
Principal Investigator
ASD / cep152 / 小頭症 / Cep152 / てんかん / phactr1 / PHACTR1 / Phactr1 / ウエスト症候群 / 大脳皮質形成 … More / 自閉症 / DUSP22 / MRTNR / Dusp22 / 自閉スペクトラム症 / corticogenesis / Autism / 樹状突起スパイン形成不全 / 軸索伸長抑制 / 樹状突起形成不全 / 神経細胞移動障害 / 神経発達障害 / 神経細胞移動 / A2BP1 Less
  • Research Projects

    (4 results)
  • Research Products

    (73 results)
  • Co-Researchers

    (4 People)
  •  細胞分裂制御遺伝子の新規機能と病態形成メカニズムの解明Principal Investigator

    • Principal Investigator
      浜田 奈々子
    • Project Period (FY)
      2023 – 2025
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 52050:Embryonic medicine and pediatrics-related
    • Research Institution
      Institute for Developmental Research Aichi Developmental Disability Center
  •  Elucidation of the molecular pathogenetic mechanism of West syndrome in the context of Phactr1 gene abnormalitiesPrincipal Investigator

    • Principal Investigator
      Hamada Nanako
    • Project Period (FY)
      2020 – 2023
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 52050:Embryonic medicine and pediatrics-related
    • Research Institution
      Institute for Developmental Research Aichi Developmental Disability Center
  •  pathophysiology of DUSP22 and MTNR1 gene abnormalitiesPrincipal Investigator

    • Principal Investigator
      HAMADA NANAKO
    • Project Period (FY)
      2017 – 2020
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Pediatrics
    • Research Institution
      Institute for Developmental Research Aichi Developmental Disability Center
  •  Pathophysiolosical analysis of A2BP1, a candidate gene for ASD, in the brain developmentPrincipal Investigator

    • Principal Investigator
      Hamada Nanako
    • Project Period (FY)
      2014 – 2017
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Pediatrics
    • Research Institution
      Institute for Developmental Research, Aichi Human Service Center

All 2024 2023 2022 2021 2020 2019 2018 2017 2016 2015 Other

All Journal Article Presentation

  • [Journal Article] Analyses of Conditional Knockout Mice for Pogz, a Gene Responsible for Neurodevelopmental Disorders in Excitatory and Inhibitory Neurons in the Brain2024

    • Author(s)
      Hamada Nanako、Nishijo Takuma、Iwamoto Ikuko、Shifman Sagiv、Nagata Koh-ichi
    • Journal Title

      Cells

      Volume: 13 Issue: 6 Pages: 540-540

    • DOI

      10.3390/cells13060540

    • Peer Reviewed / Open Access / Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-23K14997, KAKENHI-PROJECT-23K07284, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-23K24310
  • [Journal Article] Expression analysis of type I ARF small GTPases ARF1-3 during mouse brain development2024

    • Author(s)
      Matsuki Tohru、Hamada Nanako、Ito Hidenori、Sugawara Ryota、Iwamoto Ikuko、Nakayama Atsuo、Nagata Koh-ichi
    • Journal Title

      Molecular Biology Reports

      Volume: 51 Issue: 1 Pages: 106-106

    • DOI

      10.1007/s11033-023-09142-5

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-23K19577, KAKENHI-PROJECT-23K07284, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08224, KAKENHI-PROJECT-23K24310
  • [Journal Article] <scp>MED13L</scp> and its disease‐associated variants influence the dendritic development of cerebral cortical neurons in the mammalian brain2023

    • Author(s)
      Hamada Nanako、Iwamoto Ikuko、Nagata Koh‐ichi
    • Journal Title

      Journal of Neurochemistry

      Volume: 165 Issue: 3 Pages: 334-347

    • DOI

      10.1111/jnc.15783

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23K07284, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-23K24310
  • [Journal Article] Expression analyses of WAC, a responsible gene for neurodevelopmental disorders, during mouse brain development2023

    • Author(s)
      Nishikawa Masashi、Matsuki Tohru、Hamada Nanako、Nakayama Atsuo、Ito Hidenori、Nagata Koh-ichi
    • Journal Title

      Medical Molecular Morphology

      Volume: 56 Issue: 4 Pages: 266-273

    • DOI

      10.1007/s00795-023-00364-x

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-23K14946, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08224, KAKENHI-PROJECT-23K24310
  • [Journal Article] Expression Analyses of Rich2/Arhgap44, a Rho Family GTPase-Activating Protein, during Mouse Brain Development.2023

    • Author(s)
      Goto N, Nishikawa M, Ito H, Noda M, Hamada N, Tabata H, Kinoshita M, Nagata K-I
    • Journal Title

      Dev. Neurosci.

      Volume: 45 Issue: 1 Pages: 19-26

    • DOI

      10.1159/000529051

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-21K15895, KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-20K08200
  • [Journal Article] Expression analyses of WAC, a responsible gene for neurodevelopmental disorders, during mouse brain development2023

    • Author(s)
      Masashi Nishikawa, Tohru Matsuki, Nanako Hamada, Atsuo Nakayama, Hidenori Ito, Koh-Ichi Nagata
    • Journal Title

      Med Mol Morphol

      Volume: 56 Pages: 266-273

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23K07284
  • [Journal Article] Expression Analyses of Cep152, a Responsible Gene Product for Autosomal Recessive Primary Microcephaly, during Mouse Brain Development2022

    • Author(s)
      Hamada Nanako、Noda Mariko、Ito Hidenori、Iwamoto Ikuko、Nagata Koh-ichi
    • Journal Title

      Developmental Neuroscience

      Volume: 44 Issue: 3 Pages: 162-170

    • DOI

      10.1159/000523922

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-23K24310
  • [Journal Article] Expression Analyses of Polo-Like Kinase 4, a Gene Product Responsible for Autosomal Recessive Microcephaly and Seckel Syndrome, during Mouse Brain Development2022

    • Author(s)
      Hamada Nanako、Iwamoto Ikuko、Noda Mariko、Nishikawa Masashi、Nagata Koh-ichi
    • Journal Title

      Developmental Neuroscience

      Volume: 44 Issue: 6 Pages: 643-650

    • DOI

      10.1159/000526914

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-22K19498, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-23K24310
  • [Journal Article] Heterotrimeric G-protein, Gi1, is involved in the regulation of proliferation, neuronal migration and dendrite morphology during cortical development in vivo.2021

    • Author(s)
      Hamada N, Iwamoto I, Kawamura N, Nagata K.
    • Journal Title

      J. Neurochem.

      Volume: 1 Issue: 4 Pages: 1-1

    • DOI

      10.1111/jnc.15205

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-17K16294, KAKENHI-PROJECT-19H03629
  • [Journal Article] Expression Analyses of Mediator Complex Subunit 13-Like: A Responsible Gene for Neurodevelopmental Disorders during Mouse Brain Development2021

    • Author(s)
      Hamada Nanako、Iwamoto Ikuko、Nishikawa Masashi、Nagata Koh-ichi
    • Journal Title

      Developmental Neuroscience

      Volume: - Issue: 1 Pages: 1-10

    • DOI

      10.1159/000515188

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20K22888, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K21589
  • [Journal Article] Expression analyses of PLEKHG2, a Rho family-specific guanine nucleotide exchange factor, during mouse brain development2021

    • Author(s)
      Nishikawa Masashi、Ito Hidenori、Noda Mariko、Hamada Nanako、Tabata Hidenori、Nagata Koh-ichi
    • Journal Title

      Medical Molecular Morphology

      Volume: - Issue: 2 Pages: 146-155

    • DOI

      10.1007/s00795-020-00275-1

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-20K22888, KAKENHI-PROJECT-19H03629, KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K08271, KAKENHI-PROJECT-20K21589
  • [Journal Article] Pogz deficiency leads to transcription dysregulation and impaired cerebellar activity underlying autism-like behavior in mice.2020

    • Author(s)
      Reut Suliman-Lavie, Ben Title, Yahel Cohen, Hamada N, Maayan Tal, Nitzan Tal, Galya Monderer- Rothkoff, Bjorg Gudmundsdottir, Kristbjorn O Gudmundsson, Jonathan R Keller, Guo-Jen Huang, Nagata K, Yosef Yarom, Sagiv Shifman.
    • Journal Title

      Nat. Commun.

      Volume: 11 Issue: 1 Pages: 5836-5836

    • DOI

      10.1038/s41467-020-19577-0

    • Peer Reviewed / Open Access / Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-17K16294
  • [Journal Article] Neuropathophysiological significance of the c.1449T>C/p.(Tyr64Cys) mutation in the CDC42 gene responsible for Takenouchi-Kosaki syndrome2020

    • Author(s)
      Hamada Nanako、Ito Hidenori、Shibukawa Yukinao、Morishita Rika、Iwamoto Ikuko、Okamoto Nobuhiko、Nagata Koh-ichi
    • Journal Title

      Biochemical and Biophysical Research Communications

      Volume: 529 Issue: 4 Pages: 1033-1037

    • DOI

      10.1016/j.bbrc.2020.06.104

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-20K08200, KAKENHI-PROJECT-20K21589, KAKENHI-PROJECT-17K16294, KAKENHI-PROJECT-19K08314
  • [Journal Article] Expression analyses of POGZ, a responsible gene for neurodevelopmental disorders, during mouse brain development.2019

    • Author(s)
      Ibaraki K, Hamada N, Iwamoto I, Ito H, Kawamura N, Morishita R, Tabata H, Nagata KI.
    • Journal Title

      Dev Neurosci.

      Volume: 41 Issue: 1-2 Pages: 139-148

    • DOI

      10.1159/000502128

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-19K07059, KAKENHI-PROJECT-17K16294, KAKENHI-PROJECT-16K08264, KAKENHI-PROJECT-19H03629
  • [Journal Article] De novo PHACTR1 mutations in West Syndrome and their pathophysiological effects.2018

    • Author(s)
      Hamada N, Ogaya S, Nakashima M, Nishijo T, Sugawara Y, Iwamoto I, Ito H, Maki Y, Shirai K, Baba S, Maruyama K, Saitsu H, Kato M, Matsumoto M, Momiyama T, Nagata K
    • Journal Title

      Brain

      Volume: 141 Pages: 3098-3114

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Journal Article] MYCN de novo gain-of-function mutation in a patient with a novel megalencephaly syndrome.2018

    • Author(s)
      Kato K, Miya F, Hamada N, Negishi Y, Kishimoto N, Ozawa H, Ito H, Hori I, Hattori A, Okamoto N, Kato M, Tsunoda T, Kanemura Y, Kosaki K, Takahashi Y, Nagata K, Saitoh S.
    • Journal Title

      J. Med. Genet

      Volume: 0 Pages: 1-8

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Journal Article] MUNC18-1 gene abnormalities are involved in neurodevelopmental disorders through defective cortical architecture during brain development.2017

    • Author(s)
      Hamada N, Iwamoto I, Tabata H, Nagata K.
    • Journal Title

      Acta Neuropathologica Comm.

      Volume: 5

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Journal Article] Expression analyses of Dusp22 (Dual-specificity phosphatase 22) in mouse tissues2017

    • Author(s)
      Hamada N, Mizuno M, Tomita H, Iwamoto I, Hara A, Nagata K
    • Journal Title

      Med. Mol. Morphol.

      Volume: 418 Pages: 475-481

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Journal Article] MUNC18-1 gene abnormalities are involved in neurodevelopmental disorders through defective cortical architecture during brain development2017

    • Author(s)
      Hamada N, Iwamoto I, Tabata H, Nagata K.
    • Journal Title

      Acta Neuropathologica Comm.

      Volume: 5

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Journal Article] Expression analyses of Dusp22 (Dual-specificity phosphatase 22) in mouse tissues2017

    • Author(s)
      Hamada N, Mizuno M, Tomita H, Iwamoto I, Hara A, Nagata K
    • Journal Title

      Med. Mol. Morphol.

      Volume: 418 Pages: 475-781

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Journal Article] Essential role of the nuclear isoform of RBFOX1, a candidate gene for autism spectrum disorders, in the brain development.2016

    • Author(s)
      Hamada N, Ito H, Nishijo T, Iwamoto I, Morishita R, Tabata H, Momiyama T, Nagata K.
    • Journal Title

      Sci. Rep.

      Volume: 2 Pages: 00-00

    • Peer Reviewed / Acknowledgement Compliant / Open Access
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Journal Article] Role of a heterotrimeric G-protein, Gi2, in the corticogenesis: possible involvement in periventricular nodular heterotopia and intellectual disability.2016

    • Author(s)
      Hamada N, Negishi Y, Mizuno M, Miya F, Hattori A, Okamoto N, Kato M, Tsunoda T, Yamasaki M, Kanemura Y, Kosaki K, Tabata H, Saitoh S, and Nagata K.
    • Journal Title

      J. Neurochem.

      Volume: 140 Pages: 82-95

    • Peer Reviewed / Acknowledgement Compliant
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Journal Article] 発達障害の背景としての大脳皮質構築異常2015

    • Author(s)
      浜田奈々子、稲熊裕、永田浩一
    • Journal Title

      生化学

      Volume: 87 Pages: 205-208

    • NAID

      40020461541

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Journal Article] Role of the cytoplasmic isoform of RBFOX1/A2BP1 in establishing the architecture of the developing cerebral cortex.2015

    • Author(s)
      1.Hamada N, Ito H, Iwamoto I, Morishita R, Tabata H, Nagata K
    • Journal Title

      Mol. Autism.

      Volume: 6 Pages: 00-00

    • Peer Reviewed / Acknowledgement Compliant / Open Access
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Journal Article] Role of an adaptor protein Lin-7B in brain development: possible involvement in autism spectrum disorders2015

    • Author(s)
      Mizuno M, Matsumoto A, Hamada N, Ito H, Miyauchi A, Jimbo E, Momoi M, Tabata H, Yamagata T and Nagata K
    • Journal Title

      J. Neurochemistry

      Volume: 132 Pages: 61-69

    • Peer Reviewed / Acknowledgement Compliant
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] CEP152変異マウスを用いた小頭症/Seckel症候群の病態形成機構の解明2023

    • Author(s)
      浜田奈々子, 西條琢真, 永田浩一
    • Organizer
      日本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 中心体タンパク質CEP152とPLK4の相互作用と機能破綻が引き起こす小頭症/Seckel症候群の病態形成機構の解明2023

    • Author(s)
      浜田奈々子、岩本郁子、永田浩一
    • Organizer
      日本小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] CEP152変異マウスを用いた小頭症/Seckel症候群の病態形成機構の解明2023

    • Author(s)
      浜田奈々子, 西條琢真, 永田浩一
    • Organizer
      日本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-23K07284
  • [Presentation] Expression analyses of Cep152 and Plk4, a responsible gene product for autosomal recessive primary microcephaly, during mouse brain development2023

    • Author(s)
      浜田奈々子, 永田浩一
    • Organizer
      日本神経科学会大会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 中心体タンパク質CEP152とPLK4の相互作用と機能破綻が引き起こす小頭症/Seckel症候群の病態形成機構の解明2023

    • Author(s)
      浜田奈々子、岩本郁子、永田浩一
    • Organizer
      日本小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-23K07284
  • [Presentation] 発達障害原因遺伝子MED13Lの神経発達における役割と遺伝子変異がもたらす病態形成機構の解明2023

    • Author(s)
      浜田奈々子, 永田浩一
    • Organizer
      日本小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 小頭症・セッケル症候群の原因遺伝子 Polo-like kinase 4 (Plk4)のマウス発達脳における発現解析2023

    • Author(s)
      浜田奈々子, 永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] Expression analyses of Cep152 and Plk4, a responsible gene product for autosomal recessive primary microcephaly, during mouse brain development2023

    • Author(s)
      浜田奈々子, 永田浩一
    • Organizer
      日本神経科学学会
    • Data Source
      KAKENHI-PROJECT-23K07284
  • [Presentation] 小頭症/セッケル症候群の原因遺伝子CEP152の新規遺伝子変異同定と病態形成機構の解明2023

    • Author(s)
      浜田奈々子、西條琢真、上原朋子、武内俊樹、小崎健次郎、水野誠司、永田浩一
    • Organizer
      日本小児科学会
    • Data Source
      KAKENHI-PROJECT-23K07284
  • [Presentation] 小頭症・セッケル症候群の原因遺伝子 Polo-like kinase 4 (Plk4)のマウス発達脳における発現解析2023

    • Author(s)
      浜田奈々子, 永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-23K07284
  • [Presentation] 小頭症/セッケル症候群の原因遺伝子CEP152の新規遺伝子変異同定と病態形成機構の解明2023

    • Author(s)
      浜田奈々子, 西條琢真, 上原朋子, 武内俊樹, 小崎健次郎, 水野誠司, 永田浩一
    • Organizer
      日本小児科学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 電気穿孔法を用いた神経発達障害の病態解析2022

    • Author(s)
      浜田奈々子、伊東秀記、永田浩一
    • Organizer
      日本臨床分子形態学会
    • Invited
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 小頭症/セッケル症候群の原因遺伝子CEP152の新規遺伝子変異同定と病態形成機構の解明2022

    • Author(s)
      浜田奈々子、水野誠司、永田浩一
    • Organizer
      日本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] CEP152遺伝子変異を有する小頭症/セッケル症候群の病態形成機構の解明2022

    • Author(s)
      浜田奈々子, 西條琢真, 西川将司, 水野誠司, 永田浩一
    • Organizer
      神経組織培養研究会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] CEP152-deficiency promotes impaired centrosome duplication- and cell death-dependent dwarfism in mice2022

    • Author(s)
      浜田奈々子, 西條琢磨, 西川将司, 上原朋子, 武内俊樹, 小崎健次郎, 水野誠司,永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 発達障害関連遺伝子GNAI1の神経発達における機能解明2021

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] 発達障害関連遺伝子GNAI1の神経発達における機能解明2021

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] Essential role of MED13L, a responsible gene for autism spectrum disorders, in brain development.2021

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 達障害責任遺伝子MED13Lの神経発達における役割と遺伝子変異がもたらす病態機能解析2021

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 小頭症/Seckel症候群の原因遺伝子CEP152の新規遺伝子変異同定と病態形成メカニズムの解明2021

    • Author(s)
      浜田奈々子、西條琢磨、西川将司、上原朋子、武内俊樹、小崎健次郎、水野誠司、永田浩一
    • Organizer
      日本小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] Expression and functional analyses of MED13L during mouse brain development.2021

    • Author(s)
      Nanako Hamada, Koh-ichi Nagata
    • Organizer
      SIMONS FOUNDATION MED13L Scientific Meeting
    • Invited / Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] Role of a heterotrimeric G-protein, Gαi1, regulates neurogenesis, migration and development in cortical excitatory neurons.2020

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本神経科学大会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Role of a heterotrimeric G-protein, Gαi1, regulates neurogenesis, migration and development in cortical excitatory neurons.2020

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本神経科学大会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] Role of a heterotrimeric G-protein, Gi1, in the corticogenesis.2020

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] 三量体Gタンパク質Gi1の神経発達における形態学的機能解析2020

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] 三量体Gタンパク質Gi1の神経発達における形態学的機能解析2020

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-20K08200
  • [Presentation] De novo PHACTR1 mutations in West syndrome and their pathophysiological effects2020

    • Author(s)
      浜田奈々子、大萱俊介、中島光子、西條琢磨、才津浩智、加藤光広、松本直通、籾山俊彦、永田浩一
    • Organizer
      CIBoGリトリート
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Role of a heterotrimeric G-protein, Gi1, in the corticogenesis.2020

    • Author(s)
      浜田奈々子、永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] 大脳皮質神経細胞の樹状突起形成におけるGαi1の役割2019

    • Author(s)
      浜田奈々子、岩本郁子、河村則子、田畑秀典、永田浩一
    • Organizer
      日本神経科学会・日本神経化学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] De novo PHACTR1 mutations in West Syndrome and their pathophysiological effects.2019

    • Author(s)
      浜田奈々子、大萱俊介、中島光子、西條琢磨、才津浩智、加藤光広、松本直通、籾山俊彦、永田浩一
    • Organizer
      The 20th Annual Meeting of Infantile Seizure Society
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] ウエスト症候群の新規責任遺伝子の同定と病態機能解析2019

    • Author(s)
      浜田奈々子
    • Organizer
      小児遺伝学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] 小児難治性てんかん原因遺伝子の病態生理解析2019

    • Author(s)
      浜田奈々子、大萱俊介、牧 祐輝、丸山幸一、永田浩一
    • Organizer
      日本臨床分子形態学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Identification of PHACTR1 as a novel causal gene for West Syndrome and pathophysiological significance of the gene mutations2018

    • Author(s)
      浜田奈々子
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] 小児難治性てんかん責任遺伝子の病態生理解析2018

    • Author(s)
      浜田奈々子
    • Organizer
      生理学研究所研究会「神経発達・再生研究会」
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Pathophysiological significance of early infantile epilepsies caused by MUNC18-1 mutations2017

    • Author(s)
      Koh-Ichi Nagata and Nanako Hamada
    • Organizer
      48th ASN Annual Meeting
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Role of MUNC18-1 in brain development and involvement in early infantile epilepsies2017

    • Author(s)
      Koh-Ichi Nagata and Nanako Hamada
    • Organizer
      Excitatory Synapses & Brain Function
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Role of Munc18-1 in cortical neuron migration2017

    • Author(s)
      浜田奈々子、田畑秀典、永田浩一
    • Organizer
      日本生化学会
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Pathophysiological role of MUNC18-1 in early infantile epilepsies2017

    • Author(s)
      浜田奈々子、田畑秀典、永田浩一
    • Organizer
      第9回名古屋グローバルリトリート
    • Place of Presentation
      大府
    • Year and Date
      2017-02-10
    • Invited
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Role of Munc18-1 in cortical neuron migration2017

    • Author(s)
      浜田奈々子、田畑秀典、永田浩一
    • Organizer
      日本生化学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Pathophysiological mechanism of MUNC18-1 mutations in early infantile epilepsies2017

    • Author(s)
      Koh-Ichi Nagata and Nanako Hamada
    • Organizer
      ISN-ESN Meeting
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] athophysiological significance of early infantile epilepsies caused by MUNC18-1 mutations2017

    • Author(s)
      Koh-Ichi Nagata and Nanako Hamada
    • Organizer
      48th ASN Annual Meeting
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Role of MUNC18-1 in brain development and involvement in early infantile epilepsies2017

    • Author(s)
      Koh-Ichi Nagata and Nanako Hamada
    • Organizer
      Excitatory Synapses & Brain Function(国際学会)
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Munc18-1 plays an essential role for cortical neuron migration during brain development2017

    • Author(s)
      浜田奈々子、田畑秀典、永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Pathophysiological mechanism of MUNC18-1 mutations in early infantile epilepsies2017

    • Author(s)
      Koh-Ichi Nagata and Nanako Hamada
    • Organizer
      ISN-ESN Meeting
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Munc18-1 plays an essential role for cortical neuron migration during brain development2017

    • Author(s)
      浜田奈々子、田畑秀典、永田浩一
    • Organizer
      日本神経化学会
    • Data Source
      KAKENHI-PROJECT-17K16294
  • [Presentation] Rbfox1, an autism causal gene, plays an essential role in cortical development2015

    • Author(s)
      浜田奈々子、伊東秀記、田畑秀典、永田浩一
    • Organizer
      International Society for Neurochemistry
    • Place of Presentation
      Cairns, Australia
    • Year and Date
      2015-08-23
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] 自閉症・知的障害原因遺伝子A2BP1の大脳皮質形成における機能解析

    • Author(s)
      浜田奈々子、伊東秀記、田畑秀典、永田浩一
    • Organizer
      日本生化学会
    • Place of Presentation
      京都
    • Year and Date
      2014-10-15 – 2014-10-18
    • Data Source
      KAKENHI-PROJECT-26860839
  • [Presentation] Autism risk gene, A2BP1, plays an essential role in cortical development

    • Author(s)
      浜田奈々子、田畑秀典、伊東秀記、永田浩一
    • Organizer
      日本神経科学会
    • Place of Presentation
      横浜
    • Year and Date
      2014-09-11 – 2014-09-14
    • Data Source
      KAKENHI-PROJECT-26860839
  • 1.  NODA Mariko
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results
  • 2.  伊東 秀記
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 2 results
  • 3.  永田 浩一
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 3 results
  • 4.  西川 将司
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 2 results

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