Torii Satoru 鳥居暁

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TORII Satoru 鳥居暁

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Researcher Number	10444001
Other IDs	https://orcid.org/0000-0003-4841-7131
Affiliation (Current)	2024: 東京科学大学, 総合研究院, プロジェクト准教授
Affiliation (based on the past Project Information) *help	2023: 東京医科歯科大学, 難治疾患研究所, プロジェクト准教授 2020: 東京医科歯科大学, 難治疾患研究所, プロジェクト准教授 2018 – 2019: 東京医科歯科大学, 難治疾患研究所, プロジェクト講師 2016: 東京医科歯科大学, 難治疾患研究所, プロジェクト講師 2014 – 2015: 東京医科歯科大学, 難治疾患研究所, 講師 … More 2012: 東北大学, 大学院・生命科学研究科, 助教 2011 – 2012: 東北大学, 生命科学研究科, 助教 2007 – 2008: Tohoku University, 大学院・生命科学研究科, 助教 Less
Review Section/Research Field	Principal Investigator Basic Section 44010:Cell biology-related / Nerve anatomy/Neuropathology / Neurochemistry/Neuropharmacology / Cell biology
Keywords	Principal Investigator リン酸化 / Ulk1 / オートファジー / 神経変性疾患 / ゴルジ体 / 変異マウス / キナーゼ / Atg5 / RIPK3 / MPTP … More / Ｐａｒｋｉｎ / ＰＩＮＫ１ / パーキンソン病 / Parkin / PINK1 / IPAS / アポトーシス / 細胞内情報伝達 / ストレス / HIF-1 / 転写調節 / 低酸素 / 頸動脈小体 / 副腎 / マグネシウム / 低酸素応答 Less

Elucidation of physiological functions of Ulk1 phosphorylation which acts as a molecular switch leading to alternative autophagyPrincipal Investigator
- Principal Investigator
  
  鳥居暁
- Project Period (FY)
  2023 – 2025
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 44010:Cell biology-related
- Research Institution
  Tokyo Medical and Dental University
Analysis of Ulk1 phosphorylation to control alternative autophagy and elucidation of its physiological and pathological significancePrincipal Investigator
- Principal Investigator
  
  Torii Satoru
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 44010:Cell biology-related
- Research Institution
  Tokyo Medical and Dental University
Elucidation of pathogenic mechanism of Parkinson's disease through analysis of IPAS, a novel substrate of ParkinPrincipal Investigator
- Principal Investigator
  
  Torii Satoru
- Project Period (FY)
  2014 – 2016
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Nerve anatomy/Neuropathology
- Research Institution
  Tokyo Medical and Dental University
Functional analysis on the molecular and cellular biology of IPAS which induces neuronal cell death and production of its knockout mousePrincipal Investigator
- Principal Investigator
  
  TORII Satoru
- Project Period (FY)
  2011 – 2012
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Neurochemistry/Neuropharmacology
- Research Institution
  Tohoku University
Analysis of extracellular environment-dependent regulatory mechanism for intracellular signaling regarding hypoxic responsePrincipal Investigator
- Principal Investigator
  
  TORII Satoru
- Project Period (FY)
  2007 – 2009
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Cell biology
- Research Institution
  Tohoku University

All 2021 2020 2019 2018 2016 2015 2012 2011 2009 2008 Other

All Journal Article Presentation

[Journal Article] Homeostatic p62 levels and inclusion body formation in CHCHD2 knockout mice2021
- Author(s)
  Sato Shigeto、Noda Sachiko、Torii Satoru、Amo Taku、Ikeda Aya、Funayama Manabu、Yamaguchi Junji、Fukuda Takahiro、Kondo Hiromi、Tada Norihiro、Arakawa Satoko、Watanabe Masahiko、Uchiyama Yasuo、Shimizu Shigeomi、Hattori Nobutaka
- Journal Title
  
  Human Molecular Genetics
  
  Volume: 30 Issue: 6 Pages: 443-453
- DOI
  10.1093/hmg/ddab057
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20K07878, KAKENHI-PROJECT-19K22603, KAKENHI-PROJECT-18H04043, KAKENHI-PROJECT-17K09769, KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-19K08003, KAKENHI-PROJECT-20K22744, KAKENHI-PLANNED-17H06414, KAKENHI-PUBLICLY-20H05314, KAKENHI-PROJECT-18K05691, KAKENHI-PROJECT-20K07353, KAKENHI-PROJECT-21K15198, KAKENHI-PROJECT-20H00467
[Journal Article] Receptor-Interacting Protein Kinase 3 (RIPK3) inhibits autophagic flux during necroptosis in intestinal epithelial cells.2020
- Author(s)
  Otsubo K, Maeyashiki C, Nibe Y, Tamura A, Aonuma E, Matsuda H, Kobayashi M, Onizawa M, Nemoto Y, Nagaishi T, Okamoto R, Tsuchiya K, Nakamura T, Torii S, Itakura E, Watanabe M, Oshima S.
- Journal Title
  
  FEBS letters
  
  Volume: 594 Issue: 10 Pages: 1586-1595
- DOI
  10.1002/1873-3468.13748
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-19K08364, KAKENHI-PROJECT-19K22413, KAKENHI-PROJECT-19H01050, KAKENHI-PROJECT-16H06167, KAKENHI-PROJECT-17K09372, KAKENHI-PROJECT-19K08388, KAKENHI-PROJECT-19K08438, KAKENHI-PROJECT-19K08461, KAKENHI-PROJECT-20K08304, KAKENHI-PROJECT-20H03658, KAKENHI-PROJECT-18K07934, KAKENHI-PROJECT-19K17426, KAKENHI-PROJECT-17H04024, KAKENHI-PROJECT-17K19675
[Journal Article] Mitochondrial E3 Ubiquitin Ligase Parkin: Relationships with Other Causal Proteins in Familial Parkinson's Disease and Its Substrate-Involved Mouse Experimental Models.2020
- Author(s)
  Torii S, Kasai S, Yoshida T, Yasumoto KI, Shimizu S.
- Journal Title
  
  International journal of molecular sciences
  
  Volume: 21 Issue: 4 Pages: 1202-1202
- DOI
  10.3390/ijms21041202
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K06210
[Journal Article] Association Between Atg5-independent Alternative Autophagy and Neurodegenerative Diseases.2020
- Author(s)
  Honda S, Arakawa S, Yamaguchi H, Torii S, Tajima Sakurai H, Tsujioka M, Murohashi M, Shimizu S.
- Journal Title
  
  Journal of molecular biology
  
  Volume: 432 Issue: 8 Pages: 2622-2632
- DOI
  10.1016/j.jmb.2020.01.016
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-19K08859, KAKENHI-PROJECT-19K16119, KAKENHI-PROJECT-19K07382, KAKENHI-PROJECT-20K07353, KAKENHI-PUBLICLY-20H05314
[Journal Article] Identification of a phosphorylation site on Ulk1 required for genotoxic stress-induced alternative autophagy2020
- Author(s)
  Torii Satoru、Yamaguchi Hirofumi、Nakanishi Akira、Arakawa Satoko、Honda Shinya、Moriwaki Kenta、Nakano Hiroyasu、Shimizu Shigeomi
- Journal Title
  
  Nature Communications
  
  Volume: 11 Issue: 1 Pages: 1754-1754
- DOI
  10.1038/s41467-020-15577-2
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17H01533, KAKENHI-PROJECT-17H04069, KAKENHI-PROJECT-19K07382, KAKENHI-PROJECT-19K07399, KAKENHI-PROJECT-20K07353, KAKENHI-PUBLICLY-20H05314, KAKENHI-PROJECT-20H03475, KAKENHI-PROJECT-18K06210, KAKENHI-ORGANIZER-17H06413, KAKENHI-PLANNED-17H06414
[Journal Article] Autophagy involvement in oncogenesis2020
- Author(s)
  Torii Satoru、Honda Shinya、Murohashi Michiko、Yamaguchi Hirofumi、Shimizu Shigeomi
- Journal Title
  
  Cancer Science
  
  Volume: 111 Issue: 11 Pages: 3993-3999
- DOI
  10.1111/cas.14646
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-19K07382, KAKENHI-PROJECT-19K08859, KAKENHI-PROJECT-18K06210
[Journal Article] Wipi3 is essential for alternative autophagy and its loss causes neurodegeneration2020
- Author(s)
  Yamaguchi Hirofumi、Honda Shinya、Torii Satoru、Shimizu Kimiko、Katoh Kaoru、Miyake Koichi、Miyake Noriko、Fujikake Nobuhiro、Sakurai Hajime Tajima、Arakawa Satoko、Shimizu Shigeomi
- Journal Title
  
  Nature Communications
  
  Volume: 11 Issue: 1 Pages: 5311-5311
- DOI
  10.1038/s41467-020-18892-w
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-19K07382, KAKENHI-PROJECT-19K08859, KAKENHI-PROJECT-20K07353, KAKENHI-PUBLICLY-20H05314, KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-19K16119, KAKENHI-ORGANIZER-17H06413, KAKENHI-PLANNED-17H06417
[Journal Article] ER-resident sensor PERK is essential for mitochondrial thermogenesis in brown adipose tissue.2020
- Author(s)
  Kato H, Okabe K, Miyake M, Hattori K, Fukaya T, Tanimoto K, Beini S, Mizuguchi M, Torii S, Arakawa S, Ono M, Saito Y, Sugiyama T, Funatsu T, Sato K, Shimizu S, Oyadomari S, Ichijo H, Kadowaki H, Nishitoh H.
- Journal Title
  
  Life science alliance
  
  Volume: 3 Issue: 3 Pages: 201900576-201900576
- DOI
  10.26508/lsa.201900576
- NAID
  120006980545
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-18K06222, KAKENHI-PROJECT-19K08843, KAKENHI-PLANNED-15H05931, KAKENHI-PLANNED-17H06414, KAKENHI-PLANNED-17H06419, KAKENHI-PUBLICLY-18H04699, KAKENHI-PROJECT-17H01533, KAKENHI-PROJECT-18H02973, KAKENHI-PROJECT-19H02853, KAKENHI-PROJECT-17K09862, KAKENHI-PROJECT-18K06916, KAKENHI-PROJECT-18K19469, KAKENHI-PROJECT-20K07353, KAKENHI-PROJECT-18H03995, KAKENHI-PUBLICLY-20H05314, KAKENHI-ORGANIZER-17H06413
[Journal Article] Involvement of phosphorylation of ULK1 in alternative autophagy2020
- Author(s)
  Torii Satoru、Shimizu Shigeomi
- Journal Title
  
  Autophagy
  
  Volume: 16 Issue: 8 Pages: 1532-1533
- DOI
  10.1080/15548627.2020.1776476
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-18K06210
[Journal Article] Role of cyclooxygenase-2-mediated prostaglandin E2-prostaglandin E receptor 4 signaling in cardiac reprogramming2019
- Author(s)
  Muraoka Naoto、Nara Kaori、Tamura Fumiya、Kojima Hidenori、Yamakawa Hiroyuki、Sadahiro Taketaro、Miyamoto Kazutaka、Isomi Mari、Haginiwa Sho、Tani Hidenori、Kurotsu Shota、Osakabe Rina、Torii Satoru、Shimizu Shigeomi、Okano Hideyuki、Sugimoto Yukihiko、Fukuda Keiichi、Ieda Masaki
- Journal Title
  
  Nature Communications
  
  Volume: 10 Issue: 1 Pages: 674-674
- DOI
  10.1038/s41467-019-08626-y
- NAID
  120007128245
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K06210, KAKENHI-PROJECT-18K08047, KAKENHI-PROJECT-18K08114, KAKENHI-PROJECT-17K16024, KAKENHI-ORGANIZER-15H05897, KAKENHI-PLANNED-15H05905, KAKENHI-PLANNED-17H06414, KAKENHI-PROJECT-17H01533, KAKENHI-PROJECT-17H03990, KAKENHI-PROJECT-17J06637, KAKENHI-PROJECT-18J21479, KAKENHI-PROJECT-15K09147, KAKENHI-PROJECT-16K19426, KAKENHI-PROJECT-19K22503
[Journal Article] Autophagy controls centrosome number by degrading Cep63.2016
- Author(s)
  Watanabe Y, Honda S, Konishi A, Satoko Arakawa S, Murohashi M, Yamaguchi H, Torii S, Tanabe M, Tanaka S, Warabi E, Shimizu S.
- Journal Title
  
  Nature Communications
  
  Volume: 7 Issue: 1 Pages: 13508-13508
- DOI
  10.1038/ncomms13508
- NAID
  120007129257
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PUBLICLY-15H01484, KAKENHI-PUBLICLY-15H01554, KAKENHI-PLANNED-26110005, KAKENHI-PROJECT-25220103, KAKENHI-PROJECT-16H02670, KAKENHI-PROJECT-15H04926, KAKENHI-PROJECT-25440080, KAKENHI-PROJECT-26430051, KAKENHI-PROJECT-26460485, KAKENHI-PROJECT-15K15491, KAKENHI-PROJECT-15K19004, KAKENHI-PROJECT-15K08420, KAKENHI-PROJECT-16K15230
[Journal Article] Identification of PPM1D as an essential Ulk1 phosphatase for genotoxic stress-induced autophagy2016
- Author(s)
  Torii S, Yoshida T, Arakawa S, Honda S, Nakanishi A, Shimizu S
- Journal Title
  
  EMBO reports
  
  Volume: 17 Pages: 1552-1564
- Peer Reviewed / Acknowledgement Compliant
- Data Source
  KAKENHI-PROJECT-26430051
[Journal Article] Inhibitory PAS domain protein is a substrate of PINK1 and Parkin and mediates cell death in a Parkinson’s disease model2015
- Author(s)
  Shuya Kasai, Satoru Torii, Akiyoshi Kakita, Kazuhiro Sogawa
- Journal Title
  
  Cell Death & Disease
  
  Volume: 6 Issue: 9 Pages: 1-3
- DOI
  10.1038/cddis.2015.243
- Open Access
- Data Source
  KAKENHI-PROJECT-26430051
[Journal Article] Involvement of inhibitory PAS domain protein in neuronal cell death in Parkinson’s disease2015
- Author(s)
  Satoru Torii, Shuya Kasai, Ayako Suzuki, Yuta Todoroki, Kazuma Yokosawa, Ken-ichi Yasumoto, Hiroshi Kiyonari, Yoshiko Mukumoto, Akiyoshi Kakita, Kazuhiro Sogawa
- Journal Title
  
  Cell Death Discovery
  
  Volume: １ Issue: 1 Pages: 1-12
- DOI
  10.1038/cddiscovery.2015.15
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-26430051
[Journal Article] PHD1 interacts with ATF4 and negatively regulates its transcriptional activity without prolyl hydroxylation2011
- Author(s)
  Hiwatashi Y, Kanno K, Takasaki C, Goryo K, Sato T, Torii S, Sogawa K, Yasumoto K
- Journal Title
  
  Experimental Cell Research
  
  Volume: 317 Issue: 20 Pages: 2789-2799
- DOI
  10.1016/j.yexcr.2011.09.005
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23700437
[Journal Article] Pro-apoptotic activity of inhibitory PAS domain protein (IPAS), a negative regulator of HIF-1, through binding to pro-survival Bcl-2 family proteins2011
- Author(s)
  Torii S, Gotoh Y, Ishizawa T, Hoshi H, Goryo K, Yasumoto K, Fukumura H, Sogawa K
- Journal Title
  
  Cell Death & Differentiation
  
  Volume: 18 Issue: 11 Pages: 1711-1725
- DOI
  10.1038/cdd.2011.47
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23700437
[Journal Article] Tumor necrosis factor-α suppresses the hypoxic response by NF-κB-dependent induction of inhibitory PAS domain protein (IPAS) in PC12 cells2011
- Author(s)
  Goryo K, Torii S, Yasumoto K, Sogawa K
- Journal Title
  
  Journal of Biochemistry
  
  Volume: 150 Issue: 3 Pages: 311-318
- DOI
  10.1093/jb/mvr061
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23700437
[Journal Article] Role of the intracellular localization of HIF-prolyl hydroxylases2009
- Author(s)
  安元研一、木幡勇介、吉田敦、鳥居暁、十川研一
- Journal Title
  
  Biochimica et Biophysica Acta-Molecular Cell Research (印刷中)
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-19770158
[Journal Article] Role of the intracellular localization of HIF-prolyl hydroxylases2009
- Author(s)
  安元研一、木幡勇介、吉田敦、鳥居暁、十川和博
- Journal Title
  
  Biochimica et Biophysica Acta 1793
  
  Pages: 792-797
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-19770158
[Journal Article] Magnesium Deficiency Causes Loss of Response to Intermittent Hypoxia in Paraganglion Cells
- Author(s)
  鳥居暁、小林健太郎、高橋正之、片平香澄美、御領憲治、松下夏樹、安元研一、藤井義明、十川和博
- Journal Title
  
  Journal of Biological Chemistry (in Press)
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-19770158
[Presentation] RIPK3によるDNA傷害依存的な新規オートファジーの制御.2020
- Author(s)
  鳥居暁, 山口啓史, 中西啓, 荒川聡子, 本田真也, 森脇健太, 中野裕康, 清水重臣.
- Organizer
  第93回日本生化学会大会
- Invited
- Data Source
  KAKENHI-PROJECT-18K06210
[Presentation] 新規オートファジーに必須なUlk1のリン酸化とネクロプトーシス実行因子RIPK3の関与.2020
- Author(s)
  鳥居暁, 山口啓史, 中西啓, 荒川聡子, 本田真也, 森脇健太, 中野裕康, 清水重臣.
- Organizer
  第１回細胞死コロキュアム
- Invited
- Data Source
  KAKENHI-PROJECT-18K06210
[Presentation] Regulation of phosphorylation of Ulk1 in alternative autophagy.2020
- Author(s)
  Torii S, Yamaguchi H, Nakanishi A, Arakawa S, Honda S, Moriwaki K, Nakano H, and Shimizu S.
- Organizer
  The 15th International Symposium of the Institute Network for Biomedical Sciences.
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K06210
[Presentation] Regulation of DNA damage-induced apoptosis by autophagy.2018
- Author(s)
  Torii S, Yoshida T, Arakawa S, Honda S and Shimizu S.
- Organizer
  Australia-Japan Meeting on Cell Death.
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K06210
[Presentation] オートファジーによるDNA傷害誘導性アポトーシスの制御2018
- Author(s)
  鳥居暁, 吉田達士, 荒川聡子, 本田真也, 清水重臣.
- Organizer
  第27回日本Cell death学会学術集会
- Invited
- Data Source
  KAKENHI-PROJECT-18K06210
[Presentation] The role of PPM1D in regulating DNA damage-induced autophagy.2018
- Author(s)
  Torii S, Yoshida T, Arakawa S, Honda S and Shimizu S.
- Organizer
  Workshop on Frontiers in Phosphatase Research and Drug Discovery (ICPP13)
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K06210
[Presentation] DNA傷害誘導性オートファジーを制御するPPM1D分子の役割2016
- Author(s)
  鳥居暁,　吉田達士,　荒川聡子, 本田真也, 清水重臣
- Organizer
  第39回日本分子生物学会
- Place of Presentation
  パシフィコ横浜（神奈川県横浜市）
- Year and Date
  2016-11-30
- Data Source
  KAKENHI-PROJECT-26430051
[Presentation] DNA傷害誘導性オートファジーを制御するPPM1D分子の役割2016
- Author(s)
  鳥居暁,　吉田達士,　荒川聡子, 本田真也, 清水重臣
- Organizer
  第10回オートファジー研究会
- Place of Presentation
  NASPAニューオータニ（新潟県南魚沼郡湯沢町）
- Year and Date
  2016-11-13
- Data Source
  KAKENHI-PROJECT-26430051
[Presentation] HIF-3alphaの選択的スプライスバリアントであるIPASの細胞内局在とその制御機構2012
- Author(s)
  鳥居暁、安元研一、十川和博
- Organizer
  第85回日本生化学会大会
- Place of Presentation
  福岡(招待講演)
- Year and Date
  2012-12-14
- Data Source
  KAKENHI-PROJECT-23700437
[Presentation] Pro-apoptotic activity of inhibitory PAS domain protein (IPAS), a negative regulator of HIF-1, through binding to pro-survival Bcl-2 family proteins2012
- Author(s)
  鳥居暁
- Organizer
  内藤コンファレンス
- Place of Presentation
  札幌
- Data Source
  KAKENHI-PROJECT-23700437
[Presentation] IPAS acts as a dual function protein involved in transcription repression and apoptosis2011
- Author(s)
  Satoru Torii
- Organizer
  The 3rd UCL - Tohoku University Joint Symposium
- Place of Presentation
  University College London, ロンドン, イギリス
- Data Source
  KAKENHI-PROJECT-23700437
[Presentation] IPAS acts as a dual function protein involved in transcriptional repression and apoptosis2011
- Author(s)
  鳥居暁
- Organizer
  The 3rd UCL-Tohoku University Joint Symposium
- Place of Presentation
  University CollegeLondon、ロンドン、イギリス
- Year and Date
  2011-10-19
- Data Source
  KAKENHI-PROJECT-23700437
[Presentation] 慢性間欠的低酸素応答性チロシン水酸化酵素誘導に対するマグネシウム要求性に関する解析2008
- Author(s)
  鳥居暁
- Organizer
  BMB2008
- Place of Presentation
  神戸
- Year and Date
  2008-12-12
- Data Source
  KAKENHI-PROJECT-19770158
[Presentation] 慢性間欠的低酸素応答性チロシン水酸化酵素誘導に対するマグネシウム要求性に関する研究2008
- Author(s)
  鳥居暁
- Organizer
  BMB2008
- Place of Presentation
  神戸
- Year and Date
  2008-12-12
- Data Source
  KAKENHI-PROJECT-19770158
[Presentation] オルタナティブオートファジーのケミカルバイオロジー
- Author(s)
  鳥居暁、室橋道子、荒川聡子、桜井一、清水重臣
- Organizer
  第8回オートファジー学会
- Place of Presentation
  日本、札幌
- Year and Date
  2014-11-09 – 2014-11-11
- Data Source
  KAKENHI-PROJECT-26430051
[Presentation] HIF-3alphaの選択的スプライスバリアントであるIPASの細胞内局在とその制御機構
- Author(s)
  鳥居暁、安元研一、十川和博
- Organizer
  第85回日本生化学会大会
- Place of Presentation
  福岡
- Invited
- Data Source
  KAKENHI-PROJECT-23700437

1. SOGAWA Kazuhiro (80175421)

# of Collaborated Projects: 1 results

# of Collaborated Products: 2 results
2. YASUMOTO Kenichi (90241629)

# of Collaborated Projects: 1 results

# of Collaborated Products: 1 results
3. FUNAYAMA Manabu

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
4. AMO Taku

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
5. 清水重臣

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
6. 山口啓史

# of Collaborated Projects: 0 results

# of Collaborated Products: 2 results
7. 田中真二

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

Torii Satoru 鳥居 暁

TORII Satoru 鳥居 暁

Research Projects

Research Products

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Elucidation of physiological functions of Ulk1 phosphorylation which acts as a molecular switch leading to alternative autophagyPrincipal Investigator

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Analysis of Ulk1 phosphorylation to control alternative autophagy and elucidation of its physiological and pathological significancePrincipal Investigator

Principal Investigator

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Elucidation of pathogenic mechanism of Parkinson's disease through analysis of IPAS, a novel substrate of ParkinPrincipal Investigator

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Functional analysis on the molecular and cellular biology of IPAS which induces neuronal cell death and production of its knockout mousePrincipal Investigator

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Analysis of extracellular environment-dependent regulatory mechanism for intracellular signaling regarding hypoxic responsePrincipal Investigator

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[Journal Article] Homeostatic p62 levels and inclusion body formation in CHCHD2 knockout mice2021

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[Journal Article] Receptor-Interacting Protein Kinase 3 (RIPK3) inhibits autophagic flux during necroptosis in intestinal epithelial cells.2020

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[Journal Article] Mitochondrial E3 Ubiquitin Ligase Parkin: Relationships with Other Causal Proteins in Familial Parkinson's Disease and Its Substrate-Involved Mouse Experimental Models.2020

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[Journal Article] Association Between Atg5-independent Alternative Autophagy and Neurodegenerative Diseases.2020

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[Journal Article] Identification of a phosphorylation site on Ulk1 required for genotoxic stress-induced alternative autophagy2020

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[Journal Article] Autophagy involvement in oncogenesis2020

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[Journal Article] Wipi3 is essential for alternative autophagy and its loss causes neurodegeneration2020

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[Journal Article] ER-resident sensor PERK is essential for mitochondrial thermogenesis in brown adipose tissue.2020

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NAID

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[Journal Article] Involvement of phosphorylation of ULK1 in alternative autophagy2020

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Torii Satoru 鳥居暁

TORII Satoru 鳥居暁