Hukuda Koji 福田康二

… Alternative Names

Researcher Number	10722548
Other IDs
Affiliation (Current)	2021: 金沢大学, がん進展制御研究所, 助教
Affiliation (based on the past Project Information) *help	2021: 金沢大学, がん進展制御研究所, 助教 2017 – 2019: 金沢大学, がん進展制御研究所, 助教 2015 – 2016: 金沢大学, がん進展制御研究所, 特任助手 2014: 金沢大学, がん進展制御研究所, 博士研究員
Review Section/Research Field	Principal Investigator Respiratory organ internal medicine / Basic Section 50020:Tumor diagnostics and therapeutics-related
Keywords	Principal Investigator EMT / miR-200 / ALK融合遺伝子陽性肺癌 / ALK / 薬剤耐性 / EGFR肺癌 / AXL / EGFR変異肺癌 / 第3世代-TKI / ALK肺がん … More

EGFR肺癌の髄膜癌腫症におけるEGFR-TKI耐性克服治療の開発Principal InvestigatorOngoing
- Principal Investigator
  
  福田康二
- Project Period (FY)
  2021 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 50020:Tumor diagnostics and therapeutics-related
- Research Institution
  Kanazawa University
miR-200sを標的とした肺癌のEMTに起因するTKI耐性克服治療の開発Principal InvestigatorOngoing
- Principal Investigator
  
  福田康二
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 50020:Tumor diagnostics and therapeutics-related
- Research Institution
  Kanazawa University
Development of ALK-TKI-resistant treatment due to EMT of ALK lung cancerPrincipal Investigator
- Principal Investigator
  
  Fukuda Koji
- Project Period (FY)
  2016 – 2017
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Respiratory organ internal medicine
- Research Institution
  Kanazawa University
Development of therapies for overcoming EMT-associated irreversible tyrosine kinase inhibitor resistance in EGFR lung cancerPrincipal Investigator
- Principal Investigator
  
  Fukuda Koji
- Project Period (FY)
  2014 – 2015
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Respiratory organ internal medicine
- Research Institution
  Kanazawa University

All 2020 2019 2018 2016

All Journal Article Presentation

[Journal Article] GSK-3 inhibition overcomes EMT-associated resistance to osimertinib in EGFR mutant lung cancer2020
- Author(s)
  Koji Fukuda, Shinji Takeuchi, Sachiko Arai, Kenji Kita, Azusa Tanimoto, Akihiro Nishiyama, Seiji Yano
- Journal Title
  
  Cancer Science
  
  Volume: -
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K07261
[Journal Article] Epithelial-to-Mesenchymal Transition Is a Mechanism of ALK Inhibitor Resistance in Lung Cancer Independent of ALK Mutation Status2019
- Author(s)
  Koji Fukuda, Shinji Takeuchi, Sachiko Arai, Ryohei Katayama, et al
- Journal Title
  
  Cancer Reseach
  
  Volume: 79 Pages: 1658-1670
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K07261
[Journal Article] Epithelial-to-Mesenchymal Transition Is a Mechanism of ALK Inhibitor Resistance in Lung Cancer Independent of ALK Mutation Status.2019
- Author(s)
  Fukuda K, Takeuchi S, Arai S, Katayama R, Nanjo S, Tanimoto A, Nishiyama A, Nakagawa T, Taniguchi H, Suzuki T, Yamada T, Nishihara H, Ninomiya H, Ishikawa Y, Baba S, Takeuchi K, Horiike A, Yanagitani N, Nishio M, Yano S.
- Journal Title
  
  Cancer Resarch
  
  Volume: 79 Pages: 1658-1670
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K07261
[Presentation] Epithelial-to-mesenchymal transition is a mechanism of ALK inhibitor resistance in lung cancer independent of ALK mutation status.2019
- Author(s)
  福田康二
- Organizer
  IASLC 2019 World Conference on Lung Cancer. 2019年9月
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K07261
[Presentation] 肺がんにおいて上皮間葉転換はALK阻害薬耐性メカニズムとして機能する.2019
- Author(s)
  福田康二
- Organizer
  第23回日本がん分子標的治療学会学術集会 2019年6月
- Data Source
  KAKENHI-PROJECT-18K07261
[Presentation] EMTに起因した肺癌の分子標的薬耐性の克服2018
- Author(s)
  福田康二
- Organizer
  第5回 Medical Frontier Consortium beyond the Organcentric Dogma
- Data Source
  KAKENHI-PROJECT-16K19447
[Presentation] Heterogeneity of epithelial-to-mesenchymal transition and resistance mutation in ALK inhibitor-resistant lung cancer and its circumvention2018
- Author(s)
  Koji Fukuda, Shinji Takeuchi, Sachiko Arai, Ryohei Katayama, Shigeki Nanjo, Azusa Tanimoto, Akihiro Nishiyama, Takeshi Suzuki, Kengo Takeuchi, Makoto Nishio, Seiji Yano.
- Organizer
  Fifth AACR-IASLC International Joint Conference
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16K19447
[Presentation] Heterogeneity of epithelial-to-mesenchymal transition and resistance mutation in ALK inhibitor-resistant lung cancer and its circumvention2018
- Author(s)
  Koji Fukuda, Shinji Takeuchi, Sachiko Arai, Ryohei Katayama, Shigeki Nanjo, Azusa Tanimoto, Akihiro Nishiyama, Takeshi Suzuki, Kengo Takeuchi, Makoto Nishio, Seiji Yano.
- Organizer
  The 1st NanoLSI International Symposium
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16K19447
[Presentation] ALK転座陽性肺癌においてEMTは独立した機構として関与する2018
- Author(s)
  福田康二、竹内伸司、片山量平、南條成輝、山田忠明、鈴木健之、竹内健吾、西尾誠人、矢野聖二
- Organizer
  第77回日本癌学会学術総会 2018
- Data Source
  KAKENHI-PROJECT-18K07261
[Presentation] HDAC inhibition overcomes crizotinib-resistance by mesenchymal-epithelial transition (MET) in EML4-ALK lung cancer cells2016
- Author(s)
  Koji Fukuda, Shinji Takeuchi, Ryohei Katayama, Shigeki Nanjo, Kengo Takeuchi, Hiroshi Nishihara, Makoto Nishio, Seiji Yano.
- Organizer
  第75回日本癌学会学術総会
- Place of Presentation
  横浜
- Year and Date
  2016-10-06
- Data Source
  KAKENHI-PROJECT-16K19447
[Presentation] HDAC inhibition overcomes crizotinib-resistance by inducing mesenchymal-epithelial reverting transition (MERT) via miR-200c up-regulation in EML4-ALK lung cancer cells.2016
- Author(s)
  Koji Fukuda, Shigeki Nanjo, Shinji Takeuchi, Tadaaki Yamada, Ryohei Katayama, Kengo Takeuchi, Hiroshi Nishihara, Seiji Yano.
- Organizer
  Tenth AACR-JCA Joint Conference
- Place of Presentation
  Maui, Hawaii, USA
- Year and Date
  2016-02-16
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-26860603
[Presentation] HDAC inhibition overcomes crizotinib-resistance by mesenchymal-epithelial transition (MET) in EML4-ALK lung cancer cells2016
- Author(s)
  Koji Fukuda, Shinji Takeuchi, Ryohei Katayama, Shigeki Nanjo, Kengo Takeuchi, Hiroshi Nishihara, Makoto Nishio, Seiji Yano.
- Organizer
  IASLC 17th world conference on lung cancer
- Place of Presentation
  ウイーン、オーストリア
- Year and Date
  2016-12-04
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16K19447

1. Shinji Takeuchi (90565384)

# of Collaborated Projects: 2 results

# of Collaborated Products: 5 results
2. Seiji Yano (30294672)

# of Collaborated Projects: 1 results

# of Collaborated Products: 1 results

Hukuda Koji 福田 康二

福田 康二 フクダ コウジ

Fukuda Koji 福田 康二

Research Projects

Research Products

Co-Researchers

EGFR肺癌の髄膜癌腫症におけるEGFR-TKI耐性克服治療の開発Principal InvestigatorOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

miR-200sを標的とした肺癌のEMTに起因するTKI耐性克服治療の開発Principal InvestigatorOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Development of ALK-TKI-resistant treatment due to EMT of ALK lung cancerPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Development of therapies for overcoming EMT-associated irreversible tyrosine kinase inhibitor resistance in EGFR lung cancerPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] GSK-3 inhibition overcomes EMT-associated resistance to osimertinib in EGFR mutant lung cancer2020

Author(s)

Journal Title

Data Source

[Journal Article] Epithelial-to-Mesenchymal Transition Is a Mechanism of ALK Inhibitor Resistance in Lung Cancer Independent of ALK Mutation Status2019

Author(s)

Journal Title

Data Source

[Journal Article] Epithelial-to-Mesenchymal Transition Is a Mechanism of ALK Inhibitor Resistance in Lung Cancer Independent of ALK Mutation Status.2019

Author(s)

Journal Title

Data Source

[Presentation] Epithelial-to-mesenchymal transition is a mechanism of ALK inhibitor resistance in lung cancer independent of ALK mutation status.2019

Author(s)

Organizer

Data Source

[Presentation] 肺がんにおいて上皮間葉転換はALK阻害薬耐性メカニズムとして機能する.2019

Author(s)

Organizer

Data Source

[Presentation] EMTに起因した肺癌の分子標的薬耐性の克服2018

Author(s)

Organizer

Data Source

[Presentation] Heterogeneity of epithelial-to-mesenchymal transition and resistance mutation in ALK inhibitor-resistant lung cancer and its circumvention2018

Author(s)

Organizer

Data Source

[Presentation] Heterogeneity of epithelial-to-mesenchymal transition and resistance mutation in ALK inhibitor-resistant lung cancer and its circumvention2018

Author(s)

Organizer

Data Source

[Presentation] ALK転座陽性肺癌においてEMTは独立した機構として関与する2018

Author(s)

Organizer

Data Source

[Presentation] HDAC inhibition overcomes crizotinib-resistance by mesenchymal-epithelial transition (MET) in EML4-ALK lung cancer cells2016

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] HDAC inhibition overcomes crizotinib-resistance by inducing mesenchymal-epithelial reverting transition (MERT) via miR-200c up-regulation in EML4-ALK lung cancer cells.2016

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] HDAC inhibition overcomes crizotinib-resistance by mesenchymal-epithelial transition (MET) in EML4-ALK lung cancer cells2016

Author(s)

Hukuda Koji 福田康二

福田康二フクダコウジ

Fukuda Koji 福田康二