NOGUCHI Masayuki 野口昌幸

… Alternative Names

野口昌幸ノグチマサユキ

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Researcher Number	40359477
Other IDs
External Links
Affiliation (Current)	2025: 北海道大学, 遺伝子病制御研究所, 名誉教授
Affiliation (based on the past Project Information) *help	2010 – 2012: 北海道大学, 遺伝子病制御研究所, 教授 2007: Hokkaido University, Institute for Genetic Medicine, Professor 2005 – 2007: 北海道大学, 遺伝子病制御研究所, 教授 2005: 北海道大学, 遺伝子制御研究室, 教授 2003: 北海道大学, 遺伝子病制御研究所, 教授
Review Section/Research Field	Principal Investigator Functional biochemistry / Biological Sciences
Keywords	Principal Investigator 生体分子 / 遺伝子 / Protein / Nucleic acid / Enzyme reaction / Cancer / Gene / 蛋白質 / 核酸 / 酵素反応 … More / 癌 / Akt / シグナル伝達 / ＴＣＬ１ｂ / ＮＳ１ / 蛋白分解 / AKT / 酵素 / 抗腫瘍剤 / セリンスレオニン燐酸化阻害剤 / Akt (Protein Kinase B) / アポトーシス Less

Modulation of Akt kinase activity by ubiquitinationPrincipal Investigator
- Principal Investigator
  
  NOGUCHI Masayuki
- Project Period (FY)
  2010 – 2012
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  Functional biochemistry
- Research Institution
  Hokkaido University
発ガンとAKTキナーゼ活性化の分子機構Principal Investigator
- Principal Investigator
  
  野口昌幸
- Project Period (FY)
  2005
- Research Category
  
  Grant-in-Aid for Scientific Research on Priority Areas
- Review Section
  
  Biological Sciences
- Research Institution
  Hokkaido University
Characterization of the molecular mechanisms of Akt activationPrincipal Investigator
- Principal Investigator
  
  NOGUCHI Masayuki
- Project Period (FY)
  2005 – 2007
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  Functional biochemistry
- Research Institution
  Hokkaido University
ATOM機能の解明Principal Investigator
- Principal Investigator
  
  野口昌幸
- Project Period (FY)
  2003
- Research Category
  
  Grant-in-Aid for Scientific Research on Priority Areas
- Review Section
  
  Biological Sciences
- Research Institution
  Hokkaido University

All 2012 2011 2010 2008 2007 2006 2005

All Journal Article Presentation Book

[Book] Regulation of Akt by phosphorylation of distinct threonine and serine residues Threonine:Structure Biosynthesis and Functions; Advances in Medicine and Biology2011
- Author(s)
  M. Noguchi & F. Suizu.
- Publisher
  Nova Science Publishers, New York. USA
- Data Source
  KAKENHI-PROJECT-22370046
[Book] Regulation of Akt by phosphorylation of distinct threonine and serine residues Threonine : Structure, Biosynthesis and Functions ; Advances in Medicine and Biology2011
- Author(s)
  M.Noguchi, F.Suizu
- Publisher
  Nova Science Publishers
- Data Source
  KAKENHI-PROJECT-22370046
[Book] Regulation of the PI3K-Akt Network: Current Status and a Promise for the Treatment of Human Diseases.2008
- Author(s)
  M. Noguchi
- Publisher
  Curr Sig. Thera
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Book] バイオとナノの融合II2007
- Author(s)
  野口昌幸
- Publisher
  北海道大学出版会
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Book] バイオとナノの融合〔新生命科学の融合〕2007
- Author(s)
  野口昌幸
- Total Pages
  12
- Publisher
  北海道大学出版会
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] The role of tumor necrosis factor-α for interleukin-10 production by murine dendritic cells2011
- Author(s)
  N. Hirata, H.ogura, M.Satoh, M Noguchi, M.Matumoto,H.Togashi,K.Onoe,K.Iwabuc hi,Y.Yanagawa
- Journal Title
  
  Cell Immunol
  
  Volume: 266 Pages: 165-171
- NAID
  120002893812
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-22370046
[Journal Article] Regulation of Akt by phosphorylation of distinct threonine and serine residues　Threonine:Structure, Biosynthesis and Functions2011
- Author(s)
  M. Noguchi & F. Suizu
- Journal Title
  
  Advances in Medicine and Biology
  
  Volume: 47 Pages: 139-162
- Data Source
  KAKENHI-PROJECT-22370046
[Journal Article] Dendritic cell-derived TNF-αisresponsible for development of IL-10- producing CD4+ T cells2010
- Author(s)
  N.Hirata,Y.Yanagawa,M.Satoh,H.Ogura,T.Ebihara,M.Noguchi,M.Matsumoto,H.To gashi,T.Seya, K.Onoe,K.Iwabuchi
- Journal Title
  
  Cell Immunol.
  
  Volume: 261 Pages: 37-4
- NAID
  120001875433
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-22370046
[Journal Article] The roles of the PI3K-AKT network in infectious Diseases.2008
- Author(s)
  M. Noguchi
- Journal Title
  
  Jpn J. Infect Dis.
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] Regulation of the PI3K-Akt Network: Current Status and a Promise for the Treatment of Human Diseases.2008
- Author(s)
  M. Noguchi
- Journal Title
  
  Curr Sig. Thera (In press)
- Description
  「研究成果報告書概要(和文)」より
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] The roles of the PI3K-AKT network in infectious Diseases.2008
- Author(s)
  M. Noguchi
- Journal Title
  
  Jpn J. Infect Dis. (In press)
- Description
  「研究成果報告書概要(和文)」より
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] The roles of the PI3K-AKT network infections Diseases.2008
- Author(s)
  M. Noguchi
- Journal Title
  
  Jpn J. Infect Dis. In Press
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] Regulation of the PI3K-Akt Network: Current Status and a Promise for the Treatment Human Disease.2008
- Author(s)
  M. Noguchi
- Journal Title
  
  Curr Sig. Thera In Press
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] Protooncogene TCL1 : More than just as a co-activator for Akt2007
- Author(s)
  M.Noguchi
- Journal Title
  
  The FASEB Journal 21(In press)
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] Protooncogene TCL1: More than just as a co-activator for Akt.2007
- Author(s)
  M. Noguchi
- Journal Title
  
  FASEB J. 21
  
  Pages: 2273-2284
- Description
  「研究成果報告書概要(和文)」より
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] Fusion of Biotechnology and Biology2007
- Author(s)
  M. Noguchi
- Journal Title
  
  Hokkaido University Press
  
  Pages: 305-315
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Journal Article] Protooncogene TCL1: More than just a co-activator for Akt.2007
- Author(s)
  M. Noguchi
- Journal Title
  
  FASEBJ. 21
  
  Pages: 2273-2284
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Protooncogene TCL1b functions as an AKT kinase co-activator that exhibitsoncogenic potency in vivo2012
- Author(s)
  M. Hashimoto, M. Matsuda F. Suizu,T.Nagamine,N. Hirata,H. Noguchi,S. Tanaka,W. Tokuyama,M. Noguchi
- Organizer
  第71回日本癌学会学術集会
- Place of Presentation
  札幌市・札幌コンベンションセンター
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] ウイルス感染症におけるTCL1BによるAKTシグナル伝達系の活性化機構2012
- Author(s)
  野口　昌幸
- Organizer
  第８６回日本感染症学会
- Place of Presentation
  長崎ブリックホール（長崎県）
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] ウィルス感染症におけるTCL1BによるAKTシグナル伝達系の活性化機構2012
- Author(s)
  野口昌幸
- Organizer
  第86回日本感染症学会
- Place of Presentation
  長崎市・長崎ブリックホール
- Year and Date
  2012-04-25
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Modulation of the Akt signal by its binding proteins.2012
- Author(s)
  野口昌幸、水津太
- Organizer
  第35回分子生物学会
- Place of Presentation
  福岡市・福岡国際会議場
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Protooncogene TCL1b Functions as An Akt Kinase Co-activator that Exhibits On cogenic Potency In Vivo.2012
- Author(s)
  M Hashimoto,F Suizu,W Tokuyama,H Noguchi,M Matsuda,N Hirata,T Nagamine,S Tanaka,M Noguchi
- Organizer
  第35回分子生物学会
- Place of Presentation
  福岡市・福岡国際会議場
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Akt結合因子による細胞反応性の修飾の分子機構2012
- Author(s)
  野口　昌幸
- Organizer
  第３５回日本分子生物学会
- Place of Presentation
  福岡国際会議場（福岡県）
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Characterization of protooncogene TCL 1b as an Akt kinase co-activator.2011
- Author(s)
  M.Noguchi
- Organizer
  CSHL Meeting
- Place of Presentation
  Cold Spring Harbor Laboratory(ｱﾒﾘｶ）
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Characterization of the interaction of influenza virus NS1 with Akt2011
- Author(s)
  M. Matsuda,F. Suizu,N. Hirata,T. Miyazaki,C. Obuse, M.Noguchi
- Organizer
  国際微生物学連合2011会議
- Place of Presentation
  札幌市・札幌コンベンションセンター
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] インフルエンザウイルス感染症における感染宿主細胞のAKT とインフルエンザNS1 蛋白結合の機能的解析2011
- Author(s)
  野口　昌幸
- Organizer
  第８５回日本感染症学会
- Place of Presentation
  ｻﾞ･ﾌﾟﾘﾝｽ　ﾊﾟｰｸﾀﾜｰ東京（東京都）
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] TNF-α drives IL-10 production inmurine dendritic cells2011
- Author(s)
  Hirata,Y.Yanagawa ,K.Iwabuchi ,M.Sato,H.Ogura,K.Onoe ,M.Noguchi
- Organizer
  第40回日本免疫学会学術集会
- Place of Presentation
  千葉市(幕張メッセ)
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] プロトオンコジンTCL1 bのAKT活性化補助因子としての役割について2011
- Author(s)
  M.Hashimoto, F. Suizu,M. Matsuda, N. Hirata, M. Noguchi
- Organizer
  第34回日本分子生物学会年会
- Place of Presentation
  横浜市・パシフィコ横浜
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Characterization of the interaction ofinfluenza virus NS1 with Akt2011
- Author(s)
  M. Matsuda,F. Suizu,N. Hirata,T.Miyazaki,C. Obuse, M.Noguchi
- Organizer
  国際微生物学連合2011会議
- Place of Presentation
  札幌市・札幌コンベンションセンター
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Characterization of protooncogene TCL1b as an Akt kinase co-activator2011
- Author(s)
  M. Hashimoto,F. Suizu,W. Tokuyama,H.Noguchi,M. Matsuda,N. Hirata,T. Nagamine,S. Tanaka, M .Noguchi
- Organizer
  CSHL Meeting
- Place of Presentation
  Cold Spring HarborLaboratory(ニューヨーク・米)
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] インフルエンザウイルス感染症における感染宿主細胞のAKTとインフルエンザNS1蛋白結合の機能的解析2011
- Author(s)
  野口昌幸
- Organizer
  第85回日本感染症学会総会
- Place of Presentation
  東京・ザ・プリンスパークタワー東京
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] プロトオンコジーンTCL1bの生物機能解析2011
- Author(s)
  M.Hashimoto, F.Suizu, M.Matsuda, N.Hirata, M.Noguchi
- Organizer
  第70回日本癌学会学術総会
- Place of Presentation
  名古屋市・名古屋国際会議場
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Characterization of the interaction of Akt with virus NS12011
- Author(s)
  M. Matsuda, Suizu F, Hirata N, Miyazaki T, Obuse C, Noguchi M
- Organizer
  Keystone Symposia
- Place of Presentation
  Keystone Resort(コロラド・米)
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] The role of dendritic cell-produced TNF-α in generation of IL-10-producingCD4+T cells2010
- Author(s)
  Hirata,Y.Yanagawa,K.Iwabuchi,M.Satoh, H.Ogura,T.Seya,K.Onoe,M.Noguchi
- Organizer
  14TH INTERNATIONAL CONGRESS OF IMMUNOLOGY,Kobe,Japan
- Place of Presentation
  神戸市・神戸国際展示場
- Year and Date
  2010-08-24
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] ユビキチン化によるAKTの活性化制御の仕組み2010
- Author(s)
  野口昌幸
- Organizer
  第69回日本癌学会学術総会
- Place of Presentation
  大阪国際会議場(大阪府)
- Year and Date
  2010-09-24
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] ユビキチン化によるAKTの活性化制御の仕組み2010
- Author(s)
  野口昌幸
- Organizer
  第69回日本癌学会学術総会
- Place of Presentation
  大阪市・大阪国際会議場
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] ユビキチン化によるAKT活性の調節メカニズムの解析2010
- Author(s)
  F. Suizu,Y. Hiramuki,F. Okumura,M. Matsuda,A. Okumura,N. Hirata,M. Narita,T. Kohono,J. Yokota,M. Bohgaki,C. Obuse,S. Hatakeyama T. Obata,M Noguchi
- Organizer
  第33回日本分子生物学会
- Place of Presentation
  神戸市・神戸ポートアイランド
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Characterization of the interaction of influenza virus NS1 with Akt2010
- Author(s)
  M. Matsuda, F. Suizu, N. Hirata,T. Miyazaki,C. Obuse,M. Noguchi
- Organizer
  第33回日本分子生物学会
- Place of Presentation
  神戸市・神戸ポートアイランド
- Data Source
  KAKENHI-PROJECT-22370046
[Presentation] Protooncogene TCL1: More than just a co-activator for Akt.2007
- Author(s)
  M. Noguchi
- Organizer
  JAPANESE CANCER ASSOCIATION
- Place of Presentation
  Pacifico Yokohama, Yokohama
- Year and Date
  2007-10-03
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] PI3K-AKT Network as a Therapeutic Target for Human Disease.2007
- Author(s)
  M. Noguchi
- Organizer
  IDDST 2007
- Place of Presentation
  Grand New World Hotel, Xi'an, China
- Year and Date
  2007-11-08
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Identification of NBRE (nerve growth factor responsive element) of the TCL1 promoter as a novel negative regulatory element.2007
- Author(s)
  M. Noguchi
- Organizer
  Keystone Symposia
- Place of Presentation
  サンタフェ(米国)・ヒルトンサンタフェ
- Year and Date
  2007-02-16
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] PI3K-AKT Network as a Therapeutic Target For Human Disease.2007
- Author(s)
  M. Noguchi
- Organizer
  IDDST・2007
- Place of Presentation
  西安(中国)・古都新世界大酒店
- Year and Date
  2007-11-08
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Deregulation of cell death and survival and human diseases2007
- Author(s)
  M. Noguchi
- Organizer
  Japan Society for Adaptation Medicine
- Place of Presentation
  Hokkaido University, Sapporo
- Year and Date
  2007-06-09
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] 細胞死と増殖を介した生体ホメオスターシス制御の破綻と疾病2007
- Author(s)
  野口昌幸
- Organizer
  日本適応医学会
- Place of Presentation
  札幌・北海道大学
- Year and Date
  2007-06-09
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Protooncogene TCL1: More than just a co-activator for Akt.2007
- Author(s)
  野口昌幸
- Organizer
  日本癌学会
- Place of Presentation
  横浜・パシフィコ横浜
- Year and Date
  2007-10-03
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] PI3K-AKT Network as a Therapeutic Target For Human Disease.2007
- Author(s)
  M. Noguchi
- Organizer
  IDDST・2007
- Place of Presentation
  西安(中国)古都新世界大酒店
- Year and Date
  2007-11-08
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Identification of NBRE (nerve growth factor responsive element) of the TCL1 promoter as a novel negative regulatory element2007
- Author(s)
  M. Noguchi
- Organizer
  Keystone Symposia
- Place of Presentation
  Hilton Hotel Santa Fe, Santa Fe, USA
- Year and Date
  2007-02-16
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Protooncogene TCL1: More than just as a co-activator for Akt.2007
- Author(s)
  M. Noguchi
- Organizer
  日本癌学会
- Place of Presentation
  横浜・パシフィコ横浜
- Year and Date
  2007-10-03
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Identification of NBRE (nerve growth factor responsive element) of the TCL1 promoter as a novel negative regulatory element.2006
- Author(s)
  M. Noguchi
- Organizer
  The Molecular Biology Society of Japan
- Place of Presentation
  Fukuoka Dome, Fukuoka
- Year and Date
  2006-10-07
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Identification of NBRE (nerve growth factor responsive element) of the TCL1 promoter as a novel negative regulatory element.2006
- Author(s)
  野口昌幸
- Organizer
  日本免疫学会
- Place of Presentation
  大阪・大阪国際会議場
- Year and Date
  2006-12-12
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] AKTの活性化と癌2006
- Author(s)
  野口昌幸
- Organizer
  日本癌学会
- Place of Presentation
  横浜・パシフィコ横浜
- Year and Date
  2006-09-30
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Identification of NBRE (nerve growth factor responsive element) of the TCL1 promoter as a novel negative regulatory element.2006
- Author(s)
  野口昌幸
- Organizer
  日本分子生物学会
- Place of Presentation
  名古屋・名古屋国際会議場
- Year and Date
  2006-12-07
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Identification of NBRE (nerve growth factor responsive element) of the TCL1 promoter as a novel negative regulatory element.2006
- Author(s)
  M. Noguchi
- Organizer
  Japanese Society of Immunology
- Place of Presentation
  Grand Cube Osaka, Osaka
- Year and Date
  2006-10-12
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] Gene regulation of the protooncogene TCL12005
- Author(s)
  M. Noguchi
- Organizer
  The Molecular Biology Society of Japan
- Place of Presentation
  Fukuoka Dome, Fukuoka
- Year and Date
  2005-10-09
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17370044
[Presentation] AKT活性化補助因子であるプロトオンコジンTCL1の遺伝子発現調節機構2005
- Author(s)
  野口昌幸
- Organizer
  日本分子生物学会
- Place of Presentation
  福岡・福岡ドーム
- Year and Date
  2005-12-09
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17370044

1. SUIDU Futoshi (90431379)

# of Collaborated Projects: 1 results

# of Collaborated Products: 14 results

NOGUCHI Masayuki 野口 昌幸

野口 昌幸 ノグチ マサユキ

Research Projects

Research Products

Co-Researchers

Modulation of Akt kinase activity by ubiquitinationPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

発ガンとAKTキナーゼ活性化の分子機構Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Characterization of the molecular mechanisms of Akt activationPrincipal Investigator

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ATOM機能の解明Principal Investigator

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[Book] Regulation of Akt by phosphorylation of distinct threonine and serine residues Threonine:Structure Biosynthesis and Functions; Advances in Medicine and Biology2011

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[Book] Regulation of Akt by phosphorylation of distinct threonine and serine residues Threonine : Structure, Biosynthesis and Functions ; Advances in Medicine and Biology2011

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[Book] Regulation of the PI3K-Akt Network: Current Status and a Promise for the Treatment of Human Diseases.2008

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[Book] バイオとナノの融合II2007

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[Book] バイオとナノの融合〔新生命科学の融合〕2007

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[Journal Article] The role of tumor necrosis factor-α for interleukin-10 production by murine dendritic cells2011

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NAID

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[Journal Article] Regulation of Akt by phosphorylation of distinct threonine and serine residues Threonine:Structure, Biosynthesis and Functions2011

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[Journal Article] Dendritic cell-derived TNF-αisresponsible for development of IL-10- producing CD4+ T cells2010

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NAID

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[Journal Article] The roles of the PI3K-AKT network in infectious Diseases.2008

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[Journal Article] Regulation of the PI3K-Akt Network: Current Status and a Promise for the Treatment of Human Diseases.2008

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[Journal Article] The roles of the PI3K-AKT network in infectious Diseases.2008

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NOGUCHI Masayuki 野口昌幸

野口昌幸ノグチマサユキ

[Journal Article] Regulation of Akt by phosphorylation of distinct threonine and serine residues　Threonine:Structure, Biosynthesis and Functions2011