Fukushima Arata 福島新

Researcher Number	40706553
Other IDs
Affiliation *help	2017 – 2018 : 北海道大学, 大学病院, 助教 2016 – 2017 : 北海道大学, 大学病院, 医員
Review Section/Research Field	Principal Investigator Pediatrics Except Principal Investigator Basic Section 59040:Nutrition science and health science-related / Basic Section 53020:Cardiology-related
Keywords	Principal Investigator ミトコンドリア機能 / アセチル化修飾 / 心不全 / アセチル化 / 先天性心疾患 / エネルギー代謝 Except Principal Investigator 骨格筋 / 心不全 / 脳由来神経栄養因子 / マイオカイン … More

Study on the mechanism of regulation of mitochondrial function by brain-derived neurotrophic factorOngoing
- Principal Investigator
  
  絹川真太郎
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Review Section
  
  Basic Section 59040:Nutrition science and health science-related
- Research Institution
  Hokkaido University
Role of alpha-defensins derived from the small intestine and gut microbiota in heart failureOngoing
- Principal Investigator
  
  横田卓
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 53020:Cardiology-related
- Research Institution
  Hokkaido University
The role of protein lysine-acetlation in congenital heart diseasePrincipal InvestigatorOngoing
- Principal Investigator
  
  福島新
- Project Period (FY)
  2017 – 2019
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Pediatrics
- Research Institution
  Hokkaido University

All 2017

All Presentation

[Presentation] Cardiac hypertrophy in congenital heart disease delays the maturation of cardiac fatty acid β-oxidation by modifying myocardial acetylation control2017
- Author(s)
  Fukushima A, Kingawa S, Yokota T, Takada S, Furihata T, Matsumoto J, Tsuda M, Nakajima T, Katayama T, Nanbu H, Maekawa S, Kakutani N, Shirakawa R, Okita K, Kantor PF, Rebeyka, IM, Lopaschuk GD
- Organizer
  第81回日本循環器学会学術集会
- Data Source
  KAKENHI-PROJECT-17K10137
[Presentation] Protein acetylation in skeletal muscle impairs mitochondrial fatty acid oxidation and limits exercise capacity in post-infarct heart failure in mice.2017
- Author(s)
  山梨克真，津田正哉，福島　新，高田真吾，降旗高明，松本純一，前川　聡，片山貴史，横田　卓，絹川真太郎
- Organizer
  第21回日本心不全学会学術集会
- Data Source
  KAKENHI-PROJECT-17K10137
[Presentation] Mitochondrial Protein Hyperacetylation in Skeletal Muscle is Associated with Exercise Intolerance in Murine Model of Post-Infarct Heart Failure2017
- Author(s)
  Fukushima A, Tsuda M, Takada S, Yokota T, Saito A, Mizushima W, Furihata T, Matsumoto J, Katayama T, Nakajima T, Maekawa S, Nambu H, Shirakawa R, Kakutani N, Yoshikuni O, Yamanashi K, Okita K, and Kinugawa S
- Organizer
  22nd International Academy of Cardiology Annual Scientific Sessions.
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-17K10137
[Presentation] Acetylation of mitochondrial proteins alters fatty acid β-oxidation in skeletal muscle and limits exercise capacity in post-infarct heart failure in mice.2017
- Author(s)
  Masaya T, Fukushima A, Matsumoto J, Takada S, Yokota T, Furihata T, Katayama T, Nakajima T, Nambu H, Maekawa S, Shirakawa R, Kakutani N, Obata Y, Yamanashi K, Okita K, Kinugawa S
- Organizer
  American Heart Association Scientific Session
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-17K10137

1. 絹川真太郎 (60399871)

# of Collaborated Projects : 3results

# of Collaborated Products : 3results
2. 横田卓 (90374321)

# of Collaborated Projects : 3results

# of Collaborated Products : 4results
3. 高田真吾 (60722329)

# of Collaborated Projects : 2results

# of Collaborated Products : 4results
4. 安斉俊久 (60232089)

# of Collaborated Projects : 1results

# of Collaborated Products : 0results
5. 綾部時芳 (90301019)

# of Collaborated Projects : 1results

# of Collaborated Products : 0results

Fukushima Arata 福島新

Research Projects

Research Products

Co-Researchers

Study on the mechanism of regulation of mitochondrial function by brain-derived neurotrophic factorOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Role of alpha-defensins derived from the small intestine and gut microbiota in heart failureOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The role of protein lysine-acetlation in congenital heart diseasePrincipal InvestigatorOngoing

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Presentation] Cardiac hypertrophy in congenital heart disease delays the maturation of cardiac fatty acid β-oxidation by modifying myocardial acetylation control2017

Author(s)

Organizer

Data Source

[Presentation] Protein acetylation in skeletal muscle impairs mitochondrial fatty acid oxidation and limits exercise capacity in post-infarct heart failure in mice.2017

Author(s)

Organizer

Data Source

[Presentation] Mitochondrial Protein Hyperacetylation in Skeletal Muscle is Associated with Exercise Intolerance in Murine Model of Post-Infarct Heart Failure2017

Author(s)

Organizer

Data Source

[Presentation] Acetylation of mitochondrial proteins alters fatty acid β-oxidation in skeletal muscle and limits exercise capacity in post-infarct heart failure in mice.2017

Author(s)

Organizer

Data Source

Fukushima Arata 福島 新

Research Projects

Research Products

Co-Researchers

Study on the mechanism of regulation of mitochondrial function by brain-derived neurotrophic factorOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Role of alpha-defensins derived from the small intestine and gut microbiota in heart failureOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The role of protein lysine-acetlation in congenital heart diseasePrincipal InvestigatorOngoing

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Presentation] Cardiac hypertrophy in congenital heart disease delays the maturation of cardiac fatty acid β-oxidation by modifying myocardial acetylation control2017

Author(s)

Organizer

Data Source

[Presentation] Protein acetylation in skeletal muscle impairs mitochondrial fatty acid oxidation and limits exercise capacity in post-infarct heart failure in mice.2017

Author(s)

Organizer

Data Source

[Presentation] Mitochondrial Protein Hyperacetylation in Skeletal Muscle is Associated with Exercise Intolerance in Murine Model of Post-Infarct Heart Failure2017

Author(s)

Organizer

Data Source

[Presentation] Acetylation of mitochondrial proteins alters fatty acid β-oxidation in skeletal muscle and limits exercise capacity in post-infarct heart failure in mice.2017

Author(s)

Organizer

Data Source

Fukushima Arata 福島新