NAMIKI TAKESHI 並木剛

Researcher Number	50401352
Affiliation *help	2017 – 2018 : 東京医科歯科大学, 大学院医歯学総合研究科, 准教授 2015 – 2017 : 東京医科歯科大学, 医学部附属病院, 講師 2007 : 医科歯科大, 医学部附属病院, 講師 2006 : 医科歯科大, 医学部附属病院, 助手
Review Section/Research Field	Principal Investigator Dermatology / Basic Section 53050:Dermatology-related
Keywords	Principal Investigator 悪性黒色腫

NUAK2を標的とした悪性黒色腫の分子標的治療開発Principal InvestigatorOngoing
- Principal Investigator
  
  並木剛
- Project Period (FY)
  2018 – 2020
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 53050:Dermatology-related
- Research Institution
  Tokyo Medical and Dental University
Development of a novel molecular targeted therapy against acral melanomasPrincipal Investigator
- Principal Investigator
  
  並木剛
- Project Period (FY)
  2015 – 2017
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Dermatology
- Research Institution
  Tokyo Medical and Dental University
皮膚悪性黒色腫の染色体遺伝子異常解析Principal Investigator
- Principal Investigator
  
  並木剛
- Project Period (FY)
  2006 – 2007
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Dermatology
- Research Institution
  Tokyo Medical and Dental University

All 2017 2016 2015

All Presentation

[Presentation] NUAK2 is over-expressed and DNA copy number is increased in acral melanoma: its significance on the survival of patients2017
- Author(s)
  Nojima K, Hayashi M, Funazumi M, Ishikawa M, Kaneko Y, Kawaguchi M, Suzuki T, Tanemura A, Katayama I, Mori T, Yamazaki N, Yokozeki H, Hearing VJ, Namiki T
- Organizer
  47th Annual Meeting of the European Society for Dermatologic Research, SALZBURG 2017
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] NUAK2 is over-expressed and DNA copy number is increased in acral melanoma: its significance on the survival of patients2017
- Author(s)
  Nojima K, Hayashi M, Funazumi M, Ishikawa M, Kaneko Y, Kawaguchi M, Suzuki T, Tanemura A, Katayama I, Mori T, Yamazaki N, Yokozeki H, Hearing VJ, Namiki T
- Organizer
  42nd Annual Meeting of the Japanese Society for Investigative Dermatology, KOCHI 2017
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] 末端黒子型悪性黒色腫発症の分子機構解明と分子標的治療開発2017
- Author(s)
  並木　剛
- Organizer
  13回加齢皮膚医学研究会
- Invited
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] 末端黒子型悪性黒色腫に対する分子標的治療の開発2016
- Author(s)
  並木　剛
- Organizer
  第115回日本皮膚科学会総会
- Place of Presentation
  国立京都国際会館（京都府・京都市）
- Year and Date
  2016-06-03
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] 末端黒子型悪性黒色腫に対する分子標的治療の開発2016
- Author(s)
  並木　剛
- Organizer
  第56回湯島皮膚科談話会
- Place of Presentation
  ステーションカンファレンス万世橋（東京都・千代田区）
- Invited
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] The significance of over-expression both of NUAK2 and of phosphor-Akt(S473) on patient’s survival of acral melanomas2015
- Author(s)
  Namiki Takeshi
- Organizer
  The 40th Annual Meeting of the Japanese Society for Investigative Dermatology
- Place of Presentation
  岡山、岡山
- Year and Date
  2015-12-11
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] NUAK2増幅とPTEN欠失はCDK活性化を介してメラノーマ発症を促進する2015
- Author(s)
  並木　剛
- Organizer
  26回日本色素細胞学会
- Place of Presentation
  北海道、札幌
- Year and Date
  2015-11-14
- Invited
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] NUAK2増幅とPTEN欠失はCDK活性化を介してメラノーマ発症を促進する2015
- Author(s)
  並木　剛
- Organizer
  74回日本癌学会総会
- Place of Presentation
  愛媛、名古屋
- Year and Date
  2015-10-08
- Data Source
  KAKENHI-PROJECT-15K09760
[Presentation] NUAK2 amplification coupled with PTEN deficiency promote melanoma development via CDK activation2015
- Author(s)
  Namiki Takeshi
- Organizer
  45th Annual Meeting of the European Society for Dermatologic Research
- Place of Presentation
  オランダ、ロッテルダム
- Year and Date
  2015-09-09
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-15K09760

1. 河上裕 (50161287)

# of Collaborated Projects : 2results

# of Collaborated Products : 0results
2. 西村栄美 (70396331)

# of Collaborated Projects : 1results

# of Collaborated Products : 0results

NAMIKI TAKESHI 並木 剛

Research Projects

Research Products

Co-Researchers

NUAK2を標的とした悪性黒色腫の分子標的治療開発Principal InvestigatorOngoing

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Development of a novel molecular targeted therapy against acral melanomasPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

皮膚悪性黒色腫の染色体遺伝子異常解析Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Presentation] NUAK2 is over-expressed and DNA copy number is increased in acral melanoma: its significance on the survival of patients2017

Author(s)

Organizer

Data Source

[Presentation] NUAK2 is over-expressed and DNA copy number is increased in acral melanoma: its significance on the survival of patients2017

Author(s)

Organizer

Data Source

[Presentation] 末端黒子型悪性黒色腫発症の分子機構解明と分子標的治療開発2017

Author(s)

Organizer

Data Source

[Presentation] 末端黒子型悪性黒色腫に対する分子標的治療の開発2016

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] 末端黒子型悪性黒色腫に対する分子標的治療の開発2016

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] The significance of over-expression both of NUAK2 and of phosphor-Akt(S473) on patient’s survival of acral melanomas2015

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] NUAK2増幅とPTEN欠失はCDK活性化を介してメラノーマ発症を促進する2015

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] NUAK2増幅とPTEN欠失はCDK活性化を介してメラノーマ発症を促進する2015

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] NUAK2 amplification coupled with PTEN deficiency promote melanoma development via CDK activation2015

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

NAMIKI TAKESHI 並木剛