kawaguchi satoshi 川口哲

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KAWAGUCHI SATOSHI 川口哲

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Researcher Number	60814217
Other IDs
Affiliation (Current)	2025: 旭川医科大学, 医学部, 助教
Affiliation (based on the past Project Information) *help	2018 – 2024: 旭川医科大学, 医学部, 助教
Review Section/Research Field	Principal Investigator Basic Section 55060:Emergency medicine-related Except Principal Investigator Basic Section 55060:Emergency medicine-related
Keywords	Principal Investigator 敗血症性心筋症 / 敗血症 / 一酸化窒素 / 心不全 / 心筋代謝 / NO / iNOS / β3アドレナリン受容体 / β3アドレナリン受容体 / NO産生 … More / β３受容体 / 誘導型一酸化窒素 / 敗血症生心筋症 / βアドレナリン受容体 / 心機能障害 / リポポリサッカロイド / 脂肪酸代謝 / ミトコンドリア機能 / 心筋エネルギー代謝 / 敗血症性心機能障害 … More Except Principal Investigator 心筋代謝 / β３受容体 / 敗血症 / Fingolimod / S1P / CLP / スフィンゴシン1リン酸 / メタボリックシフト / 敗血症性心筋症 / LPS / NO産生 / ミトコンドリア / エンドトキシン / 心不全 Less

Development of a novel therapeutic strategy for sepsis induced cardiomyopathy through the mechanism of nitric oxide regulationPrincipal Investigator
- Principal Investigator
  
  川口哲
- Project Period (FY)
  2024 – 2026
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 55060:Emergency medicine-related
- Research Institution
  Asahikawa Medical College
The role of beta-3 receptors and sphingosine 1-phosphate in the regulation of myocardial metabolism in sepsis
- Principal Investigator
  
  Okada Motoi
- Project Period (FY)
  2021 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 55060:Emergency medicine-related
- Research Institution
  Asahikawa Medical College
Clarification of the mechanism for beta-3 adrenergic receptor regulation of Inducible nitric oxide synthase in septic cardiomyopathyPrincipal Investigator
- Principal Investigator
  
  Kawaguchi Satoshi
- Project Period (FY)
  2020 – 2023
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 55060:Emergency medicine-related
- Research Institution
  Asahikawa Medical College
Elucidation of myocardial energy metabolism via beta-3 receptor in sepsis
- Principal Investigator
  
  Okada Motoi
- Project Period (FY)
  2018 – 2021
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 55060:Emergency medicine-related
- Research Institution
  Asahikawa Medical College
The Beta-3 adrenergic receptor has a possibility to improve the mortality with sepsis-induced cardiac dysfunction by regulating cardiac metabolism.Principal Investigator
- Principal Investigator
  
  KAWAGUCHI SATOSHI
- Project Period (FY)
  2018 – 2019
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 55060:Emergency medicine-related
- Research Institution
  Asahikawa Medical College

All 2024 2023 2021 2020 2019 2018

All Journal Article Presentation

[Journal Article] Current Perspectives of Mitochondria in Sepsis-Induced Cardiomyopathy2024
- Author(s)
  Kuroshima Tatsuki、Kawaguchi Satoshi、Okada Motoi
- Journal Title
  
  International Journal of Molecular Sciences
  
  Volume: 25 Issue: 9 Pages: 4710-4710
- DOI
  10.3390/ijms25094710
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-21K09009, KAKENHI-PROJECT-20K17852
[Journal Article] Cardiac Metabolism in Sepsis2021
- Author(s)
  Kawaguchi Satoshi、Okada Motoi
- Journal Title
  
  Metabolites
  
  Volume: 11 Issue: 12 Pages: 846-846
- DOI
  10.3390/metabo11120846
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-18K08879, KAKENHI-PROJECT-20K17852, KAKENHI-PROJECT-21K09009
[Journal Article] β(3)-Adrenergic receptor blockade reduces mortality in endotoxin-induced heart failure by suppressing induced nitric oxide synthase and saving cardiac metabolism.2020
- Author(s)
  1)Kawaguchi S, Okada M, Ijiri E, Koga D, Watanabe T, Hayashi K, Kashiwagi Y, Fujita S, Hasebe N.
- Journal Title
  
  Am J Physiol Heart Circ Physiol
  
  Volume: 318 Issue: 2 Pages: H283-H294
- DOI
  10.1152/ajpheart.00108.2019
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K08093, KAKENHI-PROJECT-18K08879, KAKENHI-PROJECT-19K07261, KAKENHI-PROJECT-16K08458, KAKENHI-PROJECT-18K16505, KAKENHI-PROJECT-20K17852
[Presentation] 敗血症生心筋症の病態整理2024
- Author(s)
  川口　哲
- Organizer
  第88回日本循環器学会総会　教育セッション
- Invited
- Data Source
  KAKENHI-PROJECT-20K17852
[Presentation] 敗血症性心筋症のメカニズム2024
- Author(s)
  川口　哲
- Organizer
  日本循環器学会
- Invited
- Data Source
  KAKENHI-PROJECT-21K09009
[Presentation] 敗血症性心筋症の治療戦略2023
- Author(s)
  岡田基，黒嶋　健起、川口　哲
- Organizer
  第51回日本救急医学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-20K17852
[Presentation] 敗血症での心筋代謝障害のメカニズムと治療戦略2023
- Author(s)
  岡田　基、川口　哲、黒嶋　健起
- Organizer
  第51回日本集中治療医学会学術集会
- Data Source
  KAKENHI-PROJECT-20K17852
[Presentation] Cardiac Metabolism was Improved by Regulation of Nitric Oxide Synthase Through The Beta-3 Adrenergic Receptor in Endotoxin-induced Failing Heart2019
- Author(s)
  Satoshi Kawaguchi、Motoi Okada、Satoshi Fujita、Naoyuki Hasebe
- Organizer
  American Society Association 2019
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16505
[Presentation] Endotoxin Induced Cardiac Metabolic Shift is Controlled Through the Regulation of Fatty Acid Oxidation by a Beta-3 Adrenergic Receptor2018
- Author(s)
  Satoshi Kawaguchi、Motoi Okada、Satoshi Fujita、Naoyuki Hasebe
- Organizer
  AHA2018
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16505
[Presentation] The Blockade of Beta-3 Adrenergic Receptor Improves Prognosis of the Sepsis-Related Heart Failure by Stabilization of the Heart Metabolism2018
- Author(s)
  Satoshi Kawaguchi、Motoi Okada、Satoshi Fujita、Naoyuki Hasebe
- Organizer
  第82回日本循環器学会
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16505
[Presentation] Blockade of beta-3 adrenergic receptor ameliorates septic cardiomyopathy through the improvement of cardiac fatty acid oxidation2018
- Author(s)
  Satoshi Kawaguchi、Motoi Okada、Naoyuki Hasebe、Satoshi Fujita
- Organizer
  31st European Society of Intensive Care Medicine
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16505
[Presentation] Beta-3 adrenergic receptor blockade reduces mortality with septic cardiomyopathy by saving cardiac energy metabolism and mitochondrial function2018
- Author(s)
  Satoshi Kawaguchi、Motoi Okada、Satoshi Fujita、Naoyuki Hasebe
- Organizer
  5th INTERNATIONAL SYMPOSIUM NEW FRONTIERS in CARDIOVASCULAR RESEARCH
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16505
[Presentation] Myocardial Metabolic Regulation by the Beta-3 Adrenergic Receptor in Sepsis2018
- Author(s)
  Satoshi Kawaguchi、Motoi Okada、Satoshi Fujita、Naoyuki Hasebe
- Organizer
  ESC congress 2018
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K16505

1. Okada Motoi (80431427)

# of Collaborated Projects: 3 results

# of Collaborated Products: 2 results
2. 藤田智 (10173428)

# of Collaborated Projects: 2 results

# of Collaborated Products: 0 results
3. 黒嶋健起 (30898408)

# of Collaborated Projects: 2 results

# of Collaborated Products: 0 results
4. 井尻えり子 (40646072)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
5. 長谷部直幸

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

kawaguchi satoshi 川口 哲

KAWAGUCHI SATOSHI 川口 哲

Research Projects

Research Products

Co-Researchers

Development of a novel therapeutic strategy for sepsis induced cardiomyopathy through the mechanism of nitric oxide regulationPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The role of beta-3 receptors and sphingosine 1-phosphate in the regulation of myocardial metabolism in sepsis

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Clarification of the mechanism for beta-3 adrenergic receptor regulation of Inducible nitric oxide synthase in septic cardiomyopathyPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Elucidation of myocardial energy metabolism via beta-3 receptor in sepsis

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The Beta-3 adrenergic receptor has a possibility to improve the mortality with sepsis-induced cardiac dysfunction by regulating cardiac metabolism.Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] Current Perspectives of Mitochondria in Sepsis-Induced Cardiomyopathy2024

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Cardiac Metabolism in Sepsis2021

Author(s)

Journal Title

DOI

Data Source

[Journal Article] β(3)-Adrenergic receptor blockade reduces mortality in endotoxin-induced heart failure by suppressing induced nitric oxide synthase and saving cardiac metabolism.2020

Author(s)

Journal Title

DOI

Data Source

[Presentation] 敗血症生心筋症の病態整理2024

Author(s)

Organizer

Data Source

[Presentation] 敗血症性心筋症のメカニズム2024

Author(s)

Organizer

Data Source

[Presentation] 敗血症性心筋症の治療戦略2023

Author(s)

Organizer

Data Source

[Presentation] 敗血症での心筋代謝障害のメカニズムと治療戦略2023

Author(s)

Organizer

Data Source

[Presentation] Cardiac Metabolism was Improved by Regulation of Nitric Oxide Synthase Through The Beta-3 Adrenergic Receptor in Endotoxin-induced Failing Heart2019

Author(s)

Organizer

Data Source

[Presentation] Endotoxin Induced Cardiac Metabolic Shift is Controlled Through the Regulation of Fatty Acid Oxidation by a Beta-3 Adrenergic Receptor2018

Author(s)

Organizer

Data Source

[Presentation] The Blockade of Beta-3 Adrenergic Receptor Improves Prognosis of the Sepsis-Related Heart Failure by Stabilization of the Heart Metabolism2018

Author(s)

Organizer

Data Source

[Presentation] Blockade of beta-3 adrenergic receptor ameliorates septic cardiomyopathy through the improvement of cardiac fatty acid oxidation2018

Author(s)

kawaguchi satoshi 川口哲

KAWAGUCHI SATOSHI 川口哲