Kaneshiro Kenta 金城健太

Researcher Number	60897586
Other IDs	https://orcid.org/0000-0003-2161-7168
Affiliation (Current)	2026: 神戸大学, 医学系研究科(医), 特命助教
Affiliation (based on the past Project Information) *help	2024 – 2025: 神戸大学, 医学研究科, 特命助教 2021 – 2023: 神戸大学, 保健学研究科, 保健学研究員
Review Section/Research Field	Principal Investigator Basic Section 54020:Connective tissue disease and allergy-related / Basic Section 53020:Cardiology-related
Keywords	Principal Investigator 時計遺伝子 / 概日リズム / 炎症性メディエーター / 滑膜細胞 / Bmal1 / 炎症性サイトカイン / 関節リウマチ / 心不全 / NF-κB / 関節リウマチ滑膜細胞 / オートクライン

Development of Novel Diagnostic and Therapeutic Approaches Targeting Clock Genes for Heart FailurePrincipal Investigator
- Principal Investigator
  
  金城健太
- Project Period (FY)
  2025 – 2027
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 53020:Cardiology-related
- Research Institution
  Kobe University
Impact of the Clock gene Bmal1 on NF-kB Transcriptional Activity in RA-FLSPrincipal Investigator
- Principal Investigator
  
  Kaneshiro Kenta
- Project Period (FY)
  2023 – 2024
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 54020:Connective tissue disease and allergy-related
- Research Institution
  Kobe University
The procuction of inflammatory mediators anc autocrine via clock genes in RA-FLSPrincipal Investigator
- Principal Investigator
  
  Kaneshiro Kenta
- Project Period (FY)
  2021 – 2022
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 54020:Connective tissue disease and allergy-related
- Research Institution
  Kobe University

All 2025 2024 2023 2022 2021

All Journal Article Presentation

[Journal Article] ACLY Promotes Cardiac Fibrosis via the Regulation of DNL and Histone Acetylation2025
- Author(s)
  Kuwahara Naoya、Nagao Manabu、Shinohara Masakazu、Kaneshiro Kenta、Emoto Takuo、Yoshida Takeshi、Fukuda Terunobu、Nishimori Makoto、Satomi-Kobayashi Seimi、Otake Hiromasa、Hirata Ken-Ichi、Ishida Tatsuro、Toh Ryuji
- Journal Title
  
  Hypertension
  
  Volume: Online ahead of print. Issue: 6 Pages: 1116-1128
- DOI
  10.1161/hypertensionaha.124.24088
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-23K15347, KAKENHI-PROJECT-24K11290
[Journal Article] The clock gene Bmal1 controls inflammatory mediators in rheumatoid arthritis fibroblast-like synoviocytes2024
- Author(s)
  Kenta Kaneshiro, Kanako Nakagawa, Hikari Tsukamoto, Genta Matsuoka, Seitaro Okuno, Koji Tateishi, Yasuhiro Terashima, Nao Shibanuma, Kohsuke Yoshida, Akira Hashiramoto
- Journal Title
  
  Biochemical and Biophysical Research Communications
  
  Volume: 8 Pages: 149315-149323
- DOI
  10.1016/j.bbrc.2023.149315
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-23K15347, KAKENHI-PROJECT-22K08563
[Presentation] IL-6 interferes with apoptosis-induction of Rheumatoid Arthritis fibroblast-like synovial cells by affecting Bcl-2 family of proteins.2024
- Author(s)
  Yuri Yokomi, Hikari Tsukamoto, Kenta Kaneshiro, Kohsuke Yoshida, Koji Tateishi, Yasuhiro Terashima, Nao Shibanuma, Yoshitada Sakai, Akira Hashiramoto
- Organizer
  EULAR 2024
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] Clock Gene Bmal1 Promotes Transcriptional Activity of NF-κB to Regulate Production of Inflammatory Mediators in RA-FLS.2024
- Author(s)
  Hikari Tsukamoto, Kenta Kaneshiro, Kohsuke Yoshida, Koji Tateishi, Yasuhiro Terashima, Nao Shibanuma, Yoshitada, Sakai, Akira Hashiramoto
- Organizer
  American College of Rheumatology
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] RA滑膜細胞におけるTNFα誘導性TEF遺伝子発現低下のメカニズム:3’UTR結合miRNAを対象とした研究2024
- Author(s)
  吉田幸祐、金城健太、塚本光里、川崎善子、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第67回日本リウマチ学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] 時計制御遺伝子Tef は細胞周期調節因子を介して細胞増殖活性を制御する2024
- Author(s)
  奥野征太郎、塚本光里、吉田幸祐、金城健太、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第68回日本リウマチ学会学術集会
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] 時計遺伝子Tefは細胞増殖活性とTNFα産生を制御する2024
- Author(s)
  塚本光里、吉田幸祐、金城健太、川崎善子、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第67回日本リウマチ学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] 時計遺伝子Bmal1はRA-FLSの炎症性メディエーター発現を制御する2024
- Author(s)
  金城健太、吉田幸祐、塚本光里、川崎善子、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第67回日本リウマチ学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] Clock Gene Bmal1 Contributes to Inflammation via Phosphorylation of NF-κB/p65 in RA-FLS.2024
- Author(s)
  Hikari Tsukamoto, Kenta Kaneshiro, Kohsuke Yoshida, Koji Tateishi, Yasuhiro Terashima, Nao Shibanuma, Yoshitada Sakai, Akira Hashiramoto
- Organizer
  ACR convergence2024
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] 時計遺伝子Bmal1はNF-κB転写活性を促進してRA-FLSにおける炎症性メディエーターの産生を制御する2024
- Author(s)
  塚本光里、金城健太、奥野征太郎、吉田幸祐、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第68回日本リウマチ学会学術集会
- Data Source
  KAKENHI-PROJECT-23K15347
[Presentation] 時計遺伝子Bmal1はRA-FLSの炎症性メディエーター発現を制御する2023
- Author(s)
  金城健太、吉田幸祐、塚本光里、川崎善子、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第67回日本リウマチ学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-21K16288
[Presentation] 関節リウマチ滑膜細胞における時計遺伝子を介した炎症性メディエーター産生2022
- Author(s)
  中川加奈子，金城健太，吉田幸祐，川崎善子，立石耕司，寺島康浩，柴沼均，酒井良忠，柱本照
- Organizer
  第30回日本リウマチ学会近畿支部学術集会
- Data Source
  KAKENHI-PROJECT-21K16288
[Presentation] 時計遺伝子Bmal1はRA滑膜細胞の炎症性メディエーター産生を制御する2022
- Author(s)
  中川加奈子、金城健太、吉田幸祐、川崎善子、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第66回日本リウマチ学会総会・学術集会
- Data Source
  KAKENHI-PROJECT-21K16288
[Presentation] 時計遺伝子Bmal1はRA滑膜細胞の炎症性メディエーター産生を制御する2022
- Author(s)
  中川加奈子、金城健太、吉田幸祐、川崎善子、立石耕司、寺島康浩、柴沼均、酒井良忠、柱本照
- Organizer
  第66回日本リウマチ学会学術総会
- Data Source
  KAKENHI-PROJECT-21K16288
[Presentation] 関節リウマチ滑膜細胞における時計遺伝子を介した炎症性メディエーター産生2021
- Author(s)
  中川加奈子，金城健太，吉田幸祐，川崎善子，立石耕司，寺島康浩，柴沼均，酒井良忠，柱本照
- Organizer
  第30回日本リウマチ学会近畿支部学術集会
- Data Source
  KAKENHI-PROJECT-21K16288

1. 長尾学 (70866029)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
2. 篠原正和 (80437483)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results

Kaneshiro Kenta 金城 健太

Research Projects

Research Products

Co-Researchers

Development of Novel Diagnostic and Therapeutic Approaches Targeting Clock Genes for Heart FailurePrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Impact of the Clock gene Bmal1 on NF-kB Transcriptional Activity in RA-FLSPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

The procuction of inflammatory mediators anc autocrine via clock genes in RA-FLSPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

[Journal Article] ACLY Promotes Cardiac Fibrosis via the Regulation of DNL and Histone Acetylation2025

Author(s)

Journal Title

DOI

Data Source

[Journal Article] The clock gene Bmal1 controls inflammatory mediators in rheumatoid arthritis fibroblast-like synoviocytes2024

Author(s)

Journal Title

DOI

Data Source

[Presentation] IL-6 interferes with apoptosis-induction of Rheumatoid Arthritis fibroblast-like synovial cells by affecting Bcl-2 family of proteins.2024

Author(s)

Organizer

Data Source

[Presentation] Clock Gene Bmal1 Promotes Transcriptional Activity of NF-κB to Regulate Production of Inflammatory Mediators in RA-FLS.2024

Author(s)

Organizer

Data Source

[Presentation] RA滑膜細胞におけるTNFα誘導性TEF遺伝子発現低下のメカニズム:3’UTR結合miRNAを対象とした研究2024

Author(s)

Organizer

Data Source

[Presentation] 時計制御遺伝子Tef は細胞周期調節因子を介して細胞増殖活性を制御する2024

Author(s)

Organizer

Data Source

[Presentation] 時計遺伝子Tefは細胞増殖活性とTNFα産生を制御する2024

Author(s)

Organizer

Data Source

[Presentation] 時計遺伝子Bmal1はRA-FLSの炎症性メディエーター発現を制御する2024

Author(s)

Organizer

Data Source

[Presentation] Clock Gene Bmal1 Contributes to Inflammation via Phosphorylation of NF-κB/p65 in RA-FLS.2024

Author(s)

Organizer

Data Source

[Presentation] 時計遺伝子Bmal1はNF-κB転写活性を促進してRA-FLSにおける炎症性メディエーターの産生を制御する2024

Author(s)

Organizer

Data Source

[Presentation] 時計遺伝子Bmal1はRA-FLSの炎症性メディエーター発現を制御する2023

Author(s)

Organizer

Data Source

[Presentation] 関節リウマチ滑膜細胞における時計遺伝子を介した炎症性メディエーター産生2022

Author(s)

Organizer

Data Source

[Presentation] 時計遺伝子Bmal1はRA滑膜細胞の炎症性メディエーター産生を制御する2022

Author(s)

Organizer

Data Source

[Presentation] 時計遺伝子Bmal1はRA滑膜細胞の炎症性メディエーター産生を制御する2022

Author(s)

Organizer

Data Source

Kaneshiro Kenta 金城健太