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OKUMA ATSUSHI  大熊 敦史

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Okuma Atsushi  大熊 敦史

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Researcher Number 70726059
Other IDs
Affiliation (Current) 2026: 株式会社日立製作所(研究開発グループ), 基礎研, 研究員
Affiliation (based on the past Project Information) *help 2017: 大阪大学, 微生物病研究所, 特任助教(常勤)
2016: 公益財団法人がん研究会, がん研究所 がん生物部, 研究員
Review Section/Research Field
Principal Investigator
Pathological medical chemistry
Keywords
Principal Investigator
骨髄由来免疫抑制細胞 / CDKI / 細胞老化 / ケモタキシス / CDKインヒビター / MDSC
  • Research Projects

    (1 results)
  • Research Products

    (2 results)
  •  Analysis of functions of p16Ink4a and p21Cip1/Waf1 in myeloid-derived suppressor cellsPrincipal Investigator

    • Principal Investigator
      Okuma Atsushi
    • Project Period (FY)
      2016 – 2017
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      Osaka University
      Japanese Foundation for Cancer Research

All 2017 2016

All Journal Article Presentation

  • [Journal Article] p16Ink4a and p21Cip1/Waf1 promote tumour growth by enhancing myeloid-derived suppressor cells chemotaxis2017

    • Author(s)
      Okuma Atsushi、Hanyu Aki、Watanabe Sugiko、Hara Eiji
    • Journal Title

      Nature Communications

      Volume: 8 Issue: 1 Pages: 2050-2050

    • DOI

      10.1038/s41467-017-02281-x

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-16K19064
  • [Presentation] The role of p16Ink4a and p21Cip1/Waf1 in myeloid-derived suppressor cells2016

    • Author(s)
      Atsushi Okuma
    • Organizer
      第45回日本免疫学会学術集会
    • Place of Presentation
      沖縄
    • Year and Date
      2016-12-05
    • Data Source
      KAKENHI-PROJECT-16K19064

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