FUKUSHIMA Keiko 福島慶子

Researcher Number	80380872
External Links
Affiliation (based on the past Project Information) *help	2005 – 2008: 長崎大学, 医学部・歯学部附属病院, 医員
Review Section/Research Field	Except Principal Investigator Metabolomics
Keywords	Except Principal Investigator 膵島炎 / poly I:C / 自然免疫 / 制御性T細胞 / インスリン / 1型糖尿病 / インスリン自己抗体 / 粘膜免疫 / インスリンペプチド / 発症予防 / 1 型糖尿病

Antigen specific treatment for the inhibition and remission of type 1 diabetes with insulin B chain peptide
- Principal Investigator
  
  ABIRU Norio
- Project Period (FY)
  2005 – 2008
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Metabolomics
- Research Institution
  Nagasaki University

All 2008 2007 2006 2005

All Journal Article Presentation

[Journal Article] Combined insulin B:9-23 self-peptide and polyinosinic-polycytidylic acid accelerate insulitis but inhibit development of diabetes by ncreasing the proportion of CD4+Foxp3+ regulatory T cells in the islets in non-obese diabetic mice2008
- Author(s)
  Fukushima K, Abiru N, Nagayama Y, Kobayashi M, Satoh T.(他7名, 2番目)
- Journal Title
  
  Biochem Biophys Res Commun 367(4)
  
  Pages: 719-724
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17590940
[Journal Article] Combined insulin B:9-23 self-peptide and polyinosinic-polycytidylic acid accelerate insulitis but inhibit development of diabetes by increasing the proportion of CD4+Foxp3+ regulatory T cells in the islets in non-obese diabetic mice.2008
- Author(s)
  K Fukushima
- Journal Title
  
  Biochem Biophys Res Commun 367
  
  Pages: 719-724
- NAID
  120006983528
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17590940
[Journal Article] Altered B : 9-23 insulin, when administered intranasally with cholera toxin adjuvant, suppresses the expression of insulin autoantibodies and prevents diabetes2007
- Author(s)
  Kobayashi M, Abiru N, Arakawa T, Fukushima K, (他8名, 2 番目)
- Journal Title
  
  J Immunol 179(4)
  
  Pages: 2082-2088
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] Combined insulin B : 9-23 self-peptide and polyinosinic-polycytidylic acid accelerate insulitis but inhibit development of diabetes by increasing the proportion of CD4+Foxp3+regulatory T cells in the islets in nonobese diabetic mice2007
- Author(s)
  福島慶子
- Organizer
  9^<th> International Congress of the Immunology of Diabetes Society
- Place of Presentation
  Miami, USA
- Year and Date
  2007-11-17
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] インスリンB : 9-23 ペプチド+poly I : C 併用投与による、CD4+CD25+Foxp3制御性T細胞の膵島内誘導と糖尿病の発症抑制2007
- Author(s)
  福島慶子
- Organizer
  第50 回日本糖尿病学会年次学術集会
- Place of Presentation
  仙台
- Year and Date
  2007-05-25
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] NOD膵島局所におけるCD4+CD25+Foxp3+制御性T細胞による膵島炎から糖尿病への進展抑制2006
- Author(s)
  福島慶子
- Organizer
  第49回日本糖尿病学会年次学術集会
- Place of Presentation
  東京
- Year and Date
  2006-05-26
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] Poly 1 : C and insulin B : 9-23 self-peptide accelerate insulitis containing pathogenic and CD4^+Foxp3^+ regulatory cells which can inhibit the development of diabetes2006
- Author(s)
  福島慶子
- Organizer
  Federation of Clinical Immunology Societies (FOCIS) 6^<th> Annual Meeting
- Place of Presentation
  San Francisco, USA
- Year and Date
  2006-06-03
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] 1 型糖尿病自然発症およびインスリンB鎖ペプチドによる1型糖尿病発症抑制機序へのCD4+CD25+制御性T細胞の関与について2005
- Author(s)
  福島慶子
- Organizer
  第48 回日本糖尿病学会年次学術集会
- Place of Presentation
  神戸
- Year and Date
  2005-05-24
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] Elimination of CTL epitope from insulin peptide B : 9-23 provides nasal-induced suppression of insulitis and diabetes in the NOD mouse.2005
- Author(s)
  福島慶子
- Organizer
  8^<th> International Congress of the Immunology of Diabetes Society
- Place of Presentation
  淡路
- Year and Date
  2005-10-08
- Data Source
  KAKENHI-PROJECT-17590940
[Presentation] Early Administration of Anti-CD25 Antibody Accelerates The Spontaneous Development of Diabetes And Abrogates The Disease Inhibition by Insulin B:9-23 Peptide Vaccination2005
- Author(s)
  福島慶子
- Organizer
  66^<th>American Diabetes Association Scientific Sessions (ADA)
- Place of Presentation
  San Diego, USA
- Year and Date
  2005-06-13
- Data Source
  KAKENHI-PROJECT-17590940

1. ABIRU Norio (00380981)

# of Collaborated Projects: 1 results

# of Collaborated Products: 2 results
2. KOBAYASHI Masakazu (00380874)

# of Collaborated Projects: 1 results

# of Collaborated Products: 2 results
3. SATOH Tuyoshi (70437896)

# of Collaborated Projects: 1 results

# of Collaborated Products: 1 results

FUKUSHIMA Keiko 福島 慶子

Research Projects

Research Products

Co-Researchers

Antigen specific treatment for the inhibition and remission of type 1 diabetes with insulin B chain peptide

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] Combined insulin B:9-23 self-peptide and polyinosinic-polycytidylic acid accelerate insulitis but inhibit development of diabetes by ncreasing the proportion of CD4+Foxp3+ regulatory T cells in the islets in non-obese diabetic mice2008

Author(s)

Journal Title

Data Source

[Journal Article] Combined insulin B:9-23 self-peptide and polyinosinic-polycytidylic acid accelerate insulitis but inhibit development of diabetes by increasing the proportion of CD4+Foxp3+ regulatory T cells in the islets in non-obese diabetic mice.2008

Author(s)

Journal Title

NAID

Data Source

[Journal Article] Altered B : 9-23 insulin, when administered intranasally with cholera toxin adjuvant, suppresses the expression of insulin autoantibodies and prevents diabetes2007

Author(s)

Journal Title

Data Source

[Presentation] Combined insulin B : 9-23 self-peptide and polyinosinic-polycytidylic acid accelerate insulitis but inhibit development of diabetes by increasing the proportion of CD4+Foxp3+regulatory T cells in the islets in nonobese diabetic mice2007

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] インスリンB : 9-23 ペプチド+poly I : C 併用投与による、CD4+CD25+Foxp3制御性T細胞の膵島内誘導と糖尿病の発症抑制2007

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] NOD膵島局所におけるCD4+CD25+Foxp3+制御性T細胞による膵島炎から糖尿病への進展抑制2006

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] Poly 1 : C and insulin B : 9-23 self-peptide accelerate insulitis containing pathogenic and CD4^+Foxp3^+ regulatory cells which can inhibit the development of diabetes2006

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] 1 型糖尿病自然発症およびインスリンB鎖ペプチドによる1型糖尿病発症抑制機序へのCD4+CD25+制御性T細胞の関与について2005

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] Elimination of CTL epitope from insulin peptide B : 9-23 provides nasal-induced suppression of insulitis and diabetes in the NOD mouse.2005

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] Early Administration of Anti-CD25 Antibody Accelerates The Spontaneous Development of Diabetes And Abrogates The Disease Inhibition by Insulin B:9-23 Peptide Vaccination2005

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

FUKUSHIMA Keiko 福島慶子