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Nishinaka-Arai Yoko  西中 瑶子

ORCIDConnect your ORCID iD *help
Researcher Number 80789644
Other IDs
Affiliation (Current) 2025: 京都大学, 医学研究科, 助教
Affiliation (based on the past Project Information) *help 2019 – 2024: 京都大学, 医学研究科, 助教
Review Section/Research Field
Principal Investigator
Basic Section 52050:Embryonic medicine and pediatrics-related
Keywords
Principal Investigator
白血病 / ダウン症候群 / 多段階発がん / 発がん / 一過性骨髄異常増殖症 / GATA1sタンパク / 転写因子
  • Research Projects

    (3 results)
  • Research Products

    (5 results)
  •  一過性骨髄異常増殖症モデルを用いたがんのクローン進化のメカニズム解明Principal Investigator

    • Principal Investigator
      西中 瑶子
    • Project Period (FY)
      2024 – 2026
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 52050:Embryonic medicine and pediatrics-related
    • Research Institution
      Kyoto University
  •  Functional analysis of GATA1-short-form protein in the pathogenesis of transient abnormal myeloproliferative disordersPrincipal Investigator

    • Principal Investigator
      Nishinaka-Arai Yoko
    • Project Period (FY)
      2022 – 2023
    • Research Category
      Grant-in-Aid for Early-Career Scientists
    • Review Section
      Basic Section 52050:Embryonic medicine and pediatrics-related
    • Research Institution
      Kyoto University
  •  Exploring the mechanism of mutation acquisition to induce tumorigenesis using Down syndrome associated leukemia modelPrincipal Investigator

    • Principal Investigator
      Nishinaka-Arai Yoko
    • Project Period (FY)
      2019 – 2021
    • Research Category
      Grant-in-Aid for Early-Career Scientists
    • Review Section
      Basic Section 52050:Embryonic medicine and pediatrics-related
    • Research Institution
      Kyoto University

All 2023 2021 2019

All Journal Article Presentation

  • [Journal Article] Down syndrome-related transient abnormal myelopoiesis is attributed to a specific erythro-megakaryocytic subpopulation with GATA1 mutation.2021

    • Author(s)
      Nishinaka-Arai Y, Niwa A, Matsuo S, Kazuki Y, Yakura Y, Hiroma T, Toki T, Sakuma T, Yamamoto T, Ito E, Oshimura M, Nakahata T, Saito MK.
    • Journal Title

      Haematologica

      Volume: 106(2) Issue: 2 Pages: 635-640

    • DOI

      10.3324/haematol.2019.242693

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-19K17358, KAKENHI-PROJECT-18H04039, KAKENHI-PROJECT-19K16111
  • [Journal Article] Pluripotent stem cell model of early hematopoiesis in Down syndrome reveals quantitative effects of short-form GATA1 protein on lineage specification.2021

    • Author(s)
      Matsuo S, Nishinaka-Arai Y, Kazuki Y, Oshimura M, Nakahata T, Niwa A, Saito MK.
    • Journal Title

      PLoS One

      Volume: 16 (3) Issue: 3 Pages: e0247595-e0247595

    • DOI

      10.1371/journal.pone.0247595

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-19K17358
  • [Presentation] Elucidation of the unique function of GATA1s in the development of transient abnormal myeloproliferative disorders2023

    • Author(s)
      Yoko Nishinaka-Arai, Yohko Kitagawa, Yasuhiro Kazuki, Yasuyuki Arai, Tatsutoshi Nakahata, Akira Niwa and Megumu K. Saito
    • Organizer
      65 th ASH annual meeting and exposition
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-22K15897
  • [Presentation] hPSC model reveals impact of GATA1 mutation on a newly defined subpopulation leading to TAM2019

    • Author(s)
      Yoko Nishinaka-Arai, Akira Niwa, Shiori Matsuo, Yasuhiro Kazuki, Yuwna Yakura, Takehiro Hiroma, Tsutomu Toki, Tetsushi Sakuma, Takashi Yamamoto, Etsuro Ito, Mitsuo Oshimura, Tatsutoshi Nakahata, and Megumu K. Saito
    • Organizer
      第81回日本血液学会学術集会
    • Data Source
      KAKENHI-PROJECT-19K17358
  • [Presentation] Down syndrome-related transient abnormal myelopoiesis is derived from a newly identified hematopoietic subpopulation2019

    • Author(s)
      Yoko Nishinaka-Arai, Akira Niwa, Shiori Matsuo, Yasuhiro Kazuki, Yuwna Yakura, Takehiro Hiroma, Tsutomu Toki, Tetsushi Sakuma, Takashi Yamamoto, Etsuro Ito, Mitsuo Oshimura, Tatsutoshi Nakahata, and Megumu K. Saito
    • Organizer
      ISSCR 2019 annual meeting
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-19K17358

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