IMAI Toshio 今井敏夫

Researcher Number	90120617
External Links
Affiliation (based on the past Project Information) *help	2014 – 2017: 日本歯科大学, 生命歯学部, 教授 2009 – 2013: The Nippon Dental University, 生命歯学部, 准教授 2006: The Nippon Dental University, School of Life Dentistry at Tokyo, Assoicate Professor, 生命歯学部, 助教授 2005: 日本歯科大学, 歯学部, 助教授
Review Section/Research Field	Principal Investigator Social dentistry Except Principal Investigator Social dentistry
Keywords	Principal Investigator 硫化水素 / 破骨細胞 / 口臭物質 / RANKL / 骨芽細胞 / シグナル伝達 / 口臭 / 分化 / 予防歯科学 / PKC … More Except Principal Investigator 癌関連遺伝子 / 硫化水素 / 歯肉癌細胞 / アポトーシス / PHLDA1 / アポトーシス感受性 / 癌細胞 / 歯肉上皮細胞 Less

Physiological role of mechanical stress in alveolar bone resorption induced by the oral malodorous compounds.Principal Investigator
- Principal Investigator
  
  IMAI TOSHIO
- Project Period (FY)
  2015 – 2017
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Social dentistry
- Research Institution
  The Nippon Dental University
Elucidation of the alveolar bone absorption mechanism by the oral malodorous compounds .Principal Investigator
- Principal Investigator
  
  IMAI TOSHIO
- Project Period (FY)
  2012 – 2014
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Social dentistry
- Research Institution
  The Nippon Dental University
Influence of oral malodor compounds for expression of cancer-related gene in oral epidermal cells.
- Principal Investigator
  
  MURATA Takatoshi
- Project Period (FY)
  2010 – 2012
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Social dentistry
- Research Institution
  Tsurumi University
  The Nippon Dental University
Effect of oral malodorous compound on the osteoclast differentiation and alveolar bone loss.Principal Investigator
- Principal Investigator
  
  IMAI Toshio
- Project Period (FY)
  2009 – 2011
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Social dentistry
- Research Institution
  The Nippon Dental University
The effect of volatile sulfur compounds on the metabolism of alveolar bone.Principal Investigator
- Principal Investigator
  
  IMAI Toshio
- Project Period (FY)
  2005 – 2006
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Social dentistry
- Research Institution
  The Nippon Dental University

All 2014 2012 2011 2010 2006 Other

All Journal Article Presentation

[Journal Article] Oral Malodorous Compound Induces Osteoclast Differentiation Without RANKL.2010
- Author(s)
  Ii H, Imai T, Yaegaki K, Irie K, Ekuni D, Morita M.
- Journal Title
  
  J Periodont
  
  Volume: 81 Pages: 1691-1697
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-21592667
[Journal Article] Hydrogen sulfide inhibited cell proliferation of osteoblasts via the MAPK pathway.2006
- Author(s)
  T.IMAI, T.Kamoda, T.Murata, K.Yaegaki
- Journal Title
  
  Journal of Dental Health 56
  
  Pages: 559-559
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17592191
[Journal Article] 口臭物質(硫化水素)はMAPKシグナルを介してヒト骨芽細胞の増殖を抑制する2006
- Author(s)
  今井敏夫, 鴨田剛司, 村田貴俊, 八重垣健
- Journal Title
  
  口腔衛生会誌 56
  
  Pages: 559-559
- NAID
  10018258798
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17592191
[Presentation] 揮発性硫黄化合物による骨髄間葉系幹細胞の破骨細胞への分化誘導2014
- Author(s)
  今井敏夫、伊井久貴、岡田実緒、那須優則、八重垣健
- Organizer
  第６３回日本口腔衛生学会
- Place of Presentation
  熊本、熊本市国際交流会館
- Data Source
  KAKENHI-PROJECT-24593171
[Presentation] PKC mediate osteoclast differentiation induced by hydrogen sulfide2014
- Author(s)
  T.Imai, H.Ii, K.Yaegaki
- Organizer
  AADR annual meeting
- Place of Presentation
  Charlotte, N.C, USA
- Data Source
  KAKENHI-PROJECT-24593171
[Presentation] 歯肉上皮癌細胞株 Ca9-22 のアポトーシス誘導における PHLDA1 の役割2012
- Author(s)
  村田貴俊、角田衣理加、今井奬、花田信弘
- Organizer
  第54回歯科基礎医学会
- Place of Presentation
  奥羽大学記念講堂(福島県)
- Year and Date
  2012-09-16
- Data Source
  KAKENHI-PROJECT-22592349
[Presentation] Ca9-22 細胞への硫化水素暴露により発現が上昇する PHLDA12011
- Author(s)
  村田貴俊、角田衣理加、野村義明、今井奬、花田信弘
- Place of Presentation
  長良川国際会議場(岐阜県)
- Year and Date
  2011-10-01
- Data Source
  KAKENHI-PROJECT-22592349
[Presentation] Oral Malodorous Compound Induces Osteoclast Differentiation Without RANKL.2010
- Author(s)
  Ii H, Imai T, Calenic B, Yaegaki K, Irie K, Ekuni D, Morita G
- Organizer
  IADR
- Place of Presentation
  スペインバルセロナ
- Year and Date
  2010-07-16
- Data Source
  KAKENHI-PROJECT-21592667
[Presentation] 乳歯歯髄幹細胞の膵臓分化における硫化水素の影響
- Author(s)
  岡田実緒, Nikolay Ishkitiev，八重垣健, 今井敏夫
- Organizer
  日本口腔衛生学会
- Place of Presentation
  熊本市国際交流会館（熊本県、熊本市）
- Year and Date
  2014-05-29 – 2014-05-31
- Data Source
  KAKENHI-PROJECT-24593171
[Presentation] 硫化水素は骨髄幹細胞から破骨細胞を分化誘導する
- Author(s)
  今井敏夫，那須優則，八重垣　健
- Organizer
  日本歯科基礎医学会
- Place of Presentation
  福岡国際会議場（福岡県、福岡市）
- Year and Date
  2014-09-25 – 2014-09-27
- Data Source
  KAKENHI-PROJECT-24593171
[Presentation] 揮発性硫黄化合物による骨髄間葉系幹細胞の破骨細胞への分化誘導
- Author(s)
  今井敏夫，伊井久貴，岡田実緒，那須優則，八重垣健
- Organizer
  日本口腔衛生学会
- Place of Presentation
  熊本市国際交流会館（熊本県、熊本市）
- Year and Date
  2014-05-29 – 2014-05-31
- Data Source
  KAKENHI-PROJECT-24593171

1. YAEGAKI Ken (40166468)

# of Collaborated Projects: 4 results

# of Collaborated Products: 7 results
2. MURATA Takatoshi (10313529)

# of Collaborated Projects: 2 results

# of Collaborated Products: 2 results
3. SATO Tsutomu (60130671)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
4. KAMODA Takeshi (00366767)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
5. II HISATAKA (00746604)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
6. 那須優則 (50130688)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results

IMAI Toshio 今井 敏夫

Research Projects

Research Products

Co-Researchers

Physiological role of mechanical stress in alveolar bone resorption induced by the oral malodorous compounds.Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Elucidation of the alveolar bone absorption mechanism by the oral malodorous compounds .Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Influence of oral malodor compounds for expression of cancer-related gene in oral epidermal cells.

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Effect of oral malodorous compound on the osteoclast differentiation and alveolar bone loss.Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

The effect of volatile sulfur compounds on the metabolism of alveolar bone.Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] Oral Malodorous Compound Induces Osteoclast Differentiation Without RANKL.2010

Author(s)

Journal Title

Data Source

[Journal Article] Hydrogen sulfide inhibited cell proliferation of osteoblasts via the MAPK pathway.2006

Author(s)

Journal Title

Description

Data Source

[Journal Article] 口臭物質(硫化水素)はMAPKシグナルを介してヒト骨芽細胞の増殖を抑制する2006

Author(s)

Journal Title

NAID

Description

Data Source

[Presentation] 揮発性硫黄化合物による骨髄間葉系幹細胞の破骨細胞への分化誘導2014

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] PKC mediate osteoclast differentiation induced by hydrogen sulfide2014

Author(s)

Organizer

Place of Presentation

Data Source

[Presentation] 歯肉上皮癌細胞株 Ca9-22 のアポトー シス誘導における PHLDA1 の役割2012

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] Ca9-22 細胞への硫化水素暴露により発現が上昇する PHLDA12011

Author(s)

Place of Presentation

Year and Date

Data Source

[Presentation] Oral Malodorous Compound Induces Osteoclast Differentiation Without RANKL.2010

Author(s)

Organizer

Place of Presentation

Year and Date

Data Source

[Presentation] 乳歯歯髄幹細胞の膵臓分化における硫化水素の影響

Author(s)

Organizer

Place of Presentation

IMAI Toshio 今井敏夫

[Presentation] 歯肉上皮癌細胞株 Ca9-22 のアポトーシス誘導における PHLDA1 の役割2012