UEDA MASASHI 上田昌史

… Alternative Names

Ueda Masashi 上田昌史

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Researcher Number	90791541
Other IDs
Affiliation (Current)	2025: 愛知医科大学, 医学部, 助教
Affiliation (based on the past Project Information) *help	2022 – 2023: 愛知医科大学, 医学部, 助教 2021 – 2023: 愛知県医療療育総合センター発達障害研究所, 細胞病態研究部, リサーチレジデント 2020 – 2021: 国立研究開発法人国立精神・神経医療研究センター, 神経研究所疾病研究第二部, リサーチフェロー 2020: 愛知県医療療育総合センター発達障害研究所, 細胞病態研究部, 特別共同研究員 2019: 愛知県医療療育総合センター発達障害研究所, 細胞病態研究部, リサーチレジデント 2017 – 2018: 愛知県心身障害者コロニー発達障害研究所, 発生障害学部, リサーチレジデント
Review Section/Research Field	Principal Investigator Basic Section 52050:Embryonic medicine and pediatrics-related / Pediatrics Except Principal Investigator Experimental pathology
Keywords	Principal Investigator 知的障害 / ZTTK症候群 / シナプス形成不全 / RNA解析 / ゲノムDNA解析 / 疾患モデル / ストレス応答 / 免疫細胞 / 発生・分化 / 樹状突起スパイン形成の阻害 … More / 神経細胞移動の阻害 / シナプス形成 / 樹状突起スパイン形成 / 神経細胞移動 / SONハプロ不全 … More Except Principal Investigator lncRNA / microRNA / 逆鎖NLGN4X / AKT / メチル化 / CpG / Sp1 / CEBP / エピジェネティクス / AVP / MeCP2 / CEBPδ / iPSC / CpGメチル化 / エピジェネティックス / 自閉症 / NLGN4X Less

ZTTK症候群の知的障害発症機構解明を目指した核内蛋白質SONの機能解析Principal Investigator
- Principal Investigator
  
  上田昌史
- Project Period (FY)
  2020 – 2024
- Research Category
  
  Grant-in-Aid for Early-Career Scientists
- Review Section
  
  Basic Section 52050:Embryonic medicine and pediatrics-related
- Research Institution
  Aichi Medical University
  National Center of Neurology and Psychiatry
Analysis of Molecular Pathogenesis Caused by Altered Expression of Autism-Susceptibility Gene, NLGN4X.
- Principal Investigator
  
  IIO Akio
- Project Period (FY)
  2017 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Experimental pathology
- Research Institution
  Institute for Developmental Research Aichi Developmental Disability Center
The elucidation of the cellular pathological mechanism of ZTTK syndrome, an intellectual disability, which is caused by SON haploinsufficiencyPrincipal Investigator
- Principal Investigator
  
  Ueda Masashi
- Project Period (FY)
  2017 – 2019
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Pediatrics
- Research Institution
  Institute for Developmental Research Aichi Developmental Disability Center

All 2023 2021 2020 2019 2018 2017

All Journal Article Presentation

[Journal Article] The distribution of neuroligin4, an autism-related postsynaptic molecule, in the human brain2023
- Author(s)
  Toya Akie, Fukada Masahide, Aoki Eiko , Matsuki Tohru, Ueda Masashi, Eda Shima, Hashizume Yoshio , Iio Akio, Masaki Shigeo, Nakayama Atsuo
- Journal Title
  
  Molecular Brain
  
  Volume: 16 Issue: 1 Pages: 20-20
- DOI
  10.1186/s13041-023-00999-y
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17K08778, KAKENHI-PROJECT-20K08224, KAKENHI-PROJECT-21K06433
[Journal Article] STK25 and MST3 Have Overlapping Roles to Regulate Rho GTPases during Cortical Development2021
- Author(s)
  Matsuki Tohru、Iio Akio、Ueda Masashi、Tsuneura Yumi、Howell Brian W.、Nakayama Atsuo
- Journal Title
  
  The Journal of Neuroscience
  
  Volume: 41 Issue: 43 Pages: 8887-8903
- DOI
  10.1523/jneurosci.0523-21.2021
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-18K06543, KAKENHI-PROJECT-17K08778, KAKENHI-PROJECT-20K08224
[Journal Article] Knockdown of Son, a Mouse Homologue of the ZTTK Syndrome Gene, Causes Neuronal Migration Defects and Dendritic Spine Abnormalities2020
- Author(s)
  Masashi Ueda, Tohru Matsuki, Masahide Fukada, Shima Eda, Akie Toya, Akio Iio, Hidenori Tabata, Atsuo Nakayama
- Journal Title
  
  Molecular Brain
  
  Volume: 13 Issue: 1 Pages: 80-80
- DOI
  10.1186/s13041-020-00622-4
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-17K08778, KAKENHI-PROJECT-18K06543, KAKENHI-PROJECT-20K08224, KAKENHI-PROJECT-20K16876
[Presentation] Stk25とMST3による大脳皮質形成制御機構2019
- Author(s)
  松木亨、飯尾明生、上田昌史、戸谷明恵、中山敦雄
- Organizer
  NEURO2019 第42回日本神経科学大会
- Data Source
  KAKENHI-PROJECT-17K08778
[Presentation] SON haploinsufficiency, a cause of human intellectual disabilities, affects the neuronal migrations and dendritic spine formations in the developing mouse brain2019
- Author(s)
  上田　昌史
- Organizer
  第49回北米神経科学学会（シカゴ）
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-17K16292
[Presentation] SON haploinsufficiencuy, a cause of human intellectual disabilities, results in dysregulated neuronal migration in developing mouse brain2018
- Author(s)
  上田昌史
- Organizer
  第４１回日本分子生物学会年会
- Data Source
  KAKENHI-PROJECT-17K16292
[Presentation] Stk25 and MST3 act on neuronal polarization and migration in a compensation manner.2018
- Author(s)
  松木亨、飯尾明生、上田昌史、戸谷明恵、中山敦雄
- Organizer
  日本神経科学会
- Data Source
  KAKENHI-PROJECT-17K08778
[Presentation] Stk25 and MST3 act on neuronal polarization, migration, and cardiovascular development2017
- Author(s)
  松木亨、飯尾明生、上田昌史、戸谷明恵、中山敦雄
- Organizer
  平成29年度「先端モデル動物支援」成果発表会
- Data Source
  KAKENHI-PROJECT-17K08778

1. IIO Akio (80344349)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
2. 松木亨 (90332329)

# of Collaborated Projects: 1 results

# of Collaborated Products: 6 results
3. 飯尾明夫

# of Collaborated Projects: 0 results

# of Collaborated Products: 5 results
4. 中山敦雄

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

UEDA MASASHI 上田 昌史

Ueda Masashi 上田 昌史

Research Projects

Research Products

Co-Researchers

ZTTK症候群の知的障害発症機構解明を目指した核内蛋白質SONの機能解析Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Analysis of Molecular Pathogenesis Caused by Altered Expression of Autism-Susceptibility Gene, NLGN4X.

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

The elucidation of the cellular pathological mechanism of ZTTK syndrome, an intellectual disability, which is caused by SON haploinsufficiencyPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] The distribution of neuroligin4, an autism-related postsynaptic molecule, in the human brain2023

Author(s)

Journal Title

DOI

Data Source

[Journal Article] STK25 and MST3 Have Overlapping Roles to Regulate Rho GTPases during Cortical Development2021

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Knockdown of Son, a Mouse Homologue of the ZTTK Syndrome Gene, Causes Neuronal Migration Defects and Dendritic Spine Abnormalities2020

Author(s)

Journal Title

DOI

Data Source

[Presentation] Stk25とMST3による大脳皮質形成制御機構2019

Author(s)

Organizer

Data Source

[Presentation] SON haploinsufficiency, a cause of human intellectual disabilities, affects the neuronal migrations and dendritic spine formations in the developing mouse brain2019

Author(s)

Organizer

Data Source

[Presentation] SON haploinsufficiencuy, a cause of human intellectual disabilities, results in dysregulated neuronal migration in developing mouse brain2018

Author(s)

Organizer

Data Source

[Presentation] Stk25 and MST3 act on neuronal polarization and migration in a compensation manner.2018

Author(s)

Organizer

Data Source

[Presentation] Stk25 and MST3 act on neuronal polarization, migration, and cardiovascular development2017

Author(s)

Organizer

Data Source

UEDA MASASHI 上田昌史

Ueda Masashi 上田昌史