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Sanjo Hideki  山条 秀樹

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山条 秀樹  サンジョウ ヒデキ

SANJO Hideki  山条 秀樹

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Researcher Number 50391967
Other IDs
Affiliation (Current) 2022: 信州大学, 学術研究院医学系, 准教授
Affiliation (based on the past Project Information) *help 2021: 信州大学, 学術研究院医学系, 准教授
2016 – 2018: 信州大学, 学術研究院医学系, 准教授
2014: 信州大学, 学術研究院医学系, 助教
2012: 信州大学, 医学部, 助教
2011: 信州大学, 医学系研究科, 助教
Review Section/Research Field
Principal Investigator
Experimental pathology / Immunology / Basic Section 49010:Pathological biochemistry-related
Except Principal Investigator
Immunology
Keywords
Principal Investigator
シグナル伝達 / TAK1 / マクロファージ / 好塩基球伝達 / サイトカイン / シグナル / 好塩基球 / 遺伝子欠損マウス / 炎症 / 細胞死 … More / パイロプトーシス / TLR / アポトーシス / Caspase-8 / マクロファージ特異的TAK1欠損マウス / 組織常在マクロファージ / 無菌性炎症 / 免疫学 / TNFシグナル / 炎症性細胞死 / 細胞の生存・分化 … More
Except Principal Investigator
サイトカイン / 好塩基球 / シグナル伝達 / アレルギー / 免疫学 / Th2 / サイトカインレセプター / 細胞内シグナル伝達 / 粘膜免疫 / リンパ球 / 遺伝子発現 / 自然免疫 / 造血 / 自然リンパ球 / 粘膜免英 / T細胞 / 胸腺 / 腸管免疫 / 転写因子 Less
  • Research Projects

    (6 results)
  • Research Products

    (18 results)
  • Co-Researchers

    (4 People)
  •  TNFシグナル関連分子群に着目したマクロファージの運命決定制御機構の解明Principal Investigator

    • Principal Investigator
      山条 秀樹
    • Project Period (FY)
      2021 – 2023
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 49010:Pathological biochemistry-related
    • Research Institution
      Shinshu University
  •  Research about the mechanism of macrophage development and differentiation focusing on TNF signal-associated molecules.Principal Investigator

    • Principal Investigator
      Sanjo Hideki
    • Project Period (FY)
      2016 – 2018
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Immunology
    • Research Institution
      Shinshu University
  •  Regulation of development and function of unconventional T cells and innate lymphoid cells.

    • Principal Investigator
      Taki Shinsuke
    • Project Period (FY)
      2015 – 2017
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Immunology
    • Research Institution
      Shinshu University
  •  Positive and negative regulation of cytokine signals in basophils

    • Principal Investigator
      TAKI Shinsuke
    • Project Period (FY)
      2012 – 2014
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Immunology
    • Research Institution
      Shinshu University
  •  Regulatory mechanism of signal transduction involved in cytokine production from basophils and the control of type 2 immune response.Principal Investigator

    • Principal Investigator
      SANJO Hideki
    • Project Period (FY)
      2011 – 2012
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Experimental pathology
    • Research Institution
      Shinshu University
  •  Intracellularsignals regulating cytokine production in basophils

    • Principal Investigator
      TAKI Shinsuke
    • Project Period (FY)
      2009 – 2012
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      Immunology
    • Research Institution
      Shinshu University

All 2018 2017 2016 2015 2013 2012 2011 Other

All Journal Article Presentation

  • [Journal Article] Conditional Deletion of TAK1 in T Cells Reveals a Pivotal Role of TCRαβ+ Intraepithelial Lymphocytes in Preventing Lymphopenia-Associated Colitis.2015

    • Author(s)
      H. Sanjo, S. Tokumaru, S. Akira, S. Taki
    • Journal Title

      PloS ONE

      Volume: 10

    • DOI

      10.1371/journal.pone.0128761

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-15K08529
  • [Journal Article] Essential Roles of K63-Linked Polyubiquitin-Binding Proteins TAB2 and TAB3 in B Cell Activation via MAPKs2013

    • Author(s)
      Ori D
    • Journal Title

      The Journal of Immunology

      Volume: 190(8) Pages: 4037-4045

    • DOI

      10.4049/jimmunol.1300173

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20002008, KAKENHI-PROJECT-22689016, KAKENHI-PROJECT-23790438, KAKENHI-PROJECT-25860355
  • [Journal Article] Differential requirements for IFN regulatory factor-2 in generation of CD1d-independent T cells bearing NK cell receptors.2012

    • Author(s)
      Notake T, Horisawa S, Sanjo H, Miyagawa S, Hida S, Taki S.
    • Journal Title

      J.Immnol.

      Volume: 188 Pages: 4838-4845

    • DOI

      10.4049/jimmunol.1200210

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-21390149, KAKENHI-PUBLICLY-22021018, KAKENHI-PROJECT-22659096, KAKENHI-PROJECT-23790438, KAKENHI-PROJECT-24591419, KAKENHI-PUBLICLY-24117709
  • [Journal Article] Differential requirements for IRF-2 in generation of CD1d-independent T cells bearing NK cell receptors.2012

    • Author(s)
      Notake T, Horisawa S, Sanjo H, Miyagawa S, Hida S, Taki S.
    • Journal Title

      Journal of Immunology

      Volume: 188 Pages: 4838-4845

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] TAK1 inhibits TLR-driven macrophage cell death by blocking a TRIF-dependent pathway2018

    • Author(s)
      山条秀樹
    • Organizer
      日本分子生物学会年会
    • Data Source
      KAKENHI-PROJECT-16K08828
  • [Presentation] Loss of TAK1 leads to TLR-driven macrophage cell death and inflammation that occur by a TNF-independent mechanism2017

    • Author(s)
      山条秀樹
    • Organizer
      日本免疫学会総会
    • Data Source
      KAKENHI-PROJECT-16K08828
  • [Presentation] Innate immune cells sense Staphylococcus aureus-derived cycteine protease via their surface molecule(s) to produce proinflammatory cytokines.2017

    • Author(s)
      Arisa Morikawa, Saotomo Itoh, Hideki Sanjo, Shinsuke Taki, Shigeaki Hida
    • Organizer
      第46回日本免疫学会学術集会
    • Data Source
      KAKENHI-PROJECT-15K08529
  • [Presentation] Loss of TAK1 leads to TLR-driven macrophage cell death and inflammation that occur by a TNF-independent mechanism2017

    • Author(s)
      山条秀樹
    • Organizer
      International Cytokine and Interferon Society (ICIS2017)
    • Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-16K08828
  • [Presentation] IFN regulatory factor 2 controls ILC development by promoting PLZF+ ILC progenitor generation.2016

    • Author(s)
      Yohei Okubo, Shinsuke Taki, Hideki Sanjo
    • Organizer
      第45回日本免疫学会学術集会
    • Place of Presentation
      沖縄、沖縄コンベンションセンター、那覇市
    • Year and Date
      2016-12-05
    • Data Source
      KAKENHI-PROJECT-15K08529
  • [Presentation] The transcription factor IRF-2 controls CR8αα+ intraepithelial lymphocyte development at the level of thymic precursor.2015

    • Author(s)
      Shigeo Tokumaru, Yohei Okubo, Tahiro Shin, Hideki Sanjo, Shinsuke Taki
    • Organizer
      第44回日本免疫学会学術集会
    • Place of Presentation
      札幌コンベンションセンター
    • Year and Date
      2015-11-18
    • Data Source
      KAKENHI-PROJECT-15K08529
  • [Presentation] Interferon regulatory factor 2 is differentiation factor of Type 3 NKp46+ and NKp46-CD4- innate lymphoid cell2015

    • Author(s)
      Yohei Okubo, Shigeo Tokumaru, Tahiro Shin, Hideki Sanjo, Shinsuke Taki
    • Organizer
      第44回日本免疫学会学術集会
    • Place of Presentation
      札幌コンベンションセンター
    • Year and Date
      2015-11-18
    • Data Source
      KAKENHI-PROJECT-15K08529
  • [Presentation] Essential roles of Tab2 and Tab3 in the activation of MAPK but not NF-kB, in B cells.2012

    • Author(s)
      Ori D., Kato H., Sanjo H., Akira S., Takeuchi O.
    • Organizer
      日本免疫学会
    • Place of Presentation
      神戸
    • Year and Date
      2012-12-05
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] Paxillin wasrequired for IL-3-induced IL-4 production in basophils.2012

    • Author(s)
      Hasegawa A., Hida S., Sanjo H., Oda A., Taki S., Taniguchi S.
    • Organizer
      日本免疫学会
    • Place of Presentation
      神戸
    • Year and Date
      2012-12-06
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] IL-15-independent generation of memory-like 'innate'CD8+ T cells in IRF-2 deficient mice.2011

    • Author(s)
      Notake T., Oda A., Sanjo H., Hida S., Taki S
    • Organizer
      日本免疫学会
    • Place of Presentation
      千葉
    • Year and Date
      2011-11-28
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] IL-3-induced IL-13 production by basophils but not mast cells was sensitive to Paired immunoglobulin-like receptor(PIR)-B mediated inhibitory signals.2011

    • Author(s)
      Oda A., Sakamoto Y., Sanjo H., Notake T., Takai T., Hida S., Taki S.
    • Organizer
      日本免疫学会
    • Place of Presentation
      千葉
    • Year and Date
      2011-11-29
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] Essential roles of Tab2 and Tab3 in the activation of MAPK, but not NF-kB, in B cells.

    • Author(s)
      Ori D, Kato H, Sanjo H, Akira S, Takeuchi O.
    • Organizer
      日本免疫学会
    • Place of Presentation
      神戸
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] Paxillin was required for IL-3-induced IL-4 production in basophils.

    • Author(s)
      Hasegawa A, Hida S, Sanjo H, Oda A, Taki S, Taniguchi S.
    • Organizer
      日本免疫学会
    • Place of Presentation
      神戸
    • Data Source
      KAKENHI-PROJECT-23790438
  • [Presentation] Disease control of spontaneous colitis in LTAC mice, a unique inflammatory disease model.

    • Author(s)
      SANJO HIdeki、TOKUMARU Shigeo、AKIRA Shizuo、TAKI Shinsuke
    • Organizer
      第43回日本免疫学会学術集会
    • Place of Presentation
      京都・国際会議場
    • Year and Date
      2014-12-10 – 2014-12-12
    • Data Source
      KAKENHI-PROJECT-24590578
  • 1.  TAKI Shinsuke (50262027)
    # of Collaborated Projects: 3 results
    # of Collaborated Products: 7 results
  • 2.  HIDA Shigeaki (10345762)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 1 results
  • 3.  竹内 理
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results
  • 4.  三野 享史
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results

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