Kagoya Yuki 籠谷勇紀

… Alternative Names

KAGOYA YUKI 籠谷勇紀

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Researcher Number	70706960
Other IDs
Affiliation (Current)	2022: 愛知県がんセンター(研究所), 腫瘍免疫応答研究分野, 分野長
Affiliation (based on the past Project Information) *help	2019 – 2022: 愛知県がんセンター(研究所), 腫瘍免疫応答研究分野, 分野長 2013 – 2014: 東京大学, 医学部附属病院, 助教
Review Section/Research Field	Principal Investigator Medium-sized Section 50:Oncology and related fields / Hematology / Basic Section 50020:Tumor diagnostics and therapeutics-related Except Principal Investigator Basic Section 50020:Tumor diagnostics and therapeutics-related
Keywords	Principal Investigator 養子免疫療法 / キメラ抗原受容体 / T細胞疲弊 / メモリーT細胞 / 骨髄増殖性腫瘍 / 急性骨髄性白血病 / JAK2V617F遺伝子変異 / リポカリン-2 / DNA損傷 / p53 … More / 活性酸素 / JAK2V617F変異 / ｐ５３ / JAK2V617F / 鉄過剰 / サイトカイン / 免疫チェックポイント分子 / 長期生存能 / PD-1 / JAK-STAT経路 / 抗腫瘍T細胞 / 遺伝子変異 / T細胞性リンパ腫 / エピジェネティクス / 転写因子 / 抗体薬物複合体 / 薬剤送達 … More Except Principal Investigator 肺癌 / 腫瘍抗原 / T細胞 / 術後再発予防 / がんワクチン Less

The determination of T-cell fate by different tumor antigens and prevention of post-operative recurrence of lung cancer
- Principal Investigator
  
  松下博和
- Project Period (FY)
  2022 – 2024
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Review Section
  
  Basic Section 50020:Tumor diagnostics and therapeutics-related
- Research Institution
  Aichi Cancer Center Research Institute
Development of a combinatorial approach of antibody therapeutics and adoptive immunotherapy for cancerPrincipal Investigator
- Principal Investigator
  
  籠谷勇紀
- Project Period (FY)
  2021 – 2022
- Research Category
  
  Grant-in-Aid for Challenging Research (Exploratory)
- Review Section
  
  Medium-sized Section 50:Oncology and related fields
- Research Institution
  Aichi Cancer Center Research Institute
Generation of long-surviving antitumor T cells by genetic modification for optimal adoptive immunotherapyPrincipal Investigator
- Principal Investigator
  
  籠谷勇紀
- Project Period (FY)
  2020 – 2022
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Review Section
  
  Basic Section 50020:Tumor diagnostics and therapeutics-related
- Research Institution
  Aichi Cancer Center Research Institute
Development of a chimeric synthetic receptor to suppress T cell exhaustion for use in adoptive cancer immunotherapyPrincipal Investigator
- Principal Investigator
  
  Kagoya Yuki
- Project Period (FY)
  2019 – 2020
- Research Category
  
  Grant-in-Aid for Challenging Research (Exploratory)
- Review Section
  
  Medium-sized Section 50:Oncology and related fields
- Research Institution
  Aichi Cancer Center Research Institute
Elucidation of the mechanism of transformation of myeloproliferative neoplasm into acute myelogenous leukemia for novel therapeutic strategy for the disease.Principal Investigator
- Principal Investigator
  
  KAGOYA YUKI
- Project Period (FY)
  2013 – 2014
- Research Category
  
  Grant-in-Aid for Research Activity Start-up
- Research Field
  
  Hematology
- Research Institution
  The University of Tokyo

All 2021 2020 2019 2014 2013

All Journal Article Presentation

[Journal Article] Genetic Ablation of HLA Class I, Class II, and the T-cell Receptor Enables Allogeneic T Cells to Be Used for Adoptive T-cell Therapy2020
- Author(s)
  Kagoya Yuki、Guo Tingxi、Yeung Brian、Saso Kayoko、Anczurowski Mark、Wang Chung-Hsi、Murata Kenji、Sugata Kenji、Saijo Hiroshi、Matsunaga Yukiko、Ohashi Yota、Butler Marcus O.、Hirano Naoto
- Journal Title
  
  Cancer Immunology Research
  
  Volume: 8 Pages: 926-936
- DOI
  10.1158/2326-6066.cir-18-0508
- Peer Reviewed / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20H03543, KAKENHI-PROJECT-19K22552
[Journal Article] JAK2V617F-positive myeloproliferative neoplasm clones evoke paracrine DNA damage to adjacent normal cells through secretion of lipocalin-22014
- Author(s)
  Kagoya Y, Yoshimi A, Tsuruta-Kishino T, Arai S, Satoh T, Akira S, Kurokawa M.
- Journal Title
  
  Blood
  
  Volume: 124 Pages: 2996-3006
- DOI
  10.1182/blood-2014-04-570572
- Peer Reviewed / Acknowledgement Compliant / Open Access
- Data Source
  KAKENHI-PROJECT-24249055, KAKENHI-PROJECT-25893047, KAKENHI-PROJECT-26860474
[Presentation] 抗腫瘍T細胞の機能評価を行う上でのポイント2021
- Author(s)
  籠谷勇紀
- Organizer
  日本再生医療学会
- Invited
- Data Source
  KAKENHI-PROJECT-20H03543
[Presentation] 抗腫瘍T細胞の機能評価を行う上でのポイント2021
- Author(s)
  籠谷　勇紀
- Organizer
  日本再生医療学会
- Invited
- Data Source
  KAKENHI-PROJECT-19K22552
[Presentation] Epigenetic modification of CAR-T cells for optimal adoptive immunotherapy,2020
- Author(s)
  籠谷勇紀
- Organizer
  第79回日本癌学会学術集会
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-20H03543
[Presentation] 合成生物学による免疫細胞療法の改良2020
- Author(s)
  籠谷勇紀
- Organizer
  日本輸血・細胞治療学会
- Invited
- Data Source
  KAKENHI-PROJECT-20H03543
[Presentation] 免疫工学によるCAR-T細胞の改良開発2020
- Author(s)
  籠谷勇紀
- Organizer
  血液疾患免疫療法学会
- Invited
- Data Source
  KAKENHI-PROJECT-20H03543
[Presentation] 合成生物学による免疫細胞療法の改良2020
- Author(s)
  籠谷　勇紀
- Organizer
  日本輸血・細胞治療学会
- Invited
- Data Source
  KAKENHI-PROJECT-19K22552
[Presentation] Epigenetic modification of CAR-T cells for optimal adoptive immunotherapy2020
- Author(s)
  籠谷　勇紀
- Organizer
  第79回日本癌学会学術集会
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-19K22552
[Presentation] 免疫工学によるCAR-T細胞の改良開発2020
- Author(s)
  籠谷　勇紀
- Organizer
  血液疾患免疫療法学会
- Invited
- Data Source
  KAKENHI-PROJECT-19K22552
[Presentation] Genetic manipulation of antitumor T cells to elicit durable clinical response in adoptive immunotherapy2019
- Author(s)
  Yuki Kagoya
- Organizer
  SITC 2019 World Immunotherapy Council’s 3rd Young Investigator Symposium
- Invited / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-19K22552
[Presentation] JAK2V617F mutation evokes paracrine DNA damage to adjacent cells and drives leukemic transformation.2013
- Author(s)
  籠谷勇紀、吉見昭秀、鶴田貴子、片岡圭亮、荒井俊也、黒川峰夫
- Organizer
  第76回日本血液学会学術集会
- Place of Presentation
  札幌
- Data Source
  KAKENHI-PROJECT-25893047
[Presentation] JAK2V617F mutation evokes paracrine DNA damage to adjacent cells and drives leukemic transformation.2013
- Author(s)
  籠谷勇紀、吉見昭秀、鶴田貴子、片岡圭亮、荒井俊也、黒川峰夫
- Organizer
  第72回日本癌学会総会
- Place of Presentation
  横浜
- Data Source
  KAKENHI-PROJECT-25893047
[Presentation] JAK2V617F mutation evokes paracrine DNA damage to adjacent normal cells via secretion of lipocalin-22013
- Author(s)
  Yuki Kagoya, Shunya Arai, Akihide Yoshimi, Takako Tsuruta-Kishino1, Keisuke Kataoka, Mineo Kurokawa
- Organizer
  55th ASH Annual Meeting and Exposition
- Place of Presentation
  New Orleans
- Data Source
  KAKENHI-PROJECT-25893047

1. 松下博和 (80597782)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
2. 黒田浩章 (20365274)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
3. 真砂勝泰 (80338160)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
4. 山口類 (90380675)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
5. 黒川峰夫

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results
6. 吉見昭秀

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

Kagoya Yuki 籠谷 勇紀

KAGOYA YUKI 籠谷 勇紀

Research Projects

Research Products

Co-Researchers

The determination of T-cell fate by different tumor antigens and prevention of post-operative recurrence of lung cancer

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Development of a combinatorial approach of antibody therapeutics and adoptive immunotherapy for cancerPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Generation of long-surviving antitumor T cells by genetic modification for optimal adoptive immunotherapyPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Development of a chimeric synthetic receptor to suppress T cell exhaustion for use in adoptive cancer immunotherapyPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Elucidation of the mechanism of transformation of myeloproliferative neoplasm into acute myelogenous leukemia for novel therapeutic strategy for the disease.Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] Genetic Ablation of HLA Class I, Class II, and the T-cell Receptor Enables Allogeneic T Cells to Be Used for Adoptive T-cell Therapy2020

Author(s)

Journal Title

DOI

Data Source

[Journal Article] JAK2V617F-positive myeloproliferative neoplasm clones evoke paracrine DNA damage to adjacent normal cells through secretion of lipocalin-22014

Author(s)

Journal Title

DOI

Data Source

[Presentation] 抗腫瘍T細胞の機能評価を行う上でのポイント2021

Author(s)

Organizer

Data Source

[Presentation] 抗腫瘍T細胞の機能評価を行う上でのポイント2021

Author(s)

Organizer

Data Source

[Presentation] Epigenetic modification of CAR-T cells for optimal adoptive immunotherapy,2020

Author(s)

Organizer

Data Source

[Presentation] 合成生物学による免疫細胞療法の改良2020

Author(s)

Organizer

Data Source

[Presentation] 免疫工学によるCAR-T細胞の改良開発2020

Author(s)

Organizer

Data Source

[Presentation] 合成生物学による免疫細胞療法の改良2020

Author(s)

Organizer

Data Source

[Presentation] Epigenetic modification of CAR-T cells for optimal adoptive immunotherapy2020

Author(s)

Organizer

Data Source

[Presentation] 免疫工学によるCAR-T細胞の改良開発2020

Author(s)

Organizer

Data Source

[Presentation] Genetic manipulation of antitumor T cells to elicit durable clinical response in adoptive immunotherapy2019

Author(s)

Organizer

Kagoya Yuki 籠谷勇紀

KAGOYA YUKI 籠谷勇紀