OKA Chio 岡千緒

… Alternative Names

Researcher Number	30263445
Other IDs	https://orcid.org/0000-0001-9352-461X
Affiliation (Current)	2022: 奈良先端科学技術大学院大学, 先端科学技術研究科, 助教
Affiliation (based on the past Project Information) *help	2022: 奈良先端科学技術大学院大学, その他の研究科, 助教 2021: 奈良先端科学技術大学院大学, 先端科学技術研究科, 助教 2019: 奈良先端科学技術大学院大学, 先端科学技術研究科, 助教 2007: Nara Institute of Science and Technology, School of Biological Sciences, Assistant Professor 2004 – 2006: 奈良先端科学技術大学院大学, バイオサイエンス研究科, 助手 2000 – 2001: 奈良先端科学技術大学院大学, バイオサイエンス研究科, 助手 1996: 奈良先端科学技術大学院大学, バイオサイエンス研究科, 助手
Review Section/Research Field	Principal Investigator Basic Section 56060:Ophthalmology-related / Developmental biology / Pathological medical chemistry Except Principal Investigator General medical chemistry / Pathological medical chemistry / General medical chemistry / Basic Section 56020:Orthopedics-related
Keywords	Principal Investigator TGF-β / HtrA1 / 細胞運命決定 / 側方抑制 / 分子生物学 / 関節炎 / HtrAl / セリンプロテアーゼ / 肥大化軟骨 / ノックアウトマウス … More Except Principal Investigator コラーゲン / 遺伝子破壊マウス / HtrA1 / セリンプロテアーゼ / TGF-βファミリー / 変形性骨関節炎 / 細胞外期質 / フォリスタチン様蛋白質 / 関節炎モデル動物 / 軟骨細胞 / フォリスタチン様ドメイン / セリン蛋白質分解酵素 / 蛋白質分解酵素阻害剤 / LDL受容体ファミリー / PICK-1 / JIP-1 / JNK / 晩発性アルツハイマー病 / 細胞内取り込み / グルタミン酸受容体 / MAPキナーゼカスケード / LRP1B / JNKスカフォールド / PICK1 / NPXYモチーフ / アミロイドβ / HtrAl / 変形性関節炎 / コラーゲン分泌 / C-プロペプチド / 骨密度 / オステオカルシン / 石灰化 / 肥大軟骨細胞 / HtrAセリンプロテアーゼ / 関節炎 / 加齢黄斑変性症 / bFGF / 軟骨分化 / 骨芽細胞分化 / 細胞外基質 / プロテオグリカン / TGF-β / 肥大軟骨 / 骨芽細胞 / Osteoarthritis / Collagens / C-pronentide / Bone density / Osteocalcine / Calcification / Hypertrophic chondrocvte / 骨代謝 / 破骨細胞 / シグナル伝達 / 免疫学 / 糖鎖 Less

加齢黄斑変性の発症に関わるセリンプロテアーゼHtrA1の基質の同定と機能解析Principal Investigator
- Principal Investigator
  
  岡千緒
- Project Period (FY)
  2022 – 2024
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 56060:Ophthalmology-related
- Research Institution
  Nara Institute of Science and Technology
破骨細胞におけるDAP12を介したITAMシグナル経路を制御する分子機構の解明
- Principal Investigator
  
  北川教弘
- Project Period (FY)
  2021 – 2023
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 56020:Orthopedics-related
- Research Institution
  Nara Institute of Science and Technology
加齢黄斑変性の発症に関わるセリンプロテアーゼHtrA1の基質の同定と機能解析Principal Investigator
- Principal Investigator
  
  岡千緒
- Project Period (FY)
  2019 – 2021
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 56060:Ophthalmology-related
- Research Institution
  Nara Institute of Science and Technology
Study on HtrA Serine Protea we Family Proteins Which Inhibit Signaling of TGF-βs
- Principal Investigator
  
  KAWAICHI Masashi
- Project Period (FY)
  2005 – 2007
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  General medical chemistry
- Research Institution
  Nara Institute of Science and Technology
How HtrA1 serine protease inhibit TGF-β signal and is involved in arthritis ?Principal Investigator
- Principal Investigator
  
  OKA Chio
- Project Period (FY)
  2005 – 2006
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Pathological medical chemistry
- Research Institution
  Nara Institute of Science and Technology (NAIST)
新しいアルツハイマー病関連遺伝子LRP1Bの研究
- Principal Investigator
  
  川市正史
- Project Period (FY)
  2004 – 2005
- Research Category
  
  Grant-in-Aid for Exploratory Research
- Research Field
  
  Pathological medical chemistry
- Research Institution
  Nara Institute of Science and Technology
セリンプロテアーゼ、S8の阻害による関節炎発症抑制の検討
- Principal Investigator
  
  KAWAICHI Masashi
- Project Period (FY)
  2000 – 2001
- Research Category
  
  Grant-in-Aid for Exploratory Research
- Research Field
  
  General medical chemistry
- Research Institution
  Nara Institute of Science and Technology
P19細胞および網膜細胞の分化系におけるNotchシグナル伝達経路の解明Principal Investigator
- Principal Investigator
  
  岡千緒
- Project Period (FY)
  1996
- Research Category
  
  Grant-in-Aid for Encouragement of Young Scientists (A)
- Research Field
  
  Developmental biology
- Research Institution
  Nara Institute of Science and Technology

All 2006 2005

All Journal Article Presentation

[Journal Article] Expression of mouse HtrA1 serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis2005
- Author(s)
  Tsuchiya, A., Yano, M., Tocharus, J., Kojima, H., Fukumoto, M., Kawaichi, M., Oka, C
- Journal Title
  
  Bone 37
  
  Pages: 323-336
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17390079
[Journal Article] Expression of mouse HtrA1 serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis.2005
- Author(s)
  Tsuchiya, A., Yano, M., Tocharus, J., Kojima, H., Fukumoto, M., Kawaichi, M., Oka, C.
- Journal Title
  
  Bone 37
  
  Pages: 323-336
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17590268
[Journal Article] Expression of mouse HtrA1 serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis.2005
- Author(s)
  Tsuchiya, A., Yano, M., Tocharus, J., Kojima, H., Fukumoto, M., Kawaichi, M., Oka, C
- Journal Title
  
  Bone 37
  
  Pages: 323-336
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17590268
[Journal Article] Expression of mouse HtrAl serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis2005
- Author(s)
  Tsuchiya, A., Yano, M., Tocharus, J., Kojima, H., Fukumoto, M., Kawaichi, M. and Oka, C.
- Journal Title
  
  Bone 37
  
  Pages: 323-336
- Description
  「研究成果報告書概要(和文)」より
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-17390079
[Presentation] Roles of mouse HtrA1 serine protease in bone development and pathogenesis of osteoarthritis2006
- Author(s)
  Hisae, Kojima, Akiho, Tsuchiya, Masashi, Kawaichi, Chio, Oka
- Organizer
  20th IUBMB International Congress
- Place of Presentation
  Kyoto
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17390079
[Presentation] Mammalian HtrAl is a highly regulated serine protease2006
- Author(s)
  Chio Oka, Murwantoko, Masato Yano, Yoshifumi Ueta, Hisae Kojima, Akiho Tsuchiya, Masashi Kawaichi
- Organizer
  20th IUBMB International Congress.
- Place of Presentation
  Kyoto
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17390079
[Presentation] Roles of mouse HtrAl serine protease in bone development and pathogenesis of osteoarthritis2006
- Author(s)
  Hisae Kojima, Akiho Tsuchiya, Masashi Kawaichi, Chio Oka
- Organizer
  20th IUBMB International Congress.
- Place of Presentation
  Kyoto
- Description
  「研究成果報告書概要(和文)」より
- Data Source
  KAKENHI-PROJECT-17390079
[Presentation] Mammalian HtrA1 is a highly regulated serine protease2006
- Author(s)
  Chio, Oka, Murwantoko, Masato, Yano, Yoshifumi, Ueta, Hisae, Kojima, Akiho, Tsuchiya, Masashi, Kawaichi
- Organizer
  20th IUBMB International Congress
- Place of Presentation
  Kyoto
- Description
  「研究成果報告書概要(欧文)」より
- Data Source
  KAKENHI-PROJECT-17390079

1. KAWAICHI Masashi (00195041)

# of Collaborated Projects: 4 results

# of Collaborated Products: 8 results
2. 北川教弘 (30294284)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
3. 別所康全 (70261253)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
4. 山下照仁 (90302893)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results

OKA Chio 岡 千緒

岡 千緒 オカ チオ

Research Projects

Research Products

Co-Researchers

加齢黄斑変性の発症に関わるセリンプロテアーゼHtrA1の基質の同定と機能解析Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

破骨細胞におけるDAP12を介したITAMシグナル経路を制御する分子機構の解明

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

加齢黄斑変性の発症に関わるセリンプロテアーゼHtrA1の基質の同定と機能解析Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Study on HtrA Serine Protea we Family Proteins Which Inhibit Signaling of TGF-βs

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

How HtrA1 serine protease inhibit TGF-β signal and is involved in arthritis ?Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

新しいアルツハイマー病関連遺伝子LRP1Bの研究

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

セリンプロテアーゼ、S8の阻害による関節炎発症抑制の検討

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

P19細胞および網膜細胞の分化系におけるNotchシグナル伝達経路の解明Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] Expression of mouse HtrA1 serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis2005

Author(s)

Journal Title

Description

Data Source

[Journal Article] Expression of mouse HtrA1 serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis.2005

Author(s)

Journal Title

Description

Data Source

[Journal Article] Expression of mouse HtrA1 serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis.2005

Author(s)

Journal Title

Description

Data Source

[Journal Article] Expression of mouse HtrAl serine protease in normal bone and cartilage and its upregulation in joint cartilage damaged by experimental arthritis2005

Author(s)

Journal Title

Description

Data Source

[Presentation] Roles of mouse HtrA1 serine protease in bone development and pathogenesis of osteoarthritis2006

Author(s)

Organizer

Place of Presentation

Description

Data Source

[Presentation] Mammalian HtrAl is a highly regulated serine protease2006

OKA Chio 岡千緒

岡千緒オカチオ