itoh junta 伊東順太

… Alternative Names

伊東順太イトウジュンタ

ITO Junta 伊東順太

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Researcher Number	40609096
Affiliation (Current)	2025: 城西大学, 薬学部, 助教
Affiliation (based on the past Project Information) *help	2025: 城西大学, 薬学部, 助教 2021 – 2023: 城西大学, 薬学部, 助教 2016 – 2019: 城西大学, 薬学部, 助教 2011 – 2015: 明海大学, 歯学部, 助教
Review Section/Research Field	Principal Investigator Basic Section 59020:Sports sciences-related / Periodontology / Pathobiological dentistry/Dental radiology Except Principal Investigator Morphological basic dentistry / Digestive surgery
Keywords	Principal Investigator スダチチン / 破骨細胞形成 / 骨代謝 / 破骨細胞 / 炎症性骨破壊 / 抗酸化作用 / 酸化ストレス / 運動関連消化器症状 / 運動誘発性胃腸症候群 / 小腸粘膜 … More / タイトジャンクション / 運動誘発性胃腸障害 / 運動誘発性腸管粘膜傷害 / 歯周病 / 歯学 / ポリメトキシフラボノイド / RANKL / 骨芽細胞 / LOX-1 … More Except Principal Investigator 細胞融合 / LOX-1 / 破骨細胞 / 炎症性骨破壊 / 脂質 / 炎症整骨破壊 / ABCG1 / phosphatidylethanolamine / 細胞膜外葉 / LDL受容体 / RANKL産生 / 炎症性骨吸収 / 破骨細胞形成 / 骨芽細胞 / 腸蠕動運動 / 細胞増殖 / アポトーシス / ROS / グルタミン / nNOS / iNOS / 廃用性腸管萎縮 / 神経型NO合成酵素 / 誘導型NO合成酵素 / 再摂食 / 絶食 / 廃用性腸管粘膜萎縮 Less

Tight junctionに着目したスダチチンによる運動誘発性胃腸障害の予防メカニズムの解明Principal Investigator
- Principal Investigator
  
  伊東順太
- Project Period (FY)
  2025 – 2027
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 59020:Sports sciences-related
- Research Institution
  Josai University
運動誘発性腸管粘膜傷害に対する酸化ストレス制御を介したスダチチンの予防効果Principal Investigator
- Principal Investigator
  
  伊東順太
- Project Period (FY)
  2021 – 2024
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Review Section
  
  Basic Section 59020:Sports sciences-related
- Research Institution
  Josai University
Elucidation of Involvement of LOX-1 in inflammatory bone destruction and the molecular mechanism for the LOX-1 actions, and an approach to develop the new drug for bone diseases.
- Principal Investigator
  
  HAKEDA Yoshiyuki
- Project Period (FY)
  2016 – 2019
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  Morphological basic dentistry
- Research Institution
  Meikai University
Aims to establish a new preventive method for periodontal alveolar bone resorption using sudachi pericarp specific flavonoidsPrincipal Investigator
- Principal Investigator
  
  Ito Junta
- Project Period (FY)
  2016 – 2019
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Periodontology
- Research Institution
  Josai University
Elucidation of mechanisms underlying inflammation-induced LOX-1-dependent RANKL expression in osteoblastsPrincipal Investigator
- Principal Investigator
  
  Ito Junta
- Project Period (FY)
  2014 – 2015
- Research Category
  
  Grant-in-Aid for Young Scientists (B)
- Research Field
  
  Pathobiological dentistry/Dental radiology
- Research Institution
  Meikai University
The elucidation of the role of lipid in bone resorption of inflammatory bone diseases; in particular, the exploration of the role of LOX-1 as an oxidized LDL receptor
- Principal Investigator
  
  HAKEDA Yoshiyuki
- Project Period (FY)
  2013 – 2015
- Research Category
  
  Grant-in-Aid for Scientific Research (B)
- Research Field
  
  Morphological basic dentistry
- Research Institution
  Meikai University
The role of neuronal and inducible NOS in the regulation of intestinal apoptosis in fasting and refeeding rat
- Principal Investigator
  
  UCHIDA Hiroyuki
- Project Period (FY)
  2010 – 2012
- Research Category
  
  Grant-in-Aid for Scientific Research (C)
- Research Field
  
  Digestive surgery
- Research Institution
  Josai University

All 2022 2020 2019 2018 2016 2015 2014 2013 2011 2010 Other

All Journal Article Presentation

[Journal Article] 絶食誘導腸管粘膜萎縮と予防栄養2022
- Author(s)
  内田博之，伊東順太
- Journal Title
  
  化学と生物
  
  Volume: 60(10) Pages: 509-517
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-21K11366
[Journal Article] LDL uptake-dependent phosphatidylethanolamine translocation to the cell surface promotes fusion of osteoclast-like cells.2020
- Author(s)
  Kitano J F V, Ohyama Y, Hayashida C, Ito J, Okayasu M, Sato T, Ogasawara T, Tsujita M, Kakino A, Shimada J, Sawamura T, and Hakeda Y.
- Journal Title
  
  Journal of Cell Science
  
  Volume: ー
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-16H05505
[Journal Article] Reducing reactive oxygen spicies (ROS) in gum disease2019
- Author(s)
  Junta Ito
- Journal Title
  
  Impact
  
  Volume: June Pages: 71-73
- Data Source
  KAKENHI-PROJECT-16K20677
[Journal Article] スダチ果皮特有フラボノイドsudachitinの炎症性骨破壊に対する抑制効果2019
- Author(s)
  伊東順太
- Journal Title
  
  Bio Clinica
  
  Volume: 34 Pages: 41-47
- Data Source
  KAKENHI-PROJECT-16K20677
[Journal Article] 炎症性骨破壊に対するスダチ果皮特有フラボノイドsudachitinの抑制効果2019
- Author(s)
  伊東順太
- Journal Title
  
  Precision Medicine
  
  Volume: 2 Pages: 96-102
- Data Source
  KAKENHI-PROJECT-16K20677
[Journal Article] The polymethoxy flavonoid sudachitin suppresses inflammatory bone destruction by directly inhibiting osteoclastogenesis due to reduced ROS production and MAPK activation in osteoclast precursors2018
- Author(s)
  Ohyama Yoko、Ito Junta、Kitano Victor J.、Shimada Jun、Hakeda Yoshiyuki
- Journal Title
  
  PLOS ONE
  
  Volume: 13 Issue: 1 Pages: e0191192-e0191192
- DOI
  10.1371/journal.pone.0191192
- Peer Reviewed / Open Access / Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16K20677, KAKENHI-PROJECT-16H05505
[Journal Article] Lectin-like oxidized low-density lipoprotein receptor-1 abrogation causes resistance to inflammatory bone destruction in mice, despite promoting osteoclastogenesis in the steady state.2015
- Author(s)
  Nakayachi M, Ito J, Hayashida C, Ohyama Y, Kakino A, Okayasu M, Sato T, Ogasawara T, Kaneda T, Suda N, Sawamura T, Hakeda Y
- Journal Title
  
  Bone. 75:170-182
  
  Volume: 75 Pages: 170-182
- DOI
  10.1016/j.bone.2015.02.025
- Peer Reviewed / Open Access
- Data Source
  KAKENHI-PROJECT-24659916, KAKENHI-PROJECT-25293422, KAKENHI-PROJECT-26870540, KAKENHI-PROJECT-26463085, KAKENHI-PROJECT-15K15759, KAKENHI-PROJECT-26670895, KAKENHI-PROJECT-25293376
[Journal Article] Fasting-induced intestinal apoptosis is mediated by inducible nitric oxide synthase and interferon-γ in rat.2010
- Author(s)
  J Ito, H Uchida, et.al.
- Journal Title
  
  Am J Physiol Gastrointest Liver Physiol
  
  Volume: 298
- NAID
  120005823458
- Peer Reviewed
- Data Source
  KAKENHI-PROJECT-22591492
[Journal Article] Fasting-induced intestinal apoptosis is mediated by inducible nitric oxide synthase and interferon-r in rat2010
- Author(s)
  J Ito, H Uchida, et.al
- Journal Title
  
  Am J Physiol Gastrointest liver Physiol
  
  Volume: 298 Pages: 916-926
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] スダチ果皮特有ポリメトキシフラボノイドsudachitinは破骨細胞形成およびLPS誘導炎症性骨破壊を抑制する2016
- Author(s)
  伊東順太、大山洋子、Jose Kitano Flores Victor，林田千代美、佐藤卓也、羽毛田慈之
- Organizer
  第34回日本骨代謝学会学術集会
- Place of Presentation
  大阪
- Year and Date
  2016-07-20
- Data Source
  KAKENHI-PROJECT-16K20677
[Presentation] スダチ果皮特有ポリメトキシフラボノイドsudachitinは破骨細胞形成およびLPS誘導炎症性骨破壊を抑制する2016
- Author(s)
  伊東順太、大山洋子、林田千代美、佐藤卓也、羽毛田慈之
- Organizer
  第34回日本骨代謝学会学術集会・第3回アジア太平洋骨代謝学会議
- Place of Presentation
  大阪国際会議場
- Year and Date
  2016-07-20
- Data Source
  KAKENHI-PROJECT-16H05505
[Presentation] Sudachitin, a polymethoxyflavone derived from Citrus sudachi, suppresses lipopolysaccharide-induced inflammatory bone resorption because of inhibiting osteoclast formation in mice2016
- Author(s)
  Ohyama Y., Ito J.,Hakeda Y., Shimada J.
- Organizer
  98th Annual Meeting of American Association of Oral and Maxillofacial Surgeons
- Place of Presentation
  Las Vegas, USA
- Year and Date
  2016-09-18
- Int'l Joint Research
- Data Source
  KAKENHI-PROJECT-16H05505
[Presentation] レクチン様酸化LDL受容体-1 (LOX-1)欠損は定常状態における破骨細胞形成を促進するが、LPS誘導炎症性骨破壊に抵抗性を示す2015
- Author(s)
  伊東順太、林田千代美、大山洋子、佐藤卓也、羽毛田慈之
- Organizer
  第33回日本骨代謝学会学術集会
- Place of Presentation
  京王プラザホテル（新宿）
- Year and Date
  2015-07-23
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] レクチン様酸化LDL受容体-1 (LOX-1)欠損は定常状態における破骨細胞形成を促進するが、LPS誘導炎症性骨破壊に抵抗性をもたらす2015
- Author(s)
  伊東順太、林田千代美、大山洋子、佐藤卓也、羽毛田慈之
- Organizer
  第33回日本骨代謝学会学術集会
- Place of Presentation
  新宿（東京）
- Year and Date
  2015-07-23
- Data Source
  KAKENHI-PROJECT-26870540
[Presentation] レクチン様酸化LDL受容体-1 (LOX-1)欠損は定常状態の破骨細胞形成を促進するが、LPS誘導炎症性骨破壊には抵抗性を示す2015
- Author(s)
  伊東順太、林田千代美、大山洋子、佐藤卓也、羽毛田慈之
- Organizer
  第57回歯科基礎医学会学術大会
- Place of Presentation
  新潟（新潟）
- Year and Date
  2015-09-11
- Data Source
  KAKENHI-PROJECT-26870540
[Presentation] 炎症性骨破壊におけるRANKLの発現に対するlectin-like oxidized LDL receptor-1(LOX-1)の役割に関する研究2014
- Author(s)
  伊東順太
- Organizer
  明海歯科医学会第23回学術大会
- Place of Presentation
  坂戸（埼玉）
- Year and Date
  2014-06-05
- Data Source
  KAKENHI-PROJECT-26870540
[Presentation] レクチン様酸化LDL受容体-1(LOX-1)の破骨細胞形成と炎症性骨破壊に対する役割の解明2013
- Author(s)
  伊東順太、中谷地舞、林田千代美、岡安麻里、大山洋子、佐藤卓也、羽毛田慈之
- Organizer
  第55回歯科基礎医学会学術大会・総会
- Place of Presentation
  岡山コンベンションセンター、岡山、岡山県
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] Role of lectin-like oxidized low-density of lipoprotein receptor-1 in regulating osteoclastogenesis and inflammatory bone destruction.2013
- Author(s)
  Nakayachi M, Ito J, Okayasu M, Hayashida C, Sato T, Suda N, Sawamura T, Hakeda
- Organizer
  2nd Joint Meeting of the International Bone and Mineral Society and the Japanese Society for Bone and Mineral Research
- Place of Presentation
  Kobe Convention Center and Kobe Portpia Hotel, Kobe, Japan
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] Lectin-like oxidized low-density of lipoprotein receptor-1 is involved in RANKL-production elevated by lipopolysaccharide-injection on mouse calvaria and thereby contributes to inflammatory bone destruction2013
- Author(s)
  Nakayachi M, Ito J, Okayasu M, Hayashida C, Sato T, Suda N, Sawamura T, Hakeda Y.
- Organizer
  35th Annual Meeting of the American Society for Bone and Mineral Research
- Place of Presentation
  Baltimore Convention Center, Baltimore, Maryland, USA
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] レクチン様酸化LDL受容体-１（LOX-1）の破骨細胞形成および炎症性骨吸収に果たす役割の解明2013
- Author(s)
  中谷地舞、伊東順太、岡安麻里、須田直人、羽毛田慈之
- Organizer
  第72回日本矯正歯科学会大会
- Place of Presentation
  キッセイ文化ホール・松本市総合体育館、松本、長野県
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] 絶食後再摂食ラットの腸管粘膜における神経型および誘導型NO合成酵素の相互作用2011
- Author(s)
  内田博之、伊東順太ら
- Organizer
  日本薬学会第131年会
- Place of Presentation
  グランシップ(静岡県)
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] 絶食後再摂食ラットの腸管粘膜における神経型および誘導型NO合成酵素の相互作用2011
- Author(s)
  内田博之、伊東順太, ら
- Organizer
  日本薬学会第131年会
- Place of Presentation
  グランシップ等(静岡県)
- Year and Date
  2011-03-29
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] 再摂食による腸管粘膜の回復過程における誘導型NO合成酵素(iNOS)によるアポトーシスの制御2011
- Author(s)
  伊東順太、内田博之, ら
- Organizer
  日本薬学会第131年会
- Place of Presentation
  グランシップ等(静岡県)
- Year and Date
  2011-03-29
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] 再摂食による腸管粘膜の回復過程における誘導型NO合成酵素(iNOS)によるアポトーシスの制御2011
- Author(s)
  伊東順太、内田博之ら
- Organizer
  日本薬学会第131年会
- Place of Presentation
  グランシップ(静岡県)
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] The role of neuronal and inducible NOS in the regulation of intestinal apoptosis in fasting and refeeding rat.2010
- Author(s)
  Junta Ito, Hiroyuki Uchida, et.al.
- Organizer
  The 6^<th> International Conference on the Biology, Chemistry, and Therapeutic Applications of Nitric Oxide
- Place of Presentation
  Kyoto International Conference Center(Kyoto, Japan)
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] The role of neuronal and inducible NOS in the regulation of intestinal apoptosis in fasting and refeeding rat2010
- Author(s)
  Ito J, Uchida H, et.al
- Organizer
  The 6th international conference on the biology, chemistry, and therapeutic applications of nitric oxide
- Place of Presentation
  Kyoto international conference center (Kyoto, Japan)
- Data Source
  KAKENHI-PROJECT-22591492
[Presentation] レクチン様酸化LDL受容体-1 (LOX-1)欠損マウスは骨量を減少させるが炎症性骨吸収に抵抗を示す
- Author(s)
  中谷地舞, 伊東順太, 岡安麻里, 須田直人, 羽毛田慈之
- Organizer
  第74回日本矯正歯科学会大会・第5回日韓ジョイントミーティンク
- Place of Presentation
  幕張
- Year and Date
  2014-10-20 – 2014-10-22
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] レクチン様酸化LDL受容体-1 (LOX-1)欠損は定常状態における破骨細胞形成を促進するが、LPS誘導炎症性骨破壊に抵抗性を示す
- Author(s)
  伊東順太、林田千代美、大山洋子、佐藤卓也、羽毛田慈之
- Organizer
  第33回日本骨代謝学会学術集会
- Place of Presentation
  東京
- Year and Date
  2015-07-23 – 2015-07-25
- Data Source
  KAKENHI-PROJECT-25293376
[Presentation] レクチン様酸化LDL受容体-1 (LOX-1)欠損マウスは骨量を減少させるが炎症性骨吸収に抵抗を示す
- Author(s)
  中谷地舞,　伊東順太,　岡安麻里,　須田直人,　羽毛田慈之
- Organizer
  第73回日本矯正歯科学会大会
- Place of Presentation
  幕張（千葉）
- Year and Date
  2014-10-20 – 2014-10-22
- Data Source
  KAKENHI-PROJECT-26870540

1. HAKEDA Yoshiyuki (90164772)

# of Collaborated Projects: 2 results

# of Collaborated Products: 10 results
2. OKAYASU Mari (10610941)

# of Collaborated Projects: 2 results

# of Collaborated Products: 6 results
3. OGASAWARA Toru (20359623)

# of Collaborated Projects: 2 results

# of Collaborated Products: 1 results
4. SAWAMURA Tatsuya (30243033)

# of Collaborated Projects: 2 results

# of Collaborated Products: 3 results
5. UCHIDA Hiroyuki (20245195)

# of Collaborated Projects: 1 results

# of Collaborated Products: 8 results
6. OTAKE Kazuo (50337482)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
7. SATO Takuya (00316689)

# of Collaborated Projects: 1 results

# of Collaborated Products: 5 results
8. HAYASHIDA Chiyomi (40710900)

# of Collaborated Projects: 1 results

# of Collaborated Products: 5 results
9. KANEDA Toshio (70339521)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
10. 森芳史 (60757954)

# of Collaborated Projects: 1 results

# of Collaborated Products: 0 results
11. 須田直人

# of Collaborated Projects: 0 results

# of Collaborated Products: 1 results

itoh junta 伊東 順太

伊東 順太 イトウ ジュンタ

ITO Junta 伊東 順太

Research Projects

Research Products

Co-Researchers

Tight junctionに着目したスダチチンによる運動誘発性胃腸障害の予防メカニズムの解明Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

運動誘発性腸管粘膜傷害に対する酸化ストレス制御を介したスダチチンの予防効果Principal Investigator

Principal Investigator

Project Period (FY)

Research Category

Review Section

Research Institution

Elucidation of Involvement of LOX-1 in inflammatory bone destruction and the molecular mechanism for the LOX-1 actions, and an approach to develop the new drug for bone diseases.

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Aims to establish a new preventive method for periodontal alveolar bone resorption using sudachi pericarp specific flavonoidsPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

Elucidation of mechanisms underlying inflammation-induced LOX-1-dependent RANKL expression in osteoblastsPrincipal Investigator

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

The elucidation of the role of lipid in bone resorption of inflammatory bone diseases; in particular, the exploration of the role of LOX-1 as an oxidized LDL receptor

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

The role of neuronal and inducible NOS in the regulation of intestinal apoptosis in fasting and refeeding rat

Principal Investigator

Project Period (FY)

Research Category

Research Field

Research Institution

[Journal Article] 絶食誘導腸管粘膜萎縮と予防栄養2022

Author(s)

Journal Title

Data Source

[Journal Article] LDL uptake-dependent phosphatidylethanolamine translocation to the cell surface promotes fusion of osteoclast-like cells.2020

Author(s)

Journal Title

Data Source

[Journal Article] Reducing reactive oxygen spicies (ROS) in gum disease2019

Author(s)

Journal Title

Data Source

[Journal Article] スダチ果皮特有フラボノイドsudachitinの炎症性骨破壊に対する抑制効果2019

Author(s)

Journal Title

Data Source

[Journal Article] 炎症性骨破壊に対するスダチ果皮特有フラボノイドsudachitinの抑制効果2019

Author(s)

Journal Title

Data Source

[Journal Article] The polymethoxy flavonoid sudachitin suppresses inflammatory bone destruction by directly inhibiting osteoclastogenesis due to reduced ROS production and MAPK activation in osteoclast precursors2018

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Lectin-like oxidized low-density lipoprotein receptor-1 abrogation causes resistance to inflammatory bone destruction in mice, despite promoting osteoclastogenesis in the steady state.2015

Author(s)

Journal Title

DOI

Data Source

[Journal Article] Fasting-induced intestinal apoptosis is mediated by inducible nitric oxide synthase and interferon-γ in rat.2010

Author(s)

itoh junta 伊東順太

伊東順太イトウジュンタ

ITO Junta 伊東順太