• Search Research Projects
  • Search Researchers
  • How to Use
  1. Back to previous page

Yuasa Tomoyuki  湯浅 智之

ORCIDConnect your ORCID iD *help
… Alternative Names

YUASA Tomoyuki  湯浅 智之

Less
Researcher Number 50304556
Affiliation (Current) 2022: 徳島大学, 大学院医歯薬学研究部(医学域), 特任准教授
Affiliation (based on the past Project Information) *help 2019 – 2022: 徳島大学, 大学院医歯薬学研究部(医学域), 特任准教授
2016 – 2017: 徳島大学, 大学院医歯薬学研究部(医学系), 特任准教授
2015: 徳島大学, 大学院医歯薬学研究部, 特任准教授
2010 – 2013: 徳島大学, 疾患酵素学研究センター, 准教授
2007: The University of Tokushima, Institute for Enzyme Research, Assistant Professor
1998 – 2006: 徳島大学, 分子酵素学研究センター, 助手
Review Section/Research Field
Principal Investigator
Pathological medical chemistry / Basic Section 54040:Metabolism and endocrinology-related / Metabolomics
Except Principal Investigator
General medical chemistry / Pathological medical chemistry / Pathological medical chemistry / Biological Sciences / General medical chemistry / Structural biochemistry / Structural biochemistry
Keywords
Principal Investigator
可溶性インスリン受容体 / インスリン抵抗性 / 糖尿病 / シグナル伝達 / 妊娠糖尿病 / 女性ホルモン / インスリン受容体切断
Except Principal Investigator
糖尿病 / APS / インスリン受容体 … More / インスリン作用 / GLUT4トランスロケーション / インスリンシグナル伝達 / internalization / シグナル伝達 / インスリン / diabetes / PDGF / プロテオーム解析 / Akt / GLUT4 / トランスジェニックマウス / ノックアウトマウス / ユビキチン化 / glucose uptake / GLUT4 translocation / Gq / insulin receptor / ARF6 / 血糖値降下 / NIDDM / ポリモルフィズム / II型糖尿病 / 血中可溶性インスリン受容体 / 糖毒性 / インスリンレセプター / 血中マーカー / Aktキナーゼ / Type 2 diabetes / プロテオミクス / Insulin signalling / グルコース取り込み / インスリン感受性亢進 / ubiquitination / AS160 / トランスロケーション / AKt / 肝臓 / ゲノム的解析 / インスリン受容体遺伝子 / トランスジェニックマウスマウス / ユビチキン化 / 転写因子 / 分子病態学 / TUG / sIR / 高血糖 / 超高感度sIR測定法 / BFA / ARF / BIG2 / 生体分子医学 / 可溶性インスリン受容体 / PI3-キナーゼ / Insulin action / PI3-kinase / Akt kinase / インスリン抵抗性原因物質 / インスリン抵抗性 / トランスジェニックラット / GLUT4myc / インスリン非依存型糖尿病 / Serumsubstance caused for insulin resistance / グルコース利用 / 三量体Gタンパク / AMPキナーゼ / 三量体GタンパクGq / Insulin signal translocation / AMP-activated protein kinase / リン酸化タンパク / 抗糖尿病症 / 糖尿病薬 / 経口糖尿病薬 / protein phosphorylation / proteome / drug / 糖代謝 / APSノックマウス / インスリン感受性 / carbobydrate metabolism / APS knockout mouse / insulin resistance / リン酸化タンパク質 / insulin action / proteomics / KO mouse / Serum insulin receptor / αサブユニット / 結晶構造解析 / 抗糖尿病薬 / human insulin receptor / αsubunits / crystallization / structural analysis / インンスリン受容体 / 血中遊離インスリン受容体 / signal transductor / 可溶性インスリンレセプター / 血糖指標 / 生理活性 / Soluble Insulin Receptor / glycemic marker / 可溶性細胞外ドメイン / Gq共役受容体 / soluble ectodomain / signal transduction / Gq coupled receptor Less
  • Research Projects

    (28 results)
  • Research Products

    (39 results)
  • Co-Researchers

    (15 People)
  •  糖尿病とインスリン抵抗性疾患群を横断するインスリン受容体切断機構の解明Principal Investigator

    • Principal Investigator
      湯浅 智之
    • Project Period (FY)
      2022 – 2024
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 54040:Metabolism and endocrinology-related
    • Research Institution
      The University of Tokushima
  •  Clarification of common mechanism in diabetes-related diseases by insulin receptor cleavagePrincipal Investigator

    • Principal Investigator
      YUASA Tomoyuki
    • Project Period (FY)
      2019 – 2021
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Review Section
      Basic Section 54040:Metabolism and endocrinology-related
    • Research Institution
      The University of Tokushima
  •  Pathological mechanism of sequential cleavage model of insulin receptorPrincipal Investigator

    • Principal Investigator
      YUASA Tomoyuki
    • Project Period (FY)
      2015 – 2017
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Metabolomics
    • Research Institution
      The University of Tokushima
  •  The study of molecular mechanism of insulin receptor cleavagePrincipal Investigator

    • Principal Investigator
      YUASA Tomoyuki
    • Project Period (FY)
      2012 – 2013
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  Is insulin inactivated by the binding with serum soluble insulin receptor(sIR)?

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2011 – 2012
    • Research Category
      Grant-in-Aid for Challenging Exploratory Research
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  The study of the insulin receptor cleavage and its clinical significancePrincipal Investigator

    • Principal Investigator
      YUASA Tomoyuki
    • Project Period (FY)
      2010 – 2011
    • Research Category
      Grant-in-Aid for Young Scientists (B)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of Insulin signal transduction and diabetes mellitus

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2008 – 2010
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of insulin signal transduction and diabetes mellitus

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2006 – 2007
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  インスリン受容体細胞内ドメインは転写因子となりうるのか?

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      2005 – 2006
    • Research Category
      Grant-in-Aid for Exploratory Research
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  Soluble insulin receptor ectodomain is elevated in the plasma of patients with diabetes

    • Principal Investigator
      OBATA Toshiyuki
    • Project Period (FY)
      2005 – 2006
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  APSノックアウトマウスは肥満によるインスリン抵抗性を回避できる

    • Principal Investigator
      小畑 利之, 岸 和弘
    • Project Period (FY)
      2004 – 2005
    • Research Category
      Grant-in-Aid for Scientific Research on Priority Areas
    • Review Section
      Biological Sciences
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of insulin signal transduction and diabetes mellitus

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2004 – 2005
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  ヒト血清中に可溶性インスリンレセプターは存在するか?

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      2003 – 2004
    • Research Category
      Grant-in-Aid for Exploratory Research
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  Crystallization and structural analysis of the human insulin receptor

    • Principal Investigator
      OBATA Toshiyuki
    • Project Period (FY)
      2003 – 2004
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Structural biochemistry
    • Research Institution
      The University of Tokushima
  •  Elucidation of the molecular mechanisms of insulin transduction using APS knockout mice

    • Principal Investigator
      KISHI Kazuhiro
    • Project Period (FY)
      2003 – 2004
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of insulin signal transduction and diabetes mellitus

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2002 – 2003
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  II型糖尿病の遺伝要因のゲノム的解析と分子病態の解明

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      2001
    • Research Category
      Grant-in-Aid for Scientific Research on Priority Areas (C)
    • Review Section
      Biological Sciences
    • Research Institution
      The University of Tokushima
  •  PDGF投与により血糖値が下がるマウスを作れるか?

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      2001
    • Research Category
      Grant-in-Aid for Exploratory Research
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  Development of a drug for diabetes using human genome information

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2001 – 2003
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of glucose uptake by insulin-independent pathway

    • Principal Investigator
      KISHI Kazuhiro
    • Project Period (FY)
      2001 – 2002
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  New cDNAs and proteins phosphorylated by insulin using proteome analysis

    • Principal Investigator
      OBATA Toshiyuki
    • Project Period (FY)
      2001 – 2002
    • Research Category
      Grant-in-Aid for Scientific Research (C)
    • Research Field
      Structural biochemistry
    • Research Institution
      The University of Tokushima
  •  II型糖尿病の遺伝要因のゲノム的解析と分子病態の解明

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      2000
    • Research Category
      Grant-in-Aid for Scientific Research on Priority Areas (C)
    • Review Section
      Biological Sciences
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of insulin signal transduction and diabetas mellitus

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      2000 – 2001
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  肝臓におけるインスリン作用不全がII型糖尿病発症の真の原因か?

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      1999
    • Research Category
      Grant-in-Aid for Exploratory Research
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima
  •  インスリン受容体とその作用メカニズムの解析

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      1998
    • Research Category
      Grant-in-Aid for Scientific Research on Priority Areas (A)
    • Research Institution
      The University of Tokushima
  •  インスリンシグナル伝達転換マウスの作製

    • Principal Investigator
      蛯名 洋介
    • Project Period (FY)
      1998
    • Research Category
      Grant-in-Aid for Exploratory Research
    • Research Field
      Structural biochemistry
    • Research Institution
      The University of Tokushima
  •  Molecular mechanisms of insulin signal transduction and diabetes mellitus

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      1998 – 1999
    • Research Category
      Grant-in-Aid for Scientific Research (B)
    • Research Field
      General medical chemistry
    • Research Institution
      The University of Tokushima
  •  Development of a simple screening system for the discovery of a new drug for diabetes

    • Principal Investigator
      EBINA Yousuke
    • Project Period (FY)
      1998 – 2000
    • Research Category
      Grant-in-Aid for Scientific Research (B).
    • Research Field
      Pathological medical chemistry
    • Research Institution
      The University of Tokushima

All 2021 2019 2016 2015 2014 2013 2012 2011 2010 2009 2008 2004 Other

All Journal Article Presentation Patent

  • [Journal Article] Insulin receptor cleavage induced by estrogen impairs insulin signaling2021

    • Author(s)
      Yuasa Tomoyuki、Takata Yasunori、Aki Nanako、Kunimi Kotaro、Satoh Miki、Nii Mari、Izumi Yoshihiko、Otoda Toshiki、Hashida Seiichi、Osawa Haruhiko、Aihara Ken-ichi
    • Journal Title

      BMJ Open Diabetes Research & Care

      Volume: 9

    • DOI

      10.1136/bmjdrc-2021-002467

    • Peer Reviewed / Open Access
    • Data Source
      KAKENHI-PROJECT-19K09027
  • [Journal Article] Sequential cleavage of insulin receptor by calpain 2 and γ-secretase impairs insulin signalling.2016

    • Author(s)
      Yuasa T, Amo-Shiinoki K, Ishikura S, Takahara M, Matsuoka T, Kaneto H, Kuroda A, Matsuhisa M, Hashida S
    • Journal Title

      Diabetologia

      Volume: 59 Pages: 2711-2721

    • DOI

      10.1007/s00125-016-4102-5

    • Peer Reviewed / Acknowledgement Compliant / Open Access / Int'l Joint Research
    • Data Source
      KAKENHI-PROJECT-15K09391, KAKENHI-PROJECT-16K01846
  • [Journal Article] Development of in vitro model of insulin receptor cleavage induced by high glucose in HepG2 cells2014

    • Author(s)
      Yuasa T, Amo K, Ishikura S, Nagaya H, Uchiyama K, Hashida S, Ebina Y.
    • Journal Title

      Biochem Biophys Res Commun

      Volume: 445 Pages: 354-359

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-24790317
  • [Journal Article] Development of in vitro model of insulin receptor cleavage induced by high glucose in HepG2 cells.2014

    • Author(s)
      ①Yuasa T, Amo K, Ishikura S, Nagaya H, Uchiyama K, Hashida S, Ebina Y
    • Journal Title

      Biochemical and Biophysical Research Communications

      Volume: 445(1) Pages: 236-243

    • DOI

      10.1016/j.bbrc.2014.01.187

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-24790317, KAKENHI-PROJECT-24790929, KAKENHI-PROJECT-25350912, KAKENHI-PROJECT-25870483
  • [Journal Article] The Rab GTPase-Activating Protein AS160 as A Common Regulator of Insulin-and Gαq-Mediated Intracellular GLUT4 Vesicle Distribution2009

    • Author(s)
      Yuasa T.
    • Journal Title

      Endocrine Journal 56(3)

      Pages: 345-359

    • NAID

      10026913437

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Journal Article] The Rab GTPase-Activating Protein AS160 as A Common Regulator of Insulin- and Gq-Mediated Intracellular GLUT4 Vesicle DistributionA2009

    • Author(s)
      Yuasa Tomoyuki, Uchiyama Keiji, Yuko OGURA, Kimura Masafumi, Kiyoshi TESHIGAWARA, Hosaka Toshio, Yoshinori TANAKA, Obata Toshiyuki, Hiroyuki SANO, Kishi Kazuhiro, Ebina Yousuke
    • Journal Title

      Endocrine Journal Vol.56, No.3

      Pages: 345-359

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Journal Article] The Rab GTPase-Activating Protein AS160 as A Common Regulator of Insulin- and Gaq-Mediated Intracellular GLUT4 Vesicle DistributionA2009

    • Author(s)
      Tomoyuki YUASA, et al.
    • Journal Title

      Endocrine Journal in press

    • Peer Reviewed
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF recept or in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Description
      「研究成果報告書概要(和文)」より
    • Data Source
      KAKENHI-PROJECT-15570099
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not a affect blood glucose levels2004

    • Author(s)
      Yuasa T.
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Description
      「研究成果報告書概要(和文)」より
    • Data Source
      KAKENHI-PROJECT-16390097
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Description
      「研究成果報告書概要(欧文)」より
    • Data Source
      KAKENHI-PROJECT-15590249
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF recept or in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Description
      「研究成果報告書概要(和文)」より
    • Data Source
      KAKENHI-PROJECT-15590249
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Data Source
      KAKENHI-PROJECT-15659079
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Data Source
      KAKENHI-PROJECT-16390097
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Data Source
      KAKENHI-PROJECT-16046215
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Tomoyuki Yuasa
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Description
      「研究成果報告書概要(欧文)」より
    • Data Source
      KAKENHI-PROJECT-15570099
  • [Journal Article] Platelet-derived growth factor stimulates glucose transport in skeletal muscles of transgenic mice specifically expressing PDGF receptor in the muscle, but does not affect blood glucose levels2004

    • Author(s)
      Yuasa T. et al.
    • Journal Title

      Diabetes 53

      Pages: 2776-2786

    • Description
      「研究成果報告書概要(欧文)」より
    • Data Source
      KAKENHI-PROJECT-16390097
  • [Patent] 糖尿病治療薬の新規なスクリーニング方法2013

    • Inventor(s)
      湯浅智之,松久宗英,橋田誠一,蛯名洋介
    • Industrial Property Rights Holder
      徳島大学
    • Industrial Property Rights Type
      特許
    • Filing Date
      2013-04-17
    • Data Source
      KAKENHI-PROJECT-24790317
  • [Patent] 糖尿病治療薬の新規なスクリーニング方法2013

    • Inventor(s)
      湯浅智之,蛯名洋介,橋田誠一,松久宗英
    • Industrial Property Rights Holder
      徳島大学
    • Industrial Property Rights Type
      特許
    • Industrial Property Number
      2013-086477
    • Filing Date
      2013-04-17
    • Data Source
      KAKENHI-PROJECT-24790317
  • [Presentation] インスリン受容体切断が妊娠期に果たすインスリン作用調節機構の可能性2019

    • Author(s)
      湯浅智之 安藝菜奈子 國見幸太郎 佐藤美紀 新居真里 和泉佳彦 高田康徳 乙田敏城 大澤春彦 橋田誠一 粟飯原賢一
    • Organizer
      第62回糖尿病学会総会
    • Data Source
      KAKENHI-PROJECT-19K09027
  • [Presentation] インスリン受容体の切断が担う 新規インスリン抵抗性分子機構2015

    • Author(s)
      湯浅智之
    • Organizer
      脳心血管抗加齢研究会2015
    • Place of Presentation
      梅田スカイビル「大阪府・大阪市」
    • Year and Date
      2015-11-29
    • Invited
    • Data Source
      KAKENHI-PROJECT-15K09391
  • [Presentation] インスリン受容体二段階切断が担う 新規インスリン抵抗性分子機構2015

    • Author(s)
      湯浅智之
    • Organizer
      第58回糖尿病学会年次学術集会
    • Place of Presentation
      グランブラスセントヴァレンタイン「山口県・下関市」
    • Year and Date
      2015-05-22
    • Data Source
      KAKENHI-PROJECT-15K09391
  • [Presentation] 可溶性インスリン受容体が担うインスリン抵抗性の可能性2013

    • Author(s)
      湯浅智之,松久宗英,橋田誠一,蛯名洋介
    • Organizer
      第56回日本糖尿病学会年次学術集会特別シンポジウム
    • Place of Presentation
      熊本ホテルキャッスル(熊本県)
    • Year and Date
      2013-05-17
    • Data Source
      KAKENHI-PROJECT-24790317
  • [Presentation] 新規糖尿病関連分子可溶性インスリン受容体の基礎と臨床2012

    • Author(s)
      湯浅智之
    • Organizer
      第57回日本臨床検査医学会中国・四国支部総会,第152回日本臨床化学会中国支部例会・総会,第22回日本臨床化学会四国支部例会・総会,第8回合同地方会
    • Place of Presentation
      岡山大学(岡山市)
    • Year and Date
      2012-02-05
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] 新規糖尿病関連分子可溶性インスリン受容体の基礎と臨床2012

    • Author(s)
      湯浅智之
    • Organizer
      第57回日本臨床検査医学会中国・四国支部総会第152回日本臨床化学会中国支部例会・総会第22回日本臨床化学会四国支部例会・総会第8回合同地方会
    • Place of Presentation
      岡山大学(岡山県)(招待講演)
    • Year and Date
      2012-02-05
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] 短期血糖コントロール指標としての血中可溶性インスリン受容体-ステロイドによる糖代謝悪化を検出できるのか?-2011

    • Author(s)
      本島寛之、湯浅智之, 他
    • Organizer
      第54回日本糖尿病学会年次学術集会
    • Place of Presentation
      ロイトン札幌(札幌市)
    • Year and Date
      2011-05-19
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] Soluble insulin receptor ectodomain is elevated in the plasma of patients with diabetes.2010

    • Author(s)
      Yuasa Tomoyuki, Nagaya Hisao, Hashida Seiichi, Asako Umehara, Yokota Ichiro, Obata Toshiyuki, Ebina Yousuke
    • Organizer
      XI INTERNATIONAL SYMPOSIUM ON INSULIN RECEPTORS AND INSULIN ACTION
    • Place of Presentation
      Naples
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] 尿中成長ホルモンを指標とした運動評価の検討2010

    • Author(s)
      梅原麻子, 湯浅智之, 他
    • Organizer
      第53回日本糖尿病学会年次学術集会
    • Place of Presentation
      ホテルグランヴィア岡山(岡山市)
    • Year and Date
      2010-05-27
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] Soluble insulin receptor ectodomain is elevated in the plasma of patients with diabetes.2010

    • Author(s)
      Yuasa T., et.al.
    • Organizer
      XI INTERNATIONAL SYMPOSIUM ON INSULIN RECEPTORS AND INSULIN ACTION
    • Place of Presentation
      イタリア・ナポリ
    • Year and Date
      2010-10-28
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] 短期血糖コントロール指標としての血中可溶性インスリン受容体αサブユニットの有用性2010

    • Author(s)
      本島寛之、湯浅智之, 他
    • Organizer
      第53回日本糖尿病学会年次学術集会
    • Place of Presentation
      ホテルグランヴィア岡山(岡山市)
    • Year and Date
      2010-05-28
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] Soluble insulin receptor ectodomain is elevated in the plasma of patients with diabetes. XI INTERNATIONAL SYMPOSIUM ON INSULIN RE2010

    • Author(s)
      湯浅智之
    • Organizer
      CEPTORS AND INSULIN ACTION
    • Place of Presentation
      Royal Continental Hotel(イタリア・ナポリ)
    • Year and Date
      2010-10-28
    • Data Source
      KAKENHI-PROJECT-22790317
  • [Presentation] Soluble insulin receptor ectodomain is elevated in the plasma of patients with diabetes.2010

    • Author(s)
      Yuasa T., et.al.
    • Organizer
      XI INTERNATIONAL SYMPOSIUM ON INSULIN RECEPTORS AND INSULIN ACTION
    • Place of Presentation
      イタリア・ナポリ
    • Year and Date
      2010-10-28
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] Soluble insulin receptor ectodomain in the plasma is a possible broad-spectrum tumor marker2009

    • Author(s)
      湯浅智之, 矢野聖二, 西岡安彦, 久保宜明, 高橋正幸, 中逵弘能, 長屋寿雄, 金山博臣, 荒瀬誠治, 曽根三郎, 蛯名洋介
    • Organizer
      第68回 日本癌学会学術総会記事,pp.450-450
    • Place of Presentation
      Yokohama
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] Soluble insulin receptor ectodomain level was elevated in the plasma of cancer patients2009

    • Author(s)
      Yuasa T., et. al.
    • Organizer
      第29回 日本分子腫瘍マーカー研究会
    • Place of Presentation
      横浜
    • Year and Date
      2009-09-30
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] Soluble insulin receptor ectodomain in the plasma is a possible broad-spectrum tumor marker2009

    • Author(s)
      Yuasa T., et. al.
    • Organizer
      第68回 日本癌学会学術総会
    • Place of Presentation
      横浜
    • Year and Date
      2009-10-01
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] 可溶性インスリン受容体細胞外ドメイン(sIRα)は高血糖に相関して血中濃度が高まる2009

    • Author(s)
      湯浅智之, 他
    • Organizer
      第7回 1型糖尿病研究会
    • Place of Presentation
      日光
    • Year and Date
      2009-11-07
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] 可溶化インスリン受容体細胞外ドメイン(sIR)は癌患者血清中で増加している2009

    • Author(s)
      湯浅智之, 小畑利之, 矢野聖二, 岡本英治, 西岡安彦, 久保宜明, 高橋正幸, 中逵弘能, 長屋寿雄, 金山博臣, 荒瀬誠治, 曽根三郎, 蛯名洋介
    • Organizer
      第29回日本分子腫瘍マーカー研究会
    • Place of Presentation
      横浜
    • Year and Date
      2009-09-30
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] 可溶性インスリン受容体細胞外ドメイン(sIRα)は高血糖に相関して血中濃度が高まる2009

    • Author(s)
      湯浅智之, 小畑利之, 横田一郎, 岡本英治, 長屋寿雄, 橋田誠一, 前川聡, 柏木厚典, 松本満, 松本俊夫, 岸和弘, 蛯名洋介
    • Organizer
      第7回1型糖尿病研究会
    • Place of Presentation
      日光
    • Year and Date
      2009-11-07
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] Soluble insulin receptor ectodomain is elevated in the plasma of patients with diabetes.2008

    • Author(s)
      Yuasa Tomoyuki, Obata Toshiyuki, Yokota Ichiro, Okamoto Eiji, Kanezaki Yoshiko, Maegawa Hiroshi, Hirota Fumiko, Kishi Kazuhiro, Hashida Seiichi, Nagaya Hisao, Yuko Ogura, Kazuhiko Masuda, Matsumoto Mitsuru, Matsumoto Toshio, Kashiwagi Atsunori, Ebina Yousuke
    • Organizer
      The 1st Insulin Resistance in Metabolic Disease Forum
    • Place of Presentation
      Osaka
    • Year and Date
      2008-08-20
    • Data Source
      KAKENHI-PROJECT-20390095
  • [Presentation] 可溶性インスリン受容体が担うインスリン抵抗性の可能性

    • Author(s)
      湯浅智之,松久宗英,橋田誠一,蛯名洋介
    • Organizer
      第56回日本糖尿病学会年次学術集会
    • Place of Presentation
      熊本県(熊本ホテルキャッスル)
    • Invited
    • Data Source
      KAKENHI-PROJECT-24790317
  • 1.  EBINA Yousuke (00112227)
    # of Collaborated Projects: 23 results
    # of Collaborated Products: 6 results
  • 2.  KISHI Kazuhiro (70284320)
    # of Collaborated Projects: 16 results
    # of Collaborated Products: 0 results
  • 3.  OBATA Toshiyuki (40325296)
    # of Collaborated Projects: 15 results
    # of Collaborated Products: 0 results
  • 4.  TESHIGAWARA Kiyoshi (40403737)
    # of Collaborated Projects: 3 results
    # of Collaborated Products: 0 results
  • 5.  HOSAKA Toshio (60403698)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 0 results
  • 6.  YOKOYAMA Kazuhiro (00346602)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 0 results
  • 7.  UCHIYAMA Keiji (60294039)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 1 results
  • 8.  NAGAYA Hisao (60464343)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 5 results
  • 9.  OGURA Yuko (90464354)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 2 results
  • 10.  TANIGUCHI Hisaaki (10257636)
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 0 results
  • 11.  ASAHI Yoshihiko
    # of Collaborated Projects: 2 results
    # of Collaborated Products: 0 results
  • 12.  KUSUNOKI Masami (90135749)
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 13.  岡本 英治
    # of Collaborated Projects: 1 results
    # of Collaborated Products: 0 results
  • 14.  HASHIDA Seiichi
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 2 results
  • 15.  石倉 周平
    # of Collaborated Projects: 0 results
    # of Collaborated Products: 1 results

URL: 

Are you sure that you want to connect your ORCID iD to this researcher?
* This action can be performed only by the researcher themselves.

この研究者とORCID iDの連携を行いますか?
※ この処理は、研究者本人だけが実行できます。

Information User Guide FAQ News Terms of Use Attribution of KAKENHI

Powered by NII kakenhi